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Policy responses to the challenges associated with environmental change, including more frequent and severe climatic events, have interlinked environmental and social impacts. Less attention has been afforded to the latter, and specifically to the question of not just whether but how such responses create or entrench inequality. This paper examines policy responses to drought events in California, United States, and the Western Cape Province, South Africa, in terms of their effects on inequality, revealed in relationships to water access networks. We use concepts of water justice and hydraulic citizenship to evaluate how and why these policy responses reproduced water injustices in the two settings. We focus particularly on two mechanisms linking responses to widened inequalities: values‐reinforcement and strategic communication. Using interviews, policy documents, and media reports, we employ process tracing methods to illustrate these mechanisms through which drought policy impacts hydraulic citizenship experiences, manifesting water injustice. We contribute to emerging examinations of environmental policy responses and maladaptation by demonstrating how concepts of hydraulic citizenship and an emphasis on mechanisms can help us better understand and identify experiences of water injustice. We note policy implications and areas for future research, highlighting droughts as consequential policy sites for advancing social and environmental justice. [ABSTRACT FROM AUTHOR]

Phlyctinus callosus and P. xerophilus (Coleoptera: Curculionidae) are two cryptic species of native entimine weevils, previously grouped together under the P. callosus sensu lato concept, that are pests of economic importance to the deciduous fruit and vine industry in the Western Cape province of South Africa. Laboratory bioassays were conducted using entomopathogenic fungi (EPF) isolates of Beauveria and entomopathogenic nematodes (EPNs), Heterorhabditis indica and Steinernema yirgalemense, to determine differences in susceptibility of adult P. callosus and P. xerophilus to potential biological control agents. The test arena used was 24‐well bioassay plates with an inoculation rate of 200 infective juveniles (IJs)/insect for EPNs and 5 × 105 conidia/insect for EPF. Insects were inoculated using a 12.7 mm filter paper impregnated with 50 μL of entomopathogen suspension. Infection was determined after 96 h incubation for EPNs through dissection of cadavers. Insects inoculated with EPF were incubated in the wells for 18 days and mortality recorded daily. Cadavers were surface sterilized and observed for overt mycosis. Differential susceptibility between P. callosus and P. xerophilus was observed in EPF but not in EPN bioassays. Differential susceptibility to EPF could be due to methodology. Low adult weevil pathogenicity was found for all entomopathogens, with variable results obtained from different bioassay batches. [ABSTRACT FROM AUTHOR]

Aim: To better understand the aetiologies of epileptic spasms in infants, as well as the safety and efficacy of high dose corticosteroids in tuberculosis and human immunodeficiency virus (HIV) endemic resource‐limited settings. Method: This was a retrospective analysis of infants with epileptic spasms managed at the tertiary referral centres in the Western Cape, South Africa. Results: Of 175 children with epileptic spasms, the median age at onset was 6 months (interquartile range 4–8 months). Structural aetiologies were most common (115 out of 175 [66%]), with two‐thirds related to perinatal insults. A lead time to treatment (LTTT) of less than 1 month was more likely in the epileptic encephalopathy/developmental and epileptic encephalopathy (DEE) group: 58 out of 92 (63%), compared to 28 out of 76 (37%) of those with developmental encephalopathy (p = 0.001). Failure to recognize preceding developmental delay was common. Ninety‐nine children (57%) received first line hormonal therapy such as adrenocorticotropic hormone. A total of 111 out of 172 children (65%) from the developmental encephalopathy and epileptic encephalopathy/DEE groups had clinical and/or electroencephalogram resolution of spasms within 14 days. In our population, children in whom an aetiology could not be identified were statistically more likely to have moderate to profound developmental delay at 1 year of age: 33 out of 44 (p = 0.001). Based on reported incidence of epileptic spasms, 23 to 58 cases per annum would be expected but a far smaller proportion presented to our centres. Interpretation: Whilst this is the largest cohort of infants with epileptic spasms from sub‐Saharan Africa, the study size is less than expected; this may reflect misdiagnosis and failure of referral pathways. Despite a reported shorter LTTT, infants with DEE had worse developmental outcomes compared to international studies. Hormonal therapy was safe and effective in our setting, despite exposure to high levels of tuberculosis and HIV. What this paper adds: The number of unreferred cases of epileptic spasms in South Africa remains high.Caregivers and health care workers in primary care facilities often fail to recognize developmental delay.The burden of disease from hypoxic‐ischaemic encephalopathy remains high in our resource‐limited setting.Hormonal treatment (e.g. adrenocorticotropic hormone) was safe and effective despite the high prevalence of human immunodeficiency virus and tuberculosis. [ABSTRACT FROM AUTHOR]