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Your search keyword '"Nakamura, Fumio"' showing total 15 results
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15 results on '"Nakamura, Fumio"'

3. Right ventricular overloading is attenuated in monocrotaline-induced pulmonary hypertension model rats with a disrupted Gpr143 gene, the gene that encodes the 3,4-l-dihydroxyphenyalanine (l-DOPA) receptor

Author

Nakano, Masayuki, Koga, Motokazu, Hashimoto, Tatsuo, Matsushita, Natsuki, Masukawa, Daiki, Mizuno, Yusuke, Uchimura, Hiraku, Niikura, Ryo, Miyazaki, Tomoyuki, Nakamura, Fumio, Zou, Suo, Shimizu, Takahiro, Saito, Motoaki, Tamura, Kouichi, Goto, Takahisa, and Goshima, Yoshio

Published
2022
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7. Monoallelic CRMP1 gene variants cause neurodevelopmental disorder

Author

Ravindran, Ethiraj, primary, Arashiki, Nobuto, primary, Becker, Lena-Luise, primary, Takizawa, Kohtaro, primary, Lévy, Jonathan, additional, Rambaud, Thomas, additional, Makridis, Konstantin L, additional, Goshima, Yoshio, additional, Li, Na, additional, Vreeburg, Maaike, additional, Demeer, Bénédicte, additional, Dickmanns, Achim, additional, Stegmann, Alexander PA, additional, Hu, Hao, additional, Nakamura, Fumio, additional, and Kaindl, Angela M, additional

Published
2022
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8. Author response: Monoallelic CRMP1 gene variants cause neurodevelopmental disorder

Author

Ravindran, Ethiraj, primary, Arashiki, Nobuto, primary, Becker, Lena-Luise, primary, Takizawa, Kohtaro, primary, Lévy, Jonathan, additional, Rambaud, Thomas, additional, Makridis, Konstantin L, additional, Goshima, Yoshio, additional, Li, Na, additional, Vreeburg, Maaike, additional, Demeer, Bénédicte, additional, Dickmanns, Achim, additional, Stegmann, Alexander PA, additional, Hu, Hao, additional, Nakamura, Fumio, additional, and Kaindl, Angela M, additional

Published
2022
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9. Phosphorylated CRMP1, axon guidance protein, is a component of spheroids and is involved in axonal pathology in amyotrophic lateral sclerosis

Author

Kawamoto, Yuko, primary, Tada, Mikiko, additional, Asano, Tetsuya, additional, Nakamura, Haruko, additional, Jitsuki-Takahashi, Aoi, additional, Makihara, Hiroko, additional, Kubota, Shun, additional, Hashiguchi, Shunta, additional, Kunii, Misako, additional, Ohshima, Toshio, additional, Goshima, Yoshio, additional, Takeuchi, Hideyuki, additional, Doi, Hiroshi, additional, Nakamura, Fumio, additional, and Tanaka, Fumiaki, additional

Published
2022
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10. The Sympathetic Nervous System Contributes to the Establishment of Pre-Metastatic Pulmonary Microenvironments

Author

Ieguchi, Katsuaki, primary, Funakoshi, Masabumi, additional, Mishima, Taishi, additional, Takizawa, Kohtaro, additional, Omori, Tsutomu, additional, Nakamura, Fumio, additional, Watanabe, Makoto, additional, Tsuji, Mayumi, additional, Kiuchi, Yuji, additional, Kobayashi, Shinichi, additional, Tsunoda, Takuya, additional, Maru, Yoshiro, additional, and Wada, Satoshi, additional

Published
2022
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11. Inhibition of Crmp1 Phosphorylation at Ser522 Ameliorates Motor Function and Neuronal Pathology in Amyotrophic Lateral Sclerosis Model Mice

Author

Asano, Tetsuya, primary, Nakamura, Haruko, additional, Kawamoto, Yuko, additional, Tada, Mikiko, additional, Kimura, Yayoi, additional, Takano, Hiroshi, additional, Yao, Ryoji, additional, Saito, Hiroya, additional, Ikeda, Takuya, additional, Komiya, Hiroyasu, additional, Kubota, Shun, additional, Hashiguchi, Shunta, additional, Takahashi, Keita, additional, Kunii, Misako, additional, Tanaka, Kenichi, additional, Goshima, Yoshio, additional, Nakamura, Fumio, additional, Takeuchi, Hideyuki, additional, Doi, Hiroshi, additional, and Tanaka, Fumiaki, additional

Published
2022
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15. Exploring the Bio-Functional Breaking Point of Living Tissue Subjected to External Physical Pressure.

Author

Tanaka, Shotaro and Nakamura, Fumio

Subjects
*GENE silencing, *SYNTHETIC genes, *CELL anatomy, *GENE expression, *EXTRACELLULAR matrix
Abstract

Long before reaching its mechanical breaking point, a bio-system begins responding to stress at its own "bio-functional breaking point," a phase of life activity dysfunction. However, little is known about the correlation between tissue flexibility and the conditions under which cellular response, damage, and death occur. We are now developing a new confocal microscopy-based observation method to analyze cell aggregates (spheroids) that are under physical pressure. The method concomitantly assesses cellular responses, stress levels, and cellular structure changes. Using this method, we found that the artificial suppression of the gene expression of fibronectin, a major component of the extracellular matrix, provides different mechanical characteristics to hepatoma-derived cell line spheroids than does the control wild type. This study may aid in the prediction of the characteristics of a tissue of interest by simply analyzing the tissue gene expression pattern, providing valuable information for the development and operation of wearable devices. It may also help in the preparation of custom devices that suit specific individuals. [ABSTRACT FROM AUTHOR]

Published
2022
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