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This article undertakes both theoretical and experimental investigation on the heat and mass transfer properties of wet air and solution (configured sodium hydroxide solution with the concentration of 2%) on the surface of corrugated packings under reduced ambient pressure conditions. Theoretical analysis reveals that a decreased atmospheric pressure environment is conducive to amplifying mass transfer potential difference. However, it concurrently mitigates the maximum possible mass of water vapor per unit volume of air. Subsequently, experiments were carried out under the following conditions: vacuum degree-0 ~ 40 kPa, inlet air velocity-0.8 ~ 1.6 m/s, inlet air temperature-12.0 ~ 22.0 °C, and inlet air relative humidity-50.0 ~ 95.0%. The experimental outcomes indicate that heat transfer coefficient is predominantly influenced by inlet air velocity and vacuum degree. An enhancement of inlet air velocity or a reduction of vacuum degree leads to a corresponding increase of heat transfer coefficient. Under consistent conditions for other parameters, as vacuum degree ascends, moisture absorption rate and efficiency exhibit a steady growth, albeit with a diminishing rate of increment. Under constant inlet air velocity and relative humidity conditions, mass transfer coefficient initially experiences an upsurge as vacuum degree rises, but eventually undergoes a decline. At a fixed inlet air temperature, mass transfer coefficient behaves differently depending on air temperature. It demonstrates a gradual decrease under cooler air temperatures as vacuum degree rises, whereas under warmer air temperatures, it initially increases and then decreases. The analysis reveals that elevating vacuum degree indeed augments mass transfer capability between wet air and solution. However, there exists an upper limit to this enhancement, dictated by the maximum amount of water vapor that wet air can bear upon reaching saturation. [ABSTRACT FROM AUTHOR]

Parkinson's disease (PD) is a progressive neurodegenerative disorder. Long non-coding RNA (lncRNA) XIST has been implicated in the development of PD. Herein, we sought to investigate the diagnostic value of serum XIST expression in PD. We recorded clinical baseline data, including age, sex, body mass index (BMI), years of education, and PD family history, for PD patients and healthy controls, in addition to the disease course in PD patients. The disease severity of PD patients was assessed using the Hoehn-Yahr grading scale. We evaluated serum XIST expression levels in PD patients and healthy controls using RT-qPCR and assessed the correlation between serum XIST expression and the Unified Parkinson's Disease Rating Scale (UPDRS) score. Additionally, we used receiver operating characteristic (ROC) curve to determine the diagnostic value of XIST expression for PD. Our results showed no significant differences in age, sex, BMI, and years of education between PD patients and healthy controls. However, more PD patients had a family history of PD. We found that serum XIST expression was significantly up-regulated in PD patients, and its levels were positively correlated with the UPDRS score. Higher PD stage was also associated with increased serum XIST expression. ROC analysis revealed that XIST expression had a diagnostic value for PD. In conclusion, our findings suggest that serum lncRNA XIST expression may serve as a potential diagnostic biomarker for PD. [ABSTRACT FROM AUTHOR]