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2. Contributors

Author

Abecassis, Isaac J., primary, Abruzzo, Todd A., additional, Adel, Joseph G., additional, Aghaebrahim, Amin, additional, Aguilar-Salinas, Pedro, additional, Alimi, Marjan, additional, Aljuboori, Zaid S., additional, Andaluz, Norberto O., additional, Arthur, Adam S., additional, Babadjouni, Robin, additional, Batjer, H. Hunt, additional, Bhambri, Ankur, additional, Binning, Mandy J., additional, Boulos, Alan S., additional, Brasiliense, Leonardo B.C., additional, Brinjikji, Waleed, additional, Carroll, Christopher P., additional, Cheema, Ahmed, additional, Chen, Stephen R., additional, Cherian, Jacob, additional, Cheung, Vincent, additional, Colon, Nathan L., additional, Dalfino, John C., additional, de Oliveira Sillero, Rafael, additional, Dieppa, David S., additional, Effendi, Sabih T., additional, Elwood, Adam, additional, Fargen, Kyle M., additional, Fennell, Vernard S., additional, Fessler, Richard D., additional, Fortuny, Enzo S., additional, Galarza-Paez, Laura, additional, Reid Gooch, M., additional, Gopakumar, Sricharan, additional, Goyal, Nitin, additional, Greenberg, Karen, additional, Griessenauer, Christoph J., additional, Gupta, Vivek, additional, Hanel, Ricardo A., additional, Hamm, Carman Wayne, additional, Hedayat, Hirad S., additional, Hoit, Daniel, additional, Howington, Jay U., additional, James, Robert F., additional, Janjua, Rashid M., additional, Jimenez, Lincoln, additional, Kan, Peter, additional, Khalessi, Alexander, additional, Khan, Usman A., additional, Khattar, Nicolas K., additional, Kim, Louis J., additional, Langer, David J., additional, Lanzino, Giuseppe, additional, Levitt, Michael R., additional, Levy, Elad I., additional, Liebman, Kenneth, additional, Lodi, Yahia M., additional, Mack, William J., additional, Mascitelli, Justin, additional, Mehta, Nikhil, additional, Moore, Kenneth A., additional, Mejia Munne, Juan C., additional, Nickele, Christopher, additional, Obiora, Jeffrey C., additional, Adam Oliver, Thomas, additional, Palma, Atilio, additional, Pandey, Aditya S., additional, Pannell, Jeffrey S., additional, Patel, Maunik, additional, Pollock, Glenn, additional, Rahman, Syed A., additional, Rahme, Ralph, additional, Rennert, Robert C., additional, Ringer, Andrew J., additional, Santos, Roberta, additional, Sauvageau, Eric, additional, Savastano, Luis E., additional, Schirmer, Clemens M., additional, Serrone, Joseph C., additional, Siddiqui, Adnan H., additional, Singh, Jasmeet, additional, Spiotta, Alejandro M., additional, Srinivasan, Visish M., additional, Starke, Robert, additional, Steinberg, Jeffrey A., additional, Stofko, Douglas L., additional, Sudhakar, Tejaswi D., additional, Tackla, Ryan D., additional, Thomas, Justin G., additional, Turner, Raymond D., additional, Vargas, Jan, additional, Veznedaroglu, Erol, additional, Vibhute, Prasanna, additional, Vuong, Shawn M., additional, Walcott, Brian P., additional, Wali, Arvin R., additional, Welch, Babu G., additional, White, Andrew C., additional, White, Jonathan A., additional, White, Timothy G., additional, Wicks, Robert T., additional, Andrew Wilkinson, David, additional, Wilson, John A., additional, Wolfe, Stacey Q., additional, Yamamoto, Junichi, additional, Yim, Benjamin, additional, and Young, Christopher C., additional

Published
2018
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4. Contributors

Author

Isaac J. Abecassis, Todd A. Abruzzo, Joseph G. Adel, Amin Aghaebrahim, Pedro Aguilar-Salinas, Marjan Alimi, Zaid S. Aljuboori, Norberto O. Andaluz, Adam S. Arthur, Robin Babadjouni, H. Hunt Batjer, Ankur Bhambri, Mandy J. Binning, Alan S. Boulos, Leonardo B.C. Brasiliense, Waleed Brinjikji, Christopher P. Carroll, Ahmed Cheema, Stephen R. Chen, Jacob Cherian, Vincent Cheung, Nathan L. Colon, John C. Dalfino, Rafael de Oliveira Sillero, David S. Dieppa, Sabih T. Effendi, Adam Elwood, Kyle M. Fargen, Vernard S. Fennell, Richard D. Fessler, Enzo S. Fortuny, Laura Galarza-Paez, M. Reid Gooch, Sricharan Gopakumar, Nitin Goyal, Karen Greenberg, Christoph J. Griessenauer, Vivek Gupta, Ricardo A. Hanel, Carman Wayne Hamm, Hirad S. Hedayat, Daniel Hoit, Jay U. Howington, Robert F. James, Rashid M. Janjua, Lincoln Jimenez, Peter Kan, Alexander Khalessi, Usman A. Khan, Nicolas K. Khattar, Louis J. Kim, David J. Langer, Giuseppe Lanzino, Michael R. Levitt, Elad I. Levy, Kenneth Liebman, Yahia M. Lodi, William J. Mack, Justin Mascitelli, Nikhil Mehta, Kenneth A. Moore, Juan C. Mejia Munne, Christopher Nickele, Jeffrey C. Obiora, Thomas Adam Oliver, Atilio Palma, Aditya S. Pandey, Jeffrey S. Pannell, Maunik Patel, Glenn Pollock, Syed A. Rahman, Ralph Rahme, Robert C. Rennert, Andrew J. Ringer, Roberta Santos, Eric Sauvageau, Luis E. Savastano, Clemens M. Schirmer, Joseph C. Serrone, Adnan H. Siddiqui, Jasmeet Singh, Alejandro M. Spiotta, Visish M. Srinivasan, Robert Starke, Jeffrey A. Steinberg, Douglas L. Stofko, Tejaswi D. Sudhakar, Ryan D. Tackla, Justin G. Thomas, Raymond D. Turner, Jan Vargas, Erol Veznedaroglu, Prasanna Vibhute, Shawn M. Vuong, Brian P. Walcott, Arvin R. Wali, Babu G. Welch, Andrew C. White, Jonathan A. White, Timothy G. White, Robert T. Wicks, David Andrew Wilkinson, John A. Wilson, Stacey Q. Wolfe, Junichi Yamamoto, Benjamin Yim, and Christopher C. Young

Published
2018

5. Public-sector health-care reforms that work?: a case study of the US Veterans Health Administration

Author

Adam Oliver

Subjects
business.industry, E151 United States (General), education, Public sector, Original Articles, General Medicine, Public administration, Popular press, National health service, Veterans health, humanities, Work (electrical), Environmental health, Health care, RA Public aspects of medicine, Medicine, business, Administration (government), health care economics and organizations
Abstract

The Veterans Health Administration (VHA) provides health care for U.S. military veterans. By the early 1990s, the VHA had a reputation for delivering limited, poor-quality care, which led to health care reforms. By 2000, the VHA had substantially improved in terms of numerous indicators of process quality, and some evidence shows that its overall performance now exceeds that of the rest of U.S. health care. Recently, however, the VHA has started to become a victim of its own success, with increased demands on the system raising concerns from some that access is becoming overly restricted and from others that its annual budget appropriations are becoming excessive. Nonetheless, the apparent turnaround in the VHA's performance offers encouragement that health care that is both financed and provided by the public sector can be an effective organizational form.

Published
2008

6. Inhibitory effect of estrogen on Rac1-expression in monocytes.

Author

Adam O, Hagel M, Theobald K, Böhm M, and Laufs U

Subjects
Angiotensin II metabolism, Angiotensin II pharmacology, Down-Regulation, Estradiol pharmacology, Estrogens pharmacology, Humans, Monocytes drug effects, Reactive Oxygen Species antagonists & inhibitors, Reactive Oxygen Species metabolism, Superoxides antagonists & inhibitors, Superoxides metabolism, rac1 GTP-Binding Protein antagonists & inhibitors, rac1 GTP-Binding Protein biosynthesis, Estradiol metabolism, Estrogens metabolism, Gene Expression Regulation, Developmental, Monocytes enzymology, rac1 GTP-Binding Protein genetics
Abstract

Recruitment of circulating monocytes into the vasculature and release of reactive oxygen species (ROS) promote atherogenesis. Rac1-GTPase is an essential component of the superoxide-producing NADPH-oxidase complex. Estrogens inhibit production of vascular reactive oxygen species. Angiotensin II as well as overexpression of the constitutively active mutant RacL61 increased ROS production in monocytes. AngII-mediated ROS release was completely inhibited by overexpression of the dominant negative mutant RacN17 or treatment with 17beta-estradiol. 17beta-Estradiol reduced Rac1-expression concentration- and time-dependently and decreased basal, as well as AngII-induced Rac1 activity. The effects of 17beta-estradiol were receptor-mediated. In vivo, down-regulation of Rac1 by 17beta-estradiol was observed in human mononuclear cells of women with elevated 17beta-estradiol levels after controlled ovarian hyperstimulation. In summary, the data show that down-regulation of Rac1-GTPase contributes to the inhibition of angiotensin II-mediated superoxide release by 17beta-estradiol in monocytes.

Published
2009
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