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3. International Consensus on the Use of Genetics in the Management of Hereditary Angioedema

7. Complement genetic variants and FH desialylation in S. pneumoniae-Haemolytic Uraemic Syndrome

11. International consensus on the diagnosis and management of pediatric patients with hereditary angioedema with C1 inhibitor deficiency

12. FHR-5 Serum Levels and CFHR5 Genetic Variations in Patients With Immune Complex-Mediated Membranoproliferative Glomerulonephritis and C3-Glomerulopathy

13. A Novel Homozygous In-Frame Deletion in Complement Factor 3 Underlies Early-Onset Autosomal Recessive Atypical Hemolytic Uremic Syndrome - Case Report

14. Complement Genetic Variants and FH Desialylation in S. pneumoniae-Haemolytic Uraemic Syndrome

15. Patterns of C1-Inhibitor/Plasma Serine Protease Complexes in Healthy Humans and in Hereditary Angioedema Patients

17. CFHR5 Genetic Variations and Serum Levels in Patients with Immune-Complex-Mediated Membranoproliferative Glomerulonephritis and C3-Glomerulopathy

18. Patterns of C1-Inhibitor/Plasma Serine Protease Complexes in Healthy Humans and in Hereditary Angioedema Patients

21. Hereditary angioedema attack: what happens to vasoactive mediators?

25. Secreted Phospholipases A2 in Hereditary Angioedema With C1-Inhibitor Deficiency

36. Secreted Phospholipases A2 in Hereditary Angioedema With C1-Inhibitor Deficiency.

40. A systematic analysis of the complement pathways in patients with neuromyelitis optica indicates alteration but no activation during remission

41. Complement levels and complement activation in neuromyelitis optica

44. Complement Genetic Variants and FH Desialylation in S. pneumoniae-Haemolytic Uraemic Syndrome

45. Secreted Phospholipases A2 in Hereditary Angioedema With C1-Inhibitor Deficiency

46. [Analysis of the relationship between vitamin D 3 level and disease severity in hereditary angioedema].

47. Secreted Phospholipases A 2 in Hereditary Angioedema With C1-Inhibitor Deficiency.

48. [Effect of danazol treatment on growth in pediatric patients with hereditary angioedema due to C1-inhibitor deficiency].

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