25 results on '"Petermann, Eva"'
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2. Data from BRCA2 and RAD51 Promote Double-Strand Break Formation and Cell Death in Response to Gemcitabine
3. Supplementary Figure 1 from Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival
4. Supplementary Figure Legends from Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress
5. Data from BRCA2 and RAD51 Promote Double-Strand Break Formation and Cell Death in Response to Gemcitabine
6. Data from Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress
7. Data Supplement from BRCA2 and RAD51 Promote Double-Strand Break Formation and Cell Death in Response to Gemcitabine
8. Data from Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival
9. Supplementary Figures from Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress
10. Supplementary Figures from Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress
11. Data from Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival
12. Data from Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress
13. Supplementary Figure 1 from Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival
14. Data Supplement from BRCA2 and RAD51 Promote Double-Strand Break Formation and Cell Death in Response to Gemcitabine
15. Figure S1 to S7 from MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
16. Table S1 from MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
17. Data from MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
18. Table S1 from MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
19. Table S2 from MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
20. Figure S1 to S7 from MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
21. MYBL2 Supports DNA Double Strand Break Repair in Hematopoietic Stem Cells
22. Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place
23. BRCA2 and RAD51 Promote Double-Strand Break Formation and Cell Death in Response to Gemcitabine
24. Diminished Origin-Licensing Capacity Specifically Sensitizes Tumor Cells to Replication Stress
25. Targeted Mutations in the ATR Pathway Define Agent-Specific Requirements for Cancer Cell Growth and Survival
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