197 results on '"Strebhardt, Klaus"'
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2. Correction: The non-apoptotic function of Caspase-8 in negatively regulating the CDK9-mediated Ser2 phosphorylation of RNA polymerase II in cervical cancer
3. The non-apoptotic function of Caspase-8 in negatively regulating the CDK9-mediated Ser2 phosphorylation of RNA polymerase II in cervical cancer
4. A dimerization-dependent mechanism regulates enzymatic activation and nuclear entry of PLK1
5. Non-mitotic functions of polo-like kinases in cancer cells
6. The role of caspase-8 in the tumor microenvironment of ovarian cancer
7. Caspase-8: The double-edged sword
8. Cover Image, Volume 44, Issue 1
9. Synergistic Sensitization of High-Grade Serous Ovarian Cancer Cells Lacking Caspase-8 Expression to Chemotherapeutics Using Combinations of Small-Molecule BRD4 and CDK9 Inhibitors
10. Rescue of p53 functions by in vitro‐transcribed mRNA impedes the growth of high‐grade serous ovarian cancer
11. Blocking Mitotic Exit of Ovarian Cancer Cells by Pharmaceutical Inhibition of the Anaphase-Promoting Complex Reduces Chromosomal Instability
12. Correction: A dimerization-dependent mechanism regulates enzymatic activation and nuclear entry of PLK1
13. Synergistic Sensitization of High-Grade Serous Ovarian Cancer Cells Lacking Caspase-8 Expression to Chemotherapeutics Using Combinations of Small-Molecule BRD4 and CDK9 Inhibitors.
14. Rescue of p53 functions by in vitro‐transcribed mRNA impedes the growth of high‐grade serous ovarian cancer.
15. Data from A Spatial and Functional Interaction of a Heterotetramer Survivin–DNA-PKcs Complex in DNA Damage Response
16. Supplementary Data from A Spatial and Functional Interaction of a Heterotetramer Survivin–DNA-PKcs Complex in DNA Damage Response
17. Shifting the selectivity of pyrido[2,3-d]pyrimidin-7(8H)-one inhibitors towards the salt-inducible kinase (SIK) subfamily
18. GTPase-activating protein Rasal1 associates with ZAP-70 of the TCR and negatively regulates T-cell tumor immunity
19. Immune adaptor SKAP1 acts a scaffold for Polo-like kinase 1 (PLK1) for the optimal cell cycling of T-cells
20. Immune adaptor protein SKAP1 (SKAP-55) forms homodimers as mediated by the N-terminal region
21. Prognostic Impact of Caspase-8, CDK9 and Phospho-CDK9 (Thr 186) Expression in Patients with Uterine Cervical Cancer Treated with Definitive Chemoradiation and Brachytherapy
22. Sequential Targeting of PLK1 and PARP1 Reverses the Resistance to PARP Inhibitors and Enhances Platin-Based Chemotherapy in BRCA-Deficient High-Grade Serous Ovarian Cancer with KRAS Amplification
23. The Role of DAPK1 in the Cell Cycle Regulation of Cervical Cancer Cells and in Response to Topotecan
24. Molecular Markers to Predict Prognosis and Treatment Response in Uterine Cervical Cancer
25. A dimerization-dependent mechanism regulates enzymatic activation and nuclear entry of PLK1
26. The small-molecule inhibitor MRIA9 reveals novel insights into the cell cycle roles of SIK2 in ovarian cancer cells
27. PLK1‐dependent phosphorylation restrains EBNA2 activity and lymphomagenesis in EBV‐infected mice
28. Mitotic arrest and slippage induced by pharmacological inhibition of Polo-like kinase 1
29. The Small-Molecule Inhibitor MRIA9 Reveals Novel Insights into the Cell Cycle Roles of SIK2 in Ovarian Cancer Cells
30. Structure-Based Design of Selective Salt-Inducible Kinase Inhibitors
31. Modelling the Functions of Polo-Like Kinases in Mice and Their Applications as Cancer Targets with a Special Focus on Ovarian Cancer
32. Targeting CDK9 for Anti-Cancer Therapeutics
33. Modelling the Functions of Polo-Like Kinases in Mice and Their Applications as Cancer Targets with a Special Focus on Ovarian Cancer
34. Structure-based design of selective salt-inducible kinase (SIK) inhibitors
35. A dimerization-dependent mechanism regulates enzymatic activation and nuclear entry of PLK1
36. Molecular markers to predict prognosis and treatment response in uterine cervical cancer
37. Structure-based design of selective Salt-inducible kinase (SIK) inhibitors
38. Structure-based design of selective salt-inducible kinase (SIK) inhibitors
39. Inhibition of PLK1-dependent EBNA2 phosphorylation promotes lymphomagenesis in EBV-infected mice
40. The prognostic relevance of the proliferation markers Ki-67 and Plk1 in early-stage ovarian cancer patients with serous, low-grade carcinoma based on mRNA and protein expression
41. The role of caspase-8 in the tumor microenvironment of ovarian cancer
42. A Spatial and Functional Interaction of a Heterotetramer Survivin–DNA-PKcs Complex in DNA Damage Response
43. The role of caspase-8 in the tumor microenvironment of ovarian cancer
44. The Prognostic Relevance of the Proliferation Markers Ki-67 and Plk1 in Early-Stage Ovarian Cancer Patients With Serous, Low-Grade Carcinoma Based on mRNA and Protein Expression
45. The regulation of RNA Polymerase II mediated transcription by Caspase-8 in ovarian cancer.
46. Boosting the apoptotic response of high-grade serous ovarian cancers with CCNE1-amplification to paclitaxel by targeting APC/C and the pro-survival protein MCL-1.
47. The Role of Caspase-8 in the Regulation of Transcription of Gynecological Cancers
48. Association of polo-like kinase 3 and PhosphoT273 caspase 8 levels with disease-related outcomes among cervical squamous cell carcinoma patients treated with chemoradiation and brachytherapy
49. Synthetic lethality in CCNE1-amplified high grade serous ovarian cancer through combined inhibition of Polo-like kinase 1 and microtubule dynamics
50. Association of Polo-Like Kinase 3 and PhosphoT273 Caspase 8 Levels With Disease-Related Outcomes Among Cervical Squamous Cell Carcinoma Patients Treated With Chemoradiation and Brachytherapy
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