16 results on '"Gao, Zizheng"'
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2. Dasatinib causes keratinocyte apoptosis via inhibiting high mobility group Box 1-mediated mitophagy
3. Genome-wide DNA methylation analysis reveals layer-specific methylation patterns in deer antler tissue
4. Bisdemethoxycurcumin alleviates vandetanib-induced cutaneous toxicity in vivo and in vitro through autophagy activation
5. High-mobility group box 1 protein-mediated necroptosis contributes to dasatinib-induced cardiotoxicity
6. Inhibition of PRKAA/AMPK (Ser485/491) phosphorylation by crizotinib induces cardiotoxicity via perturbing autophagosome-lysosome fusion.
7. TEMPORARY REMOVAL: Crizotinib induces pulmonary toxicity by blocking autophagy flux in alveolar epithelial cells
8. JAK-STAT signaling as an ARDS therapeutic target: Status and future trends
9. PTX3 from vascular endothelial cells contributes to trastuzumab-induced cardiac complications
10. JAK-STAT signaling as an ARDS therapeutic target: status and future trends
11. Structure-Based Rational Design Enables Discovery of a New Selective and Potent Akt Degrader with Improved Dermatologic Safety
12. Understanding the Needs and Concerns of Communities Affected by Hate in Hamilton
13. Autophagic degradation of CCN2 (cellular communication network factor 2) causes cardiotoxicity of sunitinib
14. Discovery of N-((3S,4S)-4-(3,4-Difluorophenyl)piperidin-3-yl)-2-fluoro-4-(1-methyl-1H-pyrazol-5-yl)benzamide (Hu7691), a Potent and Selective Akt Inhibitor That Enables Decrease of Cutaneous Toxicity
15. Autophagic degradation of CCN2 (cellular communication network factor 2) causes cardiotoxicity of sunitinib.
16. Discovery of N -((3 S ,4 S )-4-(3,4-Difluorophenyl)piperidin-3-yl)-2-fluoro-4-(1-methyl-1 H -pyrazol-5-yl)benzamide (Hu7691), a Potent and Selective Akt Inhibitor That Enables Decrease of Cutaneous Toxicity.
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