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1. Supplementary Figures S1-S5 from Combined Immunotherapy Improves Outcome for Replication-Repair-Deficient (RRD) High-Grade Glioma Failing Anti–PD-1 Monotherapy: A Report from the International RRD Consortium

2. Combined Immunotherapy Improves Outcome for Replication-Repair-Deficient (RRD) High-Grade Glioma Failing Anti–PD-1 Monotherapy: A Report from the International RRD Consortium

3. Supplementary Table 1 - Cell line information from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

4. Supplementary Figure 1 - PARP1 protein is expressed in pHGA and DIPG patient samples from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

5. Supplementary Figure 4 - Niraparib induces growth arrest in SJG2 and SF188 cells. from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

6. Supplemental Figure Legends from ATM Regulates 3-Methylpurine-DNA Glycosylase and Promotes Therapeutic Resistance to Alkylating Agents

7. Data from ATM Regulates 3-Methylpurine-DNA Glycosylase and Promotes Therapeutic Resistance to Alkylating Agents

8. Supplemental Figures 1 - 9 from ATM Regulates 3-Methylpurine-DNA Glycosylase and Promotes Therapeutic Resistance to Alkylating Agents

9. Supplementary Figure 3 - Niraparib treatment increases DNA damage and decreases proliferation. from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

10. Supplemental Methods from ATM Regulates 3-Methylpurine-DNA Glycosylase and Promotes Therapeutic Resistance to Alkylating Agents

11. Supplementary Figure Legends from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

12. Supplemental Table 1 from ATM Regulates 3-Methylpurine-DNA Glycosylase and Promotes Therapeutic Resistance to Alkylating Agents

13. Supplementary Figure 2 - Cell viability assay for pediatric brain tumour lines exposed to the indicated concentrations of Veliparib, Olaparib, or Niraparib. from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

14. Supplementary Figure 5 - Low doses of Niraparib reduce the rate of DNA repair and sensitize cells to ionizing radiation from Poly-ADP-Ribose Polymerase as a Therapeutic Target in Pediatric Diffuse Intrinsic Pontine Glioma and Pediatric High-Grade Astrocytoma

15. Supplementary File 1 from Intratumoral Genetic and Functional Heterogeneity in Pediatric Glioblastoma

16. CCR Translation for This Article from Neural Tumor-Initiating Cells Have Distinct Telomere Maintenance and Can be Safely Targeted for Telomerase Inhibition

17. Supplementary File 2 from Intratumoral Genetic and Functional Heterogeneity in Pediatric Glioblastoma

18. Supplementary Data from Neural Tumor-Initiating Cells Have Distinct Telomere Maintenance and Can be Safely Targeted for Telomerase Inhibition

19. Supplementary Data from BRAF-KIAA1549 Fusion Predicts Better Clinical Outcome in Pediatric Low-Grade Astrocytoma

20. Supplementary Table S2 from BRAF-KIAA1549 Fusion Predicts Better Clinical Outcome in Pediatric Low-Grade Astrocytoma

21. Supplementary Methods and Data from Intratumoral Genetic and Functional Heterogeneity in Pediatric Glioblastoma

22. Data from ID1 Is Critical for Tumorigenesis and Regulates Chemoresistance in Glioblastoma

23. Data from Intratumoral Genetic and Functional Heterogeneity in Pediatric Glioblastoma

24. Supplementary File 3 from Intratumoral Genetic and Functional Heterogeneity in Pediatric Glioblastoma

25. Data from Monoallelic Expression Determines Oncogenic Progression and Outcome in Benign and Malignant Brain Tumors

28. Supplementary Table 5 from Monoallelic Expression Determines Oncogenic Progression and Outcome in Benign and Malignant Brain Tumors

31. Supplementary Figure 1 from Monoallelic Expression Determines Oncogenic Progression and Outcome in Benign and Malignant Brain Tumors

37. Supplementary Table 3 from Monoallelic Expression Determines Oncogenic Progression and Outcome in Benign and Malignant Brain Tumors

44. Supplementary Table 2 from Monoallelic Expression Determines Oncogenic Progression and Outcome in Benign and Malignant Brain Tumors

48. Supplementary Figure Legend 1, Table Legends 1-5 from Monoallelic Expression Determines Oncogenic Progression and Outcome in Benign and Malignant Brain Tumors

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