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Your search keyword '"H, Ohnishi"' showing total 9 results
Start Over Author "H, Ohnishi" Publication Year Range More than 50 years ago
9 results on '"H, Ohnishi"'

3. Ohnishi's Super Carding Engine

Author

H. Ohnishi and I. Hayashi

Subjects
Mechanical engineering, Carding, Mathematics
Abstract

研究目的従来のフラット式梳綿機とローラ式梳綿機とを組合せて新しい梳綿機構を発案しその開纎能力の増大により生産能力を飛躍的に増加及び品質の向上をなさんとしたものであろ.研究結果木O.S.梳綿機にて実際に紡出試験の結果, 次の諸事項が明らかとなった.1.梳綿機の生産量増加 (ドッファの回転を150%に上昇) による品質の低下は示さない.普通梳綿機のドッファ回転数7.5 r.p.m.とO.S.梳綿機の12r.p.m.とほぼ同一のネップ数となる.2.並掃除後時間の経過とともに針布中に堆積する綿量 (並掃除綿量) は普通梳綿機に比し少なく, O.S.梳綿機の4hr経過時の並掃除綿量と普通梳綿機の2hr経過のそれとほぼ同一であろ.又時間の経過にしたがってネップの増加すろ割合は普通梳綿機に比して少ない.3.トップ針布の種滓の除去能力は普通梳綿機に比して甚だしく増加する.4.鎧綿は短纎維及び種滓のみとなり落綿量が減少して良質綿はよく回収利用せられる.5.ウォーカローラ及びストリッパローラ使用により供給ラップの斑を矯正する能力が増大し, 糸の不同率を減少せしめろ.6.O.S.梳綿機の使用により加工費, 原料費の大なる節減をなし得, 建設又は改造の場合は設備費.金利償却費を減ずろことが出来ろ.

Published
1954
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4. [Pharmacology of cornary dilator agent, trapymin. (2) Analysis of its mode of action]

Author

H, Ohnishi, S, Tsukuda, K, Yamaguchi, and N, Ogawa

Subjects
Blood Glucose, Male, Epinephrine, Platelet Aggregation, Vasopressins, Vasodilator Agents, Myocardial Infarction, Dopamine beta-Hydroxylase, In Vitro Techniques, Calcium Chloride, Adenosine Triphosphate, Catecholamines, Coronary Circulation, Animals, Monoamine Oxidase, Myocardium, Isoproterenol, Arrhythmias, Cardiac, Myocardial Contraction, Propranolol, Rats, Trapidil, Pyrimidines, Cats, Cattle, Female, Rabbits, Glycogen
Abstract

Trapymin (TM) relaxed excised renal, coronary, pulmonary, femoral and mesenteric arteries and this relaxation was not antagonized by propranolol. The dose-response curve of TM was parallel to that of nitroglycerin and papaverine and steeper than that of dipyridamol or adenosine. TM exerted inotropic and chronotropic actions on excised rat atrium. TM was also effective through the oral route and the effectiveness tended to decrease slightly after repeated use for ten days. TM was effective on vasopressin induced angina in rats and electrocoagulation-induced myocardial infarction. TM suppressed adrenaline-induced arrhythmia but not CaCl2-induced arrhythmia. TM reduced catecholamine content in brain, adrenals and heart but had no influence on monoamine oxidase or dopamine-beta-hydroxylase. TM revealed ganglion-blocking and neuron-blocking actions in cervical ganglion in cats. With propranolol, TM-induced hyperglycemia and reduction in glycogen content in liver and heart was antagonized but TM-induced rise in free fatty acid in serum was not antagonized. Na+-K+ dependent ATPase of bovine heart and P/O ratio of mitochondria of rat heart was not influenced by TM. ADP-induced aggregation of platelets was antagonized by TM. These data indicate that TM induced coronary dilation is partly due to a papaverine like action and also to ganglion-blocking, neuron-blocking and anti-adrenergic action. On the other hand, TM possessed catecholamine release and cardiotonic action as related to beta-receptors.

Published
1975

7. [Pharmacology of cornary dilator agent, trapymin. (2) Analysis of its mode of action].

Author

Ohnishi H, Tsukuda S, Yamaguchi K, and Ogawa N

Subjects
Adenosine Triphosphate metabolism, Animals, Arrhythmias, Cardiac chemically induced, Blood Glucose metabolism, Calcium Chloride, Catecholamines metabolism, Cats, Cattle, Coronary Circulation drug effects, Dopamine beta-Hydroxylase metabolism, Epinephrine, Female, Glycogen metabolism, In Vitro Techniques, Isoproterenol pharmacology, Male, Monoamine Oxidase metabolism, Myocardial Contraction drug effects, Myocardial Infarction metabolism, Myocardium metabolism, Platelet Aggregation drug effects, Propranolol pharmacology, Rabbits, Rats, Vasopressins pharmacology, Pyrimidines pharmacology, Trapidil pharmacology, Vasodilator Agents pharmacology
Abstract

Trapymin (TM) relaxed excised renal, coronary, pulmonary, femoral and mesenteric arteries and this relaxation was not antagonized by propranolol. The dose-response curve of TM was parallel to that of nitroglycerin and papaverine and steeper than that of dipyridamol or adenosine. TM exerted inotropic and chronotropic actions on excised rat atrium. TM was also effective through the oral route and the effectiveness tended to decrease slightly after repeated use for ten days. TM was effective on vasopressin induced angina in rats and electrocoagulation-induced myocardial infarction. TM suppressed adrenaline-induced arrhythmia but not CaCl2-induced arrhythmia. TM reduced catecholamine content in brain, adrenals and heart but had no influence on monoamine oxidase or dopamine-beta-hydroxylase. TM revealed ganglion-blocking and neuron-blocking actions in cervical ganglion in cats. With propranolol, TM-induced hyperglycemia and reduction in glycogen content in liver and heart was antagonized but TM-induced rise in free fatty acid in serum was not antagonized. Na+-K+ dependent ATPase of bovine heart and P/O ratio of mitochondria of rat heart was not influenced by TM. ADP-induced aggregation of platelets was antagonized by TM. These data indicate that TM induced coronary dilation is partly due to a papaverine like action and also to ganglion-blocking, neuron-blocking and anti-adrenergic action. On the other hand, TM possessed catecholamine release and cardiotonic action as related to beta-receptors.

Published
1975
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