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509 results on '"Silke, John"'

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1. An immunohistochemical atlas of necroptotic pathway expression

2. Brief Notices

4. Biallelic human SHARPIN loss of function induces autoinflammation and immunodeficiency

11. A common human MLKL polymorphism confers resistance to negative regulation by phosphorylation

13. Apoptotic cell death in disease—Current understanding of the NCCD 2023

14. MLKL deficiency protects against low-grade, sterile inflammation in aged mice

17. Mutations that prevent caspase cleavage of RIPK1 cause autoinflammatory disease

19. Development of NanoLuc-targeting protein degraders and a universal reporter system to benchmark tag-targeted degradation platforms

23. Clinical MDR1 inhibitors enhance Smac-mimetic bioavailability to kill murine LSCs and improve survival in AML models

25. Telemedical cardiac risk assessment by implantable cardiac monitors in patients after myocardial infarction with autonomic dysfunction (SMART-MI-DZHK9): a prospective investigator-initiated, randomised, multicentre, open-label, diagnostic trial

26. Single-cell spatial proteomics identifies the JAK/STAT pathway as an actionable therapeutic target in lethal cutaneous drug reactions

27. cIAPs control RIPK1 kinase activity‐dependent and ‐independent cell death and tissue inflammation

29. Targeting triple-negative breast cancers with the Smac-mimetic birinapant

31. Biallelic human SHARPINloss of function induces autoinflammation and immunodeficiency

32. IAPs and Cell Death

37. Movie 1 from Antagonism of IAPs Enhances CAR T-cell Efficacy

38. Data from Antagonism of IAPs Enhances CAR T-cell Efficacy

39. Supplementary Figures 1 and 2 from Antagonism of IAPs Enhances CAR T-cell Efficacy

40. Supplementary Data Legends from Antagonism of IAPs Enhances CAR T-cell Efficacy

42. Movie 2 from Antagonism of IAPs Enhances CAR T-cell Efficacy

46. MLKL trafficking and accumulation at the plasma membrane control the kinetics and threshold for necroptosis

47. A missense mutation in the MLKL brace region promotes lethal neonatal inflammation and hematopoietic dysfunction

48. LUBAC is essential for embryogenesis by preventing cell death and enabling haematopoiesis

50. Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018

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