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27 results on '"Frappart, Lucien"'

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1. TRIP6 functions in brain ciliogenesis

2. Merlin Isoforms 1 and 2 Both Act as Tumour Suppressors and Are Required for Optimal Sperm Maturation.

3. In vivo reduction of RAD51‐mediated homologous recombination triggers aging but impairs oncogenesis

5. Supplemental Figure S3 from Impaired Planar Germ Cell Division in the Testis, Caused by Dissociation of RHAMM from the Spindle, Results in Hypofertility and Seminoma

6. Supplemental Figure Legends from Impaired Planar Germ Cell Division in the Testis, Caused by Dissociation of RHAMM from the Spindle, Results in Hypofertility and Seminoma

7. Supplemental Table S1 from Impaired Planar Germ Cell Division in the Testis, Caused by Dissociation of RHAMM from the Spindle, Results in Hypofertility and Seminoma

8. Data from Impaired Planar Germ Cell Division in the Testis, Caused by Dissociation of RHAMM from the Spindle, Results in Hypofertility and Seminoma

9. Supplemental Materials & Methods from Impaired Planar Germ Cell Division in the Testis, Caused by Dissociation of RHAMM from the Spindle, Results in Hypofertility and Seminoma

10. In vivo inactivation of RAD51-mediated homologous recombination leads to premature aging, but not to tumorigenesis

11. Isolation, ‘omics characterization and organotypic culture of alveolar type II pulmonary epithelial cells

12. Cluster of differentiation 44 promotes osteosarcoma progression in mice lacking the tumor suppressor Merlin

14. Novel function of TRIP6, in brain ciliogenesis

18. HMMR acts in the PLK1-dependent spindle positioning pathway and supports neural development

20. Author response: HMMR acts in the PLK1-dependent spindle positioning pathway and supports neural development

21. Glucocorticoids limit acute lung inflammation in concert with inflammatory stimuli by induction of SphK1

23. Impaired Planar Germ Cell Division in the Testis, Caused by Dissociation of RHAMM from the Spindle, Results in Hypofertility and Seminoma

27. Glucocorticoids limit acute lung inflammation in concert with inflammatory stimuli by induction of SphK1.

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