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Your search keyword '"Van Hooven, D"' showing total 5 results
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5 results on '"Van Hooven, D"'

1. Longterm outcome after Ulner Tunnel Release

Author

Westenberg, RF, Chen, NC, and Van Hooven, D

Subjects
musculoskeletal diseases, body regions, ulnar tunnel decompression surgery, ddc: 610, musculoskeletal, neural, and ocular physiology, ulnar nerve, 610 Medical sciences, Medicine, musculoskeletal system, guyon's canal syndrome
Abstract

Objectives/Interrogation: Ulnar tunnel syndrome (UTS) is a rare nerve compression syndrome with a variety of causative factors. The aims of this study are to describe the distribution of causes of ulnar tunnel syndrome, the rate of revision, and to evaluate the long-term outcome of ulnar tunnel[for full text, please go to the a.m. URL], 14th Triennial Congress of the International Federation of Societies for Surgery of the Hand (IFSSH), 11th Triennial Congress of the International Federation of Societies for Hand Therapy (IFSHT), 11th Triennial Congress of the International Federation of Societies for Hand Therapy (IFSHT)

Published
2020
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2. Outcome of acute staphylococcal prosthetic joint infection treated with debridement, implant retention and antimicrobial treatment with short duration of rifampicin

Author

van der Beek, M., van Hooven, D., Leo G. Visser, Rob G H H Nelissen, van de Sande, M., H. Scheper, de Boer, M., and van der Wal, R.

Subjects
0301 basic medicine, Microbiology (medical), medicine.medical_specialty, business.industry, medicine.medical_treatment, 030106 microbiology, Prosthetic joint infection, Antimicrobial, Surgery, 03 medical and health sciences, 0302 clinical medicine, Infectious Diseases, Debridement (dental), Medicine, 030212 general & internal medicine, Implant, business, Short duration, Rifampicin, medicine.drug
Published
2018

3. Veno-arterial thrombosis and microcirculation imaging in a patient with COVID-19.

Author

Akin S, van Hooven D, Ince C, and Jansen T

Abstract

The Coronavirus pandemic has brought new challenges in intensive care medicine. Understanding of the pathophysiology of the vascular complications of SARS-CoV-2 infection could bring new resuscitation and therapeutic options. In this case report, we present a patient with COVID-19 pneumonia, who was admitted to our ICU and treated with high-flow nasal cannula, dexamethasone, remdesivir and high-dose prophylactic low molecular weight heparin. During ICU admission, substantial venous and arterial thrombosis developed. Meanwhile the microcirculation showed more than double amount of organ perfusion with very high total vessel density. We hypothesize that this might be a compensatory mechanism for the generalized prothrombic state in which the microcirculation increases the oxygen extraction capacity preventing multi-organ failure., Competing Interests: Dr. Ince has developed SDF imaging and is listed as an inventor on related patents that were commercialized by Micro Vision Medical (MVM) under a license from the Academic Medical Center (AMC). He receives no royalties or benefits from this license. He has been a consultant for MVM in the past but has not been involved with this company for more than five years and holds no shares or stock. Braedius Medical, which is a company that is owned by a relative of Dr. Ince, has developed and designed a handheld microscope, namely, the CytoCam-IDF imaging microscope. Dr. Ince has no financial relationship with Braedius Medical of any sort (he has never owned shares or received consultancy or speaker fees from Braedius Medical). Dr. Ince runs an internet site (https://.microcirculationacademy.org) that offers services (e.g., training, courses, and analysis) that are related to clinical microcirculation. The other authors declare that they have no competing interests., (© 2021 The Authors.)

Published
2021
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5. Genetic variants in ANCA-associated vasculitis: a meta-analysis.

Author

Rahmattulla C, Mooyaart AL, van Hooven D, Schoones JW, Bruijn JA, Dekkers OM, and Bajema IM

Subjects
Case-Control Studies, DNA-Binding Proteins genetics, Genome-Wide Association Study, Histocompatibility Antigens Class I genetics, Humans, Major Histocompatibility Complex genetics, Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis genetics, Genetic Variation
Abstract

Background: Genetic factors may influence the pathogenic pathways leading to antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV). We performed a meta-analysis to determine the genetic variants most likely associated with AAV and investigated whether diagnostic and serological subtypes within AAV have distinct genetic backgrounds., Methods: Studies investigating the association between genetic variants and AAV in humans were searched in PubMed, EMBASE and Web of Science. All variants investigated in at least two studies were selected. Subsequently, all studies assessing these variants were included in this meta-analysis. Additionally, data on these variants from the largest genome-wide association studies in AAV were included to increase the validity of this meta-analysis., Results: The literature search yielded 5180 articles. 62 articles investigating 140 genetic variants were included, 33 of which were associated with AAV in a meta-analysis. These genetic variants were in or near the following genes: CD226, CTLA-4, FCGR2A, HLA-B, HLA-DP, HLA-DQ, HLA-DR, HSD17B8, IRF5, PTPN22, RING1/RXRB, RXRB, STAT4, SERPINA1 and TLR9. Moreover, we identified genetic distinctions between granulomatosis with polyangiitis and microscopic polyangiitis and between proteinase 3 ANCA vasculitis and myeloperoxidase ANCA vasculitis. In 76% of the genetic variants, subdivision based on ANCA serotype resulted in higher ORs than subdivision based on clinical diagnosis., Conclusions: This meta-analysis identified 33 genetic variants associated with AAV, supporting a role for alpha-1-antitrypsin, the major histocompatibility complex system, and several distinct inflammatory processes in AAV pathogenesis. Our results indicate that subdivision of AAV based on ANCA serotype has a stronger genetic basis than subdivision based on clinical diagnosis., (Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/)

Published
2016
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