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Examines the specificity of panic response to carbon dioxide inhalation in panic disorder. Comparison of carbon dioxide-induced attacks between panic disorder and depressed patients; Similarity of panic rate between premenstrual dysphoric and panic disorders; High rates of anxiety and panic attacks in panic disorder.

Examines the effects of a range of instructional types on panic rates and physiological responses to carbon dioxide (CO2) inhalation. Panicogenic effects of both hyperventilation and CO2 inhalation; Subjective and biological measures of response to five percent CO2 inhalation; Results of chi-square analysis performed across the instructional programs.

Discusses the role of respiratory abnormalities in the pathophysiology of panic disorder, while focusing on a study conducted on the respiratory response of 59 patients and 39 normal persons. Method of study employed; Details on the results of the study; Statistical information supporting the results.

Determines the effects of antipanic treatment with fluoxetine on human growth hormone (GH) response to the alpha2 agonist clonidine. Human GH responses to clonidine in patients with panic disorder; Persistence of blunted secretion of human GH in despite clinical recovery.

Examines the effects of carbonic anhydrase inhibitor acetazolamide on ventilation in panic disorder patients. Biological hypersensitivity of panic disorder patients to carbon dioxide; Anxiety levels; Subjective sense of dyspnea; Stimulation of respiration; Agents that provoke panic attacks in patients.

Studies the relationship between hyperventilation and panic disorder linking carbon dioxide (CO2) hypersensitivity, cognitive/behavioral factors, and the respiratory effects of antipanic pharmacologic and psychological treatments. Respiratory findings during laboratory pharmacologic challenges; Biology and psychology of respiratory control; Respiratory control and antipanic medications.

To assess the role of peripheral epinephrine in social anxiety, the authors infused 11 patients meeting DSM-III criteria for social phobia with intravenous epinephrine over 60 minutes. Although the mean plasma epinephrine level increased from 113 to 928 pg/mI, only one of the 11 patients experienced observable anxiety; this finding suggests that an increase in plasma epinephrine level alone is inadequate to cause social anxiety. Of the variables measured, only the average minute volume correlated with self-rated anxiety. Ventilatory indexes might be better correlates of subjective anxiety than other physiological variables. [ABSTRACT FROM AUTHOR]

Reports on a study conducted on the comparative treatment of pretreatment attrition in panic disorder with treatment outcomes for imipramine and cognitive behavior therapy. Reference to data collected from clinical research sites; Identification of the main source of pretreatment attrition.

Four men with paruresis received trials of atenolol or phenelzine or both. Atenolol was effective in one patient. Three patients had a poor response to phenelzine, and they all experienced troublesome side effects. [ABSTRACT FROM AUTHOR]

Seven male panic patients did not panic but were significantly more sensitive to steady-state carbon dioxide inhalation than five male normal control subjects. The male patients' hypersensitivity to carbon dioxide was unrelated to current state of anxiety or acute panic. [ABSTRACT FROM AUTHOR]

Presents a clinical and research report on patients with panic disorder and mitral valve prolapse who underwent repeat echocardiograms after treatment for panic disorder. Method; Subjects; `DSM-III' criteria for panic disorder with or without agoraphobia; Procedure; Tricyclic antidepressant/monoamine oxidase inhibitor (MAOI); Data analysis; Results; Discussion.

A letter to the editor is presented in response to an article by Martin H. Teicher and asscoiates on depressed persons who developed violent suicidal preoccupation during fluoxetine treatment in a previously published issue.

A letter to the editor is presented which comments on the article of Roy J. Matthew about the possibility of CO2 to trigger panic attacks in patients with panic disorder.

A letter to the editor is presented in response to the article "Lifetime Prevalence of Specific Psychiatric Disorders in Three Sites," by L. N Robins and J. E Helzer.