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210 results on '"Chancroid microbiology"'

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1. Sequence typing of Haemophilus ducreyi isolated from patients in the Namatanai region of Papua New Guinea: Infections by Class I and Class II strain types differ in ulcer duration and resurgence of infection after azithromycin treatment.

2. A Haemophilus ducreyi strain lacking the yfeABCD iron transport system is virulent in human volunteers.

3. Transmission of viable Haemophilus ducreyi by Musca domestica.

4. Interactions of the Skin Pathogen Haemophilus ducreyi With the Human Host.

5. Multiplex Mediator Displacement Loop-Mediated Isothermal Amplification for Detection of Treponema pallidum and Haemophilus ducreyi.

6. A Class I Haemophilus ducreyi Strain Containing a Class II hgbA Allele Is Partially Attenuated in Humans: Implications for HgbA Vaccine Efficacy Trials.

7. Multiple Class I and Class II Haemophilus ducreyi Strains Cause Cutaneous Ulcers in Children on an Endemic Island.

8. First reported case of chancroid in the Czech Republic.

9. Haemophilus ducreyi cutaneous ulcer contracted at Seram Island, Indonesia, presented in the Netherlands.

10. Direct Whole-Genome Sequencing of Cutaneous Strains of Haemophilus ducreyi.

11. Single-Dose Azithromycin for the Treatment of Haemophilus ducreyi Skin Ulcers in Papua New Guinea.

12. Haemophilus ducreyi Cutaneous Ulcer Strains Diverged from Both Class I and Class II Genital Ulcer Strains: Implications for Epidemiological Studies.

13. First case of chancroid in 14 years at the largest STI clinic in Paris, France.

14. Haemophilus ducreyi Seeks Alternative Carbon Sources and Adapts to Nutrient Stress and Anaerobiosis during Experimental Infection of Human Volunteers.

15. Haemophilus ducreyi: from sexually transmitted infection to skin ulcer pathogen.

16. Surveillance and sexually transmitted infections: a story of chemsex, tropical leg ulcers, gonococcal resistance and child abuse.

17. DksA and (p)ppGpp have unique and overlapping contributions to Haemophilus ducreyi pathogenesis in humans.

18. Haemophilus ducreyi Cutaneous Ulcer Strains Are Nearly Identical to Class I Genital Ulcer Strains.

19. Phosphoethanolamine Transferase LptA in Haemophilus ducreyi Modifies Lipid A and Contributes to Human Defensin Resistance In Vitro.

20. Defining Potential Vaccine Targets of Haemophilus ducreyi Trimeric Autotransporter Adhesin DsrA.

21. Molecular phylogenetic analysis of non-sexually transmitted strains of Haemophilus ducreyi.

22. Haemophilus ducreyi associated with skin ulcers among children, Solomon Islands.

23. Chancroid - desperate patient makes own diagnosis.

24. Cytolethal distending toxins require components of the ER-associated degradation pathway for host cell entry.

25. Outer membrane protein P4 is not required for virulence in the human challenge model of Haemophilus ducreyi infection.

26. The Haemophilus ducreyi LspA1 protein inhibits phagocytosis by using a new mechanism involving activation of C-terminal Src kinase.

27. Haemophilus ducreyi as a cause of skin ulcers in children from a yaws-endemic area of Papua New Guinea: a prospective cohort study.

28. Chancroid, lymphogranuloma venereum, granuloma inguinale, genital herpes simplex infection, and molluscum contagiosum.

29. Haemophilus ducreyi Hfq contributes to virulence gene regulation as cells enter stationary phase.

30. Carbon storage regulator A contributes to the virulence of Haemophilus ducreyi in humans by multiple mechanisms.

31. Haemophilus ducreyi-induced interleukin-10 promotes a mixed M1 and M2 activation program in human macrophages.

32. Permeases of the sap transporter are required for cathelicidin resistance and virulence of Haemophilus ducreyi in humans.

33. Sialylation of lipooligosaccharides is dispensable for the virulence of Haemophilus ducreyi in humans.

34. Chancroid transmission dynamics: a mathematical modeling approach.

35. Expression of the Flp proteins by Haemophilus ducreyi is necessary for virulence in human volunteers.

36. Chancroid in an HIV-negative woman in the Midwestern United States.

37. On the evolution of the sexually transmitted bacteria Haemophilus ducreyi and Klebsiella granulomatis.

38. A Haemophilus ducreyi CpxR deletion mutant is virulent in human volunteers.

39. Deletion of mtrC in Haemophilus ducreyi increases sensitivity to human antimicrobial peptides and activates the CpxRA regulon.

40. Use of signature-tagged mutagenesis to identify virulence determinants in Haemophilus ducreyi responsible for ulcer formation.

41. Chronic cutaneous ulcers secondary to Haemophilus ducreyi infection.

42. Etiology of genital ulcer disease. A prospective study of 278 cases seen in an STD clinic in Paris.

43. Haemophilus ducreyi SapA contributes to cathelicidin resistance and virulence in humans.

44. Host-pathogen interplay of Haemophilus ducreyi.

45. Mechanism of human natural killer cell activation by Haemophilus ducreyi.

46. Inactivation of the Haemophilus ducreyi luxS gene affects the virulence of this pathogen in human subjects.

47. Experimental infection of human volunteers with Haemophilus ducreyi: fifteen years of clinical data and experience.

48. Haemophilus ducreyi detection by polymerase chain reaction in oesophageal lesions of HIV patients.

49. Comparative proteomic analysis of the Haemophilus ducreyi porin-deficient mutant 35000HP::P2AB.

50. A fibrinogen-binding lipoprotein contributes to the virulence of Haemophilus ducreyi in humans.

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