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Your search keyword '"Cardiac autonomic modulation"' showing total 6 results
Start Over Descriptor "Cardiac autonomic modulation" Publication Type Electronic Resources
6 results on '"Cardiac autonomic modulation"'

1. Biological Mechanisms Underlying Physical Fitness and Sports Performance.

Author

Georgian, Badicu, Clemente, Filipe Manuel, Georgian, Badicu, and Murawska-Ciałowicz, Eugenia

Subjects
Biology, life sciences, Research & information: general, 25(OH)D, BIA, BIVA, FTP, HRQoL, IgA, Paralympic Powerlifting, Tabata training, acid-base balance, acute exercise, adolescent, aerobic capacity, aerobic training, age, antioxidant, aquatic training, athletic performance, back squat, biological mechanisms, blood pressure, body composition, body fat, cancer survivorship, cardiac autonomic modulation, cardiorespiratory fitness, cardiovascular fitness, chronic low back pain, cognitive performance, combined training, core endurance test, coronavirus disease, cycling, depression, detraining, dry land training, electromyography, endurance, ergogenic aids, erythroferrone, erythropoietin, executive functions, exercise, fat mass, flanker, football, glucagon-like peptide-1, glucose-dependent insulinotropic polypeptide, health, heart rate, heart rate recovery, heart rate variability, high-intensity, high-intensity exercise, high-intensity interval training, high-speed running, hormonal response, hormones, hypoxic dose, ibuprofen, immunomodulation, inflammation, kickboxing, kinanthropometry, knowledge, lean body mass, load, log-linear analysis, mathematical model, mental health, metabolic acidosis, monotony, multivariate correspondence analysis, muscle damage, muscle fatigue, muscle mass, muscle regeneration, muscle soreness, muscle strength, muscular activity, muscular endurance, muscular performance, myogenic regulatory factors, n/a, non-planned agility, nutritional supplement, obesity, one repetition maximum, oxidative stress, oxygen uptake, performance, physical activity, physical education, physical education lessons, physical exercise, physical health, physiology of performances, power, pre-planned agility, psychophysiological responses, puberty, recovery, recovery of function, resistance training, rheumatic diseases, rowing, saliva, satellite cells, sex difference, skin surface temperature, sleep, small-sided games, soccer, spinal cord injury, sport, sports, sports and exercise physiology, sprint, strain, strength, strength training, stress, surface electromyography, swimming performance, team sports, thermal imaging, threshold, triathlon, tumour, type 2 diabetes, vibrating exercise equipment, water parachute, weight loss, women, young athlete, youth
Abstract

Summary: The concept of mechanism in biology has three distinct meanings. It may refer to a philosophical thesis about the nature of life and biology, to the internal workings of a machine-like structure, or to the causal explanation of a particular phenomenon. In this Special Issue, we try to discuss these possible biological mechanisms that underlie the beneficial effects of physical fitness and sports performance, as well their importance and role/influences on physical health.Despite the significant body of knowledge regarding the physiological and physical effects of different training methods (based on dimensions of load), some of the biological causes for those changes are still unknown. Additionally, few studies have focused on the natural biological variability in humans and how specific properties of humans may justify different effects for the same training intervention. Thus, more original research is needed to provide plausible biological mechanisms that may explain the physiological and physical effects of exercise and training in humans.In this Special Issue, we gather the contributions that describe and list the links between physical fitness, sports performance, and human biology.

3. The use of cardiac autonomic responses to aerobic exercise in elderly stroke patients: functional rehabilitation as a public health policy

Author

Faculdade de Medicina do ABC and Acre State Government., Raimundo, Rodrigo Daminello, Zangirolami-Raimundo, Juliana, Leone, Claudio, de Carvalho, Tatiana Dias, da Silva, Talita Dias, Bezerra, Italla Maria Pinheiro, de Almeida, Alvaro Dantas, Valenti, Vitor Engracia, de Abreu, Luiz Carlos, Faculdade de Medicina do ABC and Acre State Government., Raimundo, Rodrigo Daminello, Zangirolami-Raimundo, Juliana, Leone, Claudio, de Carvalho, Tatiana Dias, da Silva, Talita Dias, Bezerra, Italla Maria Pinheiro, de Almeida, Alvaro Dantas, Valenti, Vitor Engracia, and de Abreu, Luiz Carlos

Abstract

peer-reviewed, Background and purpose: The development of public policies must be guided by full knowledge of the health–disease process of the population. Aerobic exercises are recommended for rehabilitation in stroke patients, and have been shown to improve heart rate variability (HRV). Our aim was to compare the cardiac autonomic modulation of elderly stroke patients with that of healthy elderly people during and after an acute bout of aerobic exercise. Methods: A total of 60 elderly people participated in the study (30 in the control group, mean age of 67 ± 4 years; 30 in the stroke group, mean age of 69 ± 3 years). HRV was analyzed in rest—10 min of rest in supine position; exercise—the 30 min of peak exercise; and recovery—30 min in supine position post-exercise. Results: Taking rest and exercises together, for SDNN, RMSSD, pNN50, RRTri, and TINN, there was no difference between the stroke and control groups (p = 0.062; p = 0.601; p = 0.166; p = 0.224, and p = 0.059, respectively). The HF (ms2 ) was higher and the LF/HF ratio was lower for the stroke group than the control group (p < 0.001 and p = 0.007, respectively). The SD2 was lower for the stroke group than for the control group (p = 0.041). Conclusion: Stroke patients present reduced variability at rest, sympathetic predominance during exercise, and do not return to baseline after the 30 min of recovery, with similar responses found in the healthy elderly group. Keywords: autonomic nervous system; aging; heart rate variability.

4. Cardiovascular responsiveness to sympathoexcitatory stress in subjects with and without mild hypertension

Author

White, LD, Carthy, ER, White, L, Russell, FD, Holmes, M, Leicht, AS, Brooks, PR, Hitchen-Holmes, D, Askew, CD, White, LD, Carthy, ER, White, L, Russell, FD, Holmes, M, Leicht, AS, Brooks, PR, Hitchen-Holmes, D, and Askew, CD

Abstract

Purpose: This study compared blood pressure, heart rate variability (HRV) and forearm blood flow, at rest and in response to sympathoexcitatory stressors between normotensive and mildly hypertensive participants. Methods: Participants aged 30-79 years with normal blood pressure (n = 49) or mild hypertension (n = 17), with no history of taking antihypertensive medication, were recruited. Participants completed a cold pressor test (CPT) followed by an ischaemic handgrip test (IHGT). Blood pressure, HRV, forearm blood flow and vascular resistance were measured at rest and in response to each test. Results: The CPT and IHGT evoked greater increases in mean arterial blood pressure in hypertensive participants (CPT: 10 ± 2 mmHg, IHGT: 9 ± 1 mmHg) compared with normotensive participants (CPT: 5 ± 1 mmHg, IHGT: 3 ± 1 mmHg; P < 0.05). Resting high frequency power, which is a parameter of HRV associated with parasympathetic cardiac modulation, was lower in hypertensive participants (hypertensive: 31.73 ± 4.07 nu; normotensive: 42.08 ± 2.22 nu; P = 0.026) and was negatively correlated with systolic blood pressure (r = -0.272, P = 0.03) and mean arterial pressure across all participants (r = -0.258, P < 0.05). There were no differences in HRV or forearm blood flow responses to the CPT or IHGT between groups. Conclusion: This study demonstrated that sympathoexcitatory stress evoked by the CPT and IHGT induces an augmented blood pressure response in individuals with mild hypertension, which supports the notion that autonomic dysfunction is likely to contribute to the pathogenesis of hypertension. It remains to be determined whether the hypertensive response is mediated through alterations in cardiac activity, peripheral vascular resistance or both.

5. Cardiovascular responsiveness to sympathoexcitatory stress in subjects with and without mild hypertension

Author

White, LD, Carthy, ER, White, L, Russell, FD, Holmes, M, Leicht, AS, Brooks, PR, Hitchen-Holmes, D, Askew, CD, White, LD, Carthy, ER, White, L, Russell, FD, Holmes, M, Leicht, AS, Brooks, PR, Hitchen-Holmes, D, and Askew, CD

Abstract

Purpose: This study compared blood pressure, heart rate variability (HRV) and forearm blood flow, at rest and in response to sympathoexcitatory stressors between normotensive and mildly hypertensive participants. Methods: Participants aged 30-79 years with normal blood pressure (n = 49) or mild hypertension (n = 17), with no history of taking antihypertensive medication, were recruited. Participants completed a cold pressor test (CPT) followed by an ischaemic handgrip test (IHGT). Blood pressure, HRV, forearm blood flow and vascular resistance were measured at rest and in response to each test. Results: The CPT and IHGT evoked greater increases in mean arterial blood pressure in hypertensive participants (CPT: 10 ± 2 mmHg, IHGT: 9 ± 1 mmHg) compared with normotensive participants (CPT: 5 ± 1 mmHg, IHGT: 3 ± 1 mmHg; P < 0.05). Resting high frequency power, which is a parameter of HRV associated with parasympathetic cardiac modulation, was lower in hypertensive participants (hypertensive: 31.73 ± 4.07 nu; normotensive: 42.08 ± 2.22 nu; P = 0.026) and was negatively correlated with systolic blood pressure (r = -0.272, P = 0.03) and mean arterial pressure across all participants (r = -0.258, P < 0.05). There were no differences in HRV or forearm blood flow responses to the CPT or IHGT between groups. Conclusion: This study demonstrated that sympathoexcitatory stress evoked by the CPT and IHGT induces an augmented blood pressure response in individuals with mild hypertension, which supports the notion that autonomic dysfunction is likely to contribute to the pathogenesis of hypertension. It remains to be determined whether the hypertensive response is mediated through alterations in cardiac activity, peripheral vascular resistance or both.

6. Cardiovascular responsiveness to sympathoexcitatory stress in subjects with and without mild hypertension

Author

Carthy, ER, White, LD, White, L, Russell, FD, Holmes, M, Leicht, AS, Brooks, PR, Hitchen-Holmes, D, Askew, CD, Carthy, ER, White, LD, White, L, Russell, FD, Holmes, M, Leicht, AS, Brooks, PR, Hitchen-Holmes, D, and Askew, CD

Abstract

Purpose: This study compared blood pressure, heart rate variability (HRV) and forearm blood flow, at rest and in response to sympathoexcitatory stressors between normotensive and mildly hypertensive participants. Methods: Participants aged 30-79 years with normal blood pressure (n = 49) or mild hypertension (n = 17), with no history of taking antihypertensive medication, were recruited. Participants completed a cold pressor test (CPT) followed by an ischaemic handgrip test (IHGT). Blood pressure, HRV, forearm blood flow and vascular resistance were measured at rest and in response to each test. Results: The CPT and IHGT evoked greater increases in mean arterial blood pressure in hypertensive participants (CPT: 10 ± 2 mmHg, IHGT: 9 ± 1 mmHg) compared with normotensive participants (CPT: 5 ± 1 mmHg, IHGT: 3 ± 1 mmHg; P < 0.05). Resting high frequency power, which is a parameter of HRV associated with parasympathetic cardiac modulation, was lower in hypertensive participants (hypertensive: 31.73 ± 4.07 nu; normotensive: 42.08 ± 2.22 nu; P = 0.026) and was negatively correlated with systolic blood pressure (r = -0.272, P = 0.03) and mean arterial pressure across all participants (r = -0.258, P < 0.05). There were no differences in HRV or forearm blood flow responses to the CPT or IHGT between groups. Conclusion: This study demonstrated that sympathoexcitatory stress evoked by the CPT and IHGT induces an augmented blood pressure response in individuals with mild hypertension, which supports the notion that autonomic dysfunction is likely to contribute to the pathogenesis of hypertension. It remains to be determined whether the hypertensive response is mediated through alterations in cardiac activity, peripheral vascular resistance or both.

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