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Between the end of March and June 2020, COVID-19 quickly spread in Mexico. As a response, the Mexican government announced its national prevention campaign, called 'Susana Distancia,' a set of measures, recommendations, conferences, educational resources, and a chatbot. At the centre of the campaign was Susana Distancia's character, a young woman designed in a comic-book-style, echoing the Mexican lucha libre heroes, who recommended keeping 1.5 m (6 feet) of physical distance to prevent contagion. In the following visual essay, we analyse Susana Distancia's role, from its institutional origins to its later transformation as a popular culture meme. Based on the Latin American theory of the popular, we argue that the Susana Distancia, as a meme, used various mechanisms to support and challenge the institutional discourse. In this sense, Susana Distancia can be considered a meme that embraces the popular: a cultural artefact that travels across different languages, spaces, and rituals, from cyberspace to the streets, from governmental intentions to everyday performativity. When we use lo popular as a category, we refer to the possibility of enacting subaltern narratives in aesthetic and ethical terms. Multiple readings and manifestations of Susana Distancia demonstrate popular culture as a means of travelling through diverse semantic appropriations. Various forms of popular culture: the politised, the artistic, the erotic, the authentic, the mainstream, and the political, converge in the translation of meaning, from the institutional discourse to the lived experiences of the Mexican population. [ABSTRACT FROM AUTHOR]

Copyright of Revista de Enfermedades Infecciosas en Pediatría is the property of Ediciones Franco S.A. de C.V. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Revista de Enfermedades Infecciosas en Pediatría is the property of Ediciones Franco S.A. de C.V. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Smoking prevalence in schizophrenia is considerably larger than in general population, playing an important role in early mortality. We compared the polygenic contribution to smoking in schizophrenic patients and controls to assess if genetic factors may explain the different prevalence. Polygenic risk scores (PRSs) for smoking initiation and four genetically correlated traits were calculated in 1108 schizophrenic patients (64.4% smokers) and 1584 controls (31.1% smokers). PRSs for smoking initiation, educational attainment, body mass index and age at first birth were associated with smoking in patients and controls, explaining a similar percentage of variance in both groups. Attention‐deficit hyperactivity disorder (ADHD) PRS was associated with smoking only in schizophrenia. This association remained significant after adjustment by psychiatric cross‐disorder PRS. A PRS combining all the traits was more explanative than smoking initiation PRS alone, indicating that genetic susceptibility to the other traits plays an additional role in smoking behaviour. Smoking initiation PRS was also associated with schizophrenia in the whole sample, but the significance was lost after adjustment for smoking status. This same pattern was observed in the analysis of specific SNPs at the CHRNA5–CHRNA3–CHRNB4 cluster associated with both traits. Overall, the results indicate that the same genetic factors are involved in smoking susceptibility in schizophrenia and in general population and are compatible with smoking acting, directly or indirectly, as a risk factor for schizophrenia that contributes to the high prevalence of smoking in these patients. The contrasting results for ADHD PRS may be related to higher ADHD symptomatology in schizophrenic patients. [ABSTRACT FROM AUTHOR]