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Copyright of China Tropical Medicine is the property of China Tropical Medicine Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Food Research & Development is the property of Food Research & Development Editorial Department and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Telecommunication Engineering is the property of Telecommunication Engineering and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of West China Journal of Stomatology is the property of Sichuan University, West China College of Stomatology and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Modern Preventive Medicine is the property of Modern Preventive Medicine Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Journal of Chengdu University (Social Science) is the property of Journal of Chengdu University (Social Science Edition) Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Science Technology & Industry / Keji he Chanye is the property of Chinese Society of Technology Economics and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

The green-flesh (gf) mutant of the tomato fruit ripen to a muddy brown color and has been demonstrated previously to be a loss-of-function mutant. Here, we provide more evidence to support this view that SlSGR1 is involved in color change in ripening tomato fruits. Knocking out SlSGR1 expression using a clustered regularly interspaced short palindromic repeats (CRISPR)/CRISPR-associated protein 9 genome editing strategy showed obviously a muddy brown color with significantly higher chlorophyll and carotenoid content compared with wild-type (WT) fruits. To further verify the role of SlSGR1 in fruit color change, we performed transcriptome deep sequencing (RNA-seq) analysis, where a total of 354 differentially expressed genes (124/230 downregulated/upregulated) were identified between WT and slsgr1. Additionally, the expression of numerous genes associated with photosynthesis and chloroplast function changed significantly when SlSGR1 was knocked out. Taken together, these results indicate that SlSGR1 is involved in color change in ripening fruit via chlorophyll degradation and carotenoid biosynthesis. [ABSTRACT FROM AUTHOR]

Copyright of Architectural Journal / Jian Zhu Xue Bao is the property of Architectural Journal Editorial Office and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Shandong Medical Journal is the property of Shandong Medical Health Newspapers and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Food Research & Development is the property of Food Research & Development Editorial Department and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Shandong Medical Journal is the property of Shandong Medical Health Newspapers and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

The micromorphological characteristics of leaf epidemis of 30 Rosa L. species from China was observed by using light microscope and scanning electron microscope(SEM). Both the adaxial and abaxial epidermal cells were polygonal or irregular in shape with anticlinal walls undulate or straight and arched. The size of leaf epidermal cells has greater interspecific difference in general. The upper epidermal cells of the genus were almost all polygonal and the upper epidermal cells were slightly larger than the lower epidermal cells. Periclinal walls of most epidermal cells of the genus were sunken and some fraction of epidermal cells were bulging under the scanning electron microscope. All 29 plants of the subgen. Rosa L. were hypostomatic except for amphistomatic R. berberifolia belonging to subgen. Hulthemia. Most stomatal types were anomocytic with some others anomotetracytic or paratetracytic as well as stomatal shape elliptic, widely elliptic or oblong elliptic. Inner margin of outer stomatal rim were nearly smooth or sinuolate, or with sinuous and sinuate, besides, ornamentation of outer stomatal rim were smooth or nearly smooth or with scaly waxy layer with most stomata apparatus “T” type thickening of guard cells poles. Two kinds of trichomes were distinguished in this study. They were pubescence or glandular in Rosa L., and most species had long pubescence or short pubescence, as well a small number of plants were glabrous. Bases of most single pubescences had no specialization and were surrounded by ordinary epidermal cells. Only a very small group of species were surrounded by specialized radial cells. The cuticular wax ornamentation were nearly smooth, papillose or granulate, or with layer of small processes and flake-like waxy layer. All the observed species of the subgen. Rosa L. were unilaminar outer stomatal rim except for R. berberifolia belonging to subgen. Hulthemia and that R. berberifolia also had trichomes in just the upper epidermis. So R. berberifolia belonged to subgen. Hulthemia had a special position in evolution, which was also explained by the research results of this paper. There were no clear regularity in the micro morphological characteristics of the leaf epidermis of Rosa L. in each group within the genus, but it can provide the basis for systematic taxonomy and phylogenetic relationships between species, and leaf epidermis characters can be used to differentiate similar species that were easily confused with each other. The significance of these epidermal features was discussed. [ABSTRACT FROM AUTHOR]

目的探讨强脉冲光（IPL）联合射频治疗对黄褐斑的疗效及安全性。方法选取2013年12月至2014 年8月该院收治的黄褐斑患者133例，同时筛选出混合型患者，排除使用其他相关措施治疗的黄褐斑后80例女性患者纳 入该研究。将纳入的80例患者随机分为IPL联合射频组、IPL治疗组、射频治疗组和空白组，各20例。对各组采用IPL治 疗仪⁃M22，具有完美脉冲技术，并联合使用深蓝射频治疗，观察各组疗效。结果IPL联合射频组治疗总有效率[95.0% （19/20）]明显高于IPL治疗组[50.0%（10/20）]，差异有统计学意义（P<0.05）；IPL联合射频治疗组治疗后皮肤黑素指数、皮 肤红斑指数检测结果（114.13±50.17、125.04±49.53）均明显低于治疗前（153.01±43.17、197.07±45.23），差异均有统计学意 义（P<0.05）；1例患者出现原黄褐斑区域色素沉着加深。结论IPL联合射频治疗黄褐斑安全有效，值得推广应用。 [ABSTRACT FROM AUTHOR]

目的观察青蒿琥酯对人肺腺癌细胞系A549增殖的抑制作用,并探讨其作用机制。方法常规培养A549细胞后,不同浓度(0、25、50、75、100μmol/L)的青蒿琥酯作用A549细胞,采用CCK-8法检测青蒿琥酯对A549细胞的增殖抑制率,Western blotting法检测m TOR信号通路关键分子和自噬相关蛋白p62、LC3B表达变化。结果25μmol/L及以上浓度的青蒿琥酯可抑制A549细胞增殖,并呈时间和浓度依赖性(P均<0.05)。不同浓度(0、25、50、75、100μmol/L)的青蒿琥酯作用A549细胞48 h可降低p62表达,增加LC3B表达,并降低m TOR信号通路关键分子的表达,并呈剂量依赖性。青蒿琥酯联合自噬抑制剂3-MA可增加A549细胞的生存率(P<0.05)。结论青蒿琥酯可能通过下调m TOR信号通路关键分子表达、诱导A549细胞发生自噬从而抑制A549细胞增殖。 [ABSTRACT FROM AUTHOR]