23 results on '"Allert, Stefanie"'
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2. Secretion of the fungal toxin candidalysin is dependent on conserved precursor peptide sequences
3. Candida albicans translocation through the intestinal epithelial barrier is promoted by fungal zinc acquisition and limited by NFκB-mediated barrier protection
4. Nanobody-mediated neutralization of candidalysin prevents epithelial damage and inflammatory responses that drive vulvovaginal candidiasis pathogenesis
5. FSP1-specific SMAD2 knockout in renal tubular, endothelial, and interstitial cells reduces fibrosis and epithelial-to-mesenchymal transition in murine STZ-induced diabetic nephropathy
6. Candidalysin Is the Hemolytic Factor of Candida albicans
7. From environmental adaptation to host survival: Attributes that mediate pathogenicity of Candida auris
8. Calcium-dependent ESCRT recruitment and lysosome exocytosis maintain epithelial integrity during Candida albicans invasion
9. Cover Image: Candidalysin delivery to the invasion pocket is critical for host epithelial damage induced by Candida albicans (Cellular Microbiology 10/2021)
10. A variant ECE1 allele contributes to reduced pathogenicity of Candida albicans during vulvovaginal candidiasis
11. Candidalysin delivery to the invasion pocket is critical for host epithelial damage induced by Candida albicans
12. Calcium-Dependent ESCRT Recruitment and Lysosome Exocytosis Maintain Epithelial Integrity During Candida albicans Invasion
13. Survival Strategies of Pathogenic Candida Species in Human Blood Show Independent and Specific Adaptations
14. Fungal biotin homeostasis is essential for immune evasion after macrophage phagocytosis and virulence
15. Keeping Candida commensal – How lactobacilli antagonize pathogenicity of Candida albicans in an in vitro gut model
16. Candida albicans-Induced Epithelial Damage Mediates Translocation through Intestinal Barriers
17. FSP1-specific SMAD2 knockout in renal tubular, endothelial, and interstitial cells reduces fibrosis and epithelial-to-mesenchymal transition in murine STZ-induced diabetic nephropathy
18. The Snf1-activating kinase Sak1 is a key regulator of metabolic adaptation andin vivofitness ofCandida albicans
19. In Vivo Transcriptional Profiling of Human Pathogenic Fungi during Infection: Reflecting the Real Life?
20. FSP1-specific <italic>SMAD2</italic> knockout in renal tubular, endothelial, and interstitial cells reduces fibrosis and epithelial-to-mesenchymal transition in murine STZ-induced diabetic nephropathy.
21. The Snf1-activating kinase Sak1 is a key regulator of metabolic adaptation and in vivo fitness of Candida albicans.
22. Survival Strategies of Pathogenic CandidaSpecies in Human Blood Show Independent and Specific Adaptations
23. A highly conserved tRNA modification contributes to C. albicans filamentation and virulence.
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