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In the photothermal synergistic catalytic conversion of CO2 and H2O, the catalyst harnesses solar energy to accumulate heat, thereby elevating the reaction system's temperature. The influence of this temperature effect on surface chemical reactions remains an underexplored area. Here the impact of temperature on the surface-level thermodynamic reactions and conversion of CO2 with H2O on oxide semiconductors at the atomic scale was investigated using first-principle calculations. 13 different metal oxides and 5 transition metal clusters were used to introduce surface functional sites on the TiO2 supporting catalyst. The potential metal oxide cocatalysts that could be most beneficial to the following conversion of CO2 by H2O were initially screened by calculating the degrees of promotion of CO2 adsorption and activation of surface H to provide protons. The proton donation and hydrogen evolution difficulty from H2O were further analyzed, identifying transition metal cocatalysts that promote direct CO2 hydrogenation. Upon introducing bifunctional sites to facilitate adsorption and reduction, the production of CH3OH and CH4 could be further enhanced through the facilitation of the proton donation process of H2O. The results of Gibbs free-energy calculations revealed that increasing temperature enhances the reaction thermodynamics for each C1 product formation at different surface sites to varying degrees. These findings offer valuable theoretical insights for designing and regulating active sites on oxide semiconductor surfaces for efficient photothermal catalytic CO2 reduction by H2O. [ABSTRACT FROM AUTHOR]

New media technologies have opened up new possibilities for the communication of cultural heritage, and digital reading aligns with the participatory shift in museums, becoming an indispensable part of information communication. Currently, there exists a certain contradiction between the professional content of cultural heritage and the non-linear and immersive narrative style of new media. In order to find a balance between form and content, and explore the information communication pattern of cultural heritage in the era of mobile media, starting from practical issues, this article introduces the theory of embodied cognition, combines it with relevant theories in communication studies, and systematically explores the impact of embodied factors on audience reading behavior by using semi-structured interviews and grounded theory. The research reveals that embodied cognition, situational cognition, and interactive cognition have a significant impact on the digital reading information communication of cultural heritage on social media, which includes six factors: functional experience, sensory experience, interactive experience, scene simulation, emotional elevation and social experience. This study further constructed a theoretical model of the factors influencing the effectiveness of information communication in cultural heritage digital reading on the social media platform under the influence of embodied cognition theory, which confirms that these six factors play a significant role in the process of museum content generation, audience understanding, as well as the emergence of and the feedback loop of new meanings. [ABSTRACT FROM AUTHOR]

As one of the emerging two-dimensional lead halide materials, lead iodide (PbI2) nanosheets have proven to possess strong application potential in the fields of high-energy radiation detection and highly efficient perovskite solar cells. However, the underlying photophysical properties such as hot-exciton-related carrier dynamics remain unclear for PbI2 nanosheets. Here, we report the exciton dynamics of a single PbI2 nanoflake prepared by an aqueous solution method. Through a three-dimensional (3D) diffusion model, we obtain the exciton annihilation radius and diffusion coefficient of a single PbI2 nanoflake under non-resonant and resonant excitation conditions of band-edge exciton state. As initial exciton densities increase, we find the carrier recombination mechanism for a single PbI2 nanoflake gradually changes from exciton-exciton annihilation to free-carrier recombination. Finally, we reveal the room-temperature circular polarization of a single PbI2 nanoflake is due to free-carrier recombination with a band-edge exciton dissociation time of ~120 fs under the resonant excitation condition. [ABSTRACT FROM AUTHOR]

Background: Studies have highlighted a possible crosstalk between the pathogeneses of COVID-19 and systemic lupus erythematosus (SLE); however, the interactive mechanisms remain unclear. We aimed to elucidate the impact of COVID-19 on SLE using clinical information and the underlying mechanisms of both diseases. Methods: RNA-seq datasets were used to identify shared hub gene signatures between COVID-19 and SLE, while genome-wide association study datasets were used to delineate the interaction mechanisms of the key signaling pathways. Finally, single-cell RNA-seq datasets were used to determine the primary target cells expressing the shared hub genes and key signaling pathways. Results: COVID-19 may affect patients with SLE through hematologic involvement and exacerbated inflammatory responses. We identified 14 shared hub genes between COVID-19 and SLE that were significantly associated with interferon (IFN)-I/II. We also screened and obtained four core transcription factors related to these hub genes, confirming the regulatory role of the IFN-I/II-mediated Janus kinase/signal transducers and activators of transcription (JAK-STAT) signaling pathway on these hub genes. Further, SLE and COVID-19 can interact via IFN-I/II and IFN-I/II receptors, promoting the levels of monokines, including interleukin (IL)-6/10, tumor necrosis factor-α, and IFN-γ, and elevating the incidence rate and risk of cytokine release syndrome. Therefore, in SLE and COVID-19, both hub genes and core TFs are enriched within monocytes/macrophages. Conclusions: The interaction between SLE and COVID-19 promotes the activation of the IFN-I/II-triggered JAK-STAT signaling pathway in monocytes/macrophages. These findings provide a new direction and rationale for diagnosing and treating patients with SLE–COVID-19 comorbidity. [ABSTRACT FROM AUTHOR]