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21 results on '"Ellonen, Pekka"'

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1. Single-Cell Functional Genomics of Natural Killer Cell Interaction with Blood Cancers

2. Somatic Mutations in T Cells in Patients with Hematological Diseases

3. Molecular features encoded in the ctDNA reveal heterogeneity and predict outcome in high-risk aggressive B-cell lymphoma

4. Integrated drug profiling and CRISPR screening identify essential pathways for CAR T-cell cytotoxicity

5. Genomic Profiling of Circulating Tumor DNA Reveals Patterns of Response and Refractoriness in Aggressive B-Cell Lymphoma - a Nordic Lymphoma Group Correlative Study

6. CRISPR Screens Identify Mechanisms of Natural Killer Cell Evasion across Blood Cancers

7. Somatic Mutations in T Cells As Possible Regulators of Immunodeficiency

10. High incidence of activating STAT5B mutations in CD4-positive T-cell large granular lymphocyte leukemia

11. Analysis of Clonal Evolution in Chemorefractory Acute Myeloid Leukemia from Diagnosis to Relapse

12. Multiple STAT3 Mutations In Different Lymphocyte Clones Of Large Granular Lymphocytic Leukemia Patients

13. Identification Of AML Subtype-Selective Drugs By Functional Ex Vivo Drug Sensitivity and Resistance Testing and Genomic Profiling

14. Novel Activating STAT5B Mutations As Drivers Of T-ALL

15. STAT3 mutations indicate the presence of subclinical T-cell clones in a subset of aplastic anemia and myelodysplastic syndrome patients

16. Discovery of somatic STAT5b mutations in large granular lymphocytic leukemia

17. Discovery of STAT5b Mutations and Small Subclones of STAT3 Mutations in Large Granular Lymphocytic (LGL) Leukemia

18. STAT3-Mutations Indicate the Presence of Subclinical Self-Reactive Cytotoxic T Cell Clones in Aplastic Anemia and Myelodysplastic Syndromes

19. Somatic PTPRT and ANGPT2 Mutations in Large Granulocyte Leukemia

20. High-Throughput Ex Vivo Drug Sensitivity and Resistance Testing (DSRT) Integrated with Deep Genomic and Molecular Profiling Reveal New Therapy Options with Targeted Drugs in Subgroups of Relapsed Chemorefractory AML

21. Recurrent Missense Mutations in the STAT3 Gene in LGL Leukemia Provide Insights to Pathogenetic Mechanisms and Suggest Potential Diagnostic and Therapeutic Applications

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