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6 results on '"Forbess, Lindsy"'

2. Contributors

Author

Agmon-Levin, Nancy, primary, Alarcón, Graciela S., additional, Amengual, Olga, additional, Ardoin, Stacy P., additional, Arora, Swati, additional, Atisha-Fregoso, Yemil, additional, Atkinson, John P., additional, Atsumi, Tatsuya, additional, Ayoub, Isabelle, additional, Barilla-LaBarca, Maria-Louise, additional, Bermas, Bonnie L., additional, Bernatsky, Sasha, additional, Bertsias, George, additional, Bichile, Tanmayee, additional, Blanco, Patrick, additional, Bohgaki, Miyuki, additional, Bonsmann, Gisela, additional, Borghi, Maria Orietta, additional, Boumpas, Dimitrios T., additional, Bourn, Rebecka, additional, Buyon, Jill P., additional, Caricchio, Roberto, additional, Chan, Edward K.L., additional, Chang, Christopher, additional, Charrier, Manon, additional, Chighizola, Cecilia Beatrice, additional, Clarke, Ann E., additional, Crispín, José C., additional, Cuneo, Bettina, additional, Dörner, Thomas, additional, Damato, Erika M., additional, Denniston, Alastair K.O., additional, Devlin, Amy, additional, Diamond, Betty, additional, Ernandez, T., additional, Falasinnu, Titilola, additional, Fernandez-Ruiz, Ruth, additional, Fitzpatrick, Brianna, additional, Forbess, Lindsy, additional, Frangou, Eleni A., additional, Fritzler, Marvin J., additional, Fu, Shu Man, additional, Furie, Richard, additional, Gaskin, Felicia, additional, Gladman, Dafna, additional, Gordon, Caroline, additional, Grammer, Amrie C., additional, Greidinger, Eric L., additional, Greiling, Teri M., additional, Han, Shuhong, additional, Hansen, James E., additional, Hasni, Sarfaraz A., additional, Hassan, Fadi, additional, Hedrich, Christian M., additional, Hiromura, Keiju, additional, Horowitz, Diane, additional, Huang, Xin, additional, Hunt, David, additional, Izmirly, Peter M., additional, James, Judith A., additional, Jarjour, Wael N., additional, Jefferies, Caroline A., additional, Jefferies, Caroline, additional, Jiang, Xiaoyue, additional, Kaplan, Mariana J., additional, Katsuyama, Takayuki, additional, Khamashta, Munther, additional, Kingsmore, Kathryn M., additional, Koike, Takao, additional, Kono, Dwight H., additional, Kriegel, Martin A., additional, Kuhn, Annegret, additional, Kyttaris, Vasileios C, additional, La Cava, Antonio, additional, Ladouceur, Alexandra, additional, Lahita, Robert G., additional, Landmann, Aysche, additional, Lazaro, Estibaliz, additional, Lennard Richard, Mara L., additional, Lino, Andreia C., additional, Lipsky, Peter E., additional, Liszewski, M. Kathryn, additional, Lo, Mindy S., additional, Lu, Qianjin, additional, Mahieu, Mary, additional, Malkiel, Susan, additional, Manzi, Susan, additional, Marder, Galina, additional, Mayadas, T.N., additional, Meroni, Pier Luigi, additional, Merrill, Joan T., additional, Mohan, Chandra, additional, Mok, Chi Chiu, additional, Moulton, Vaishali R., additional, Murray, Philip I., additional, Naffaa, Mohammad E., additional, Nangaku, Masaomi, additional, Niewold, Timothy, additional, Okubo, K., additional, Olsen, Nancy J., additional, Pal, Trina, additional, Paz, Ziv, additional, Perl, Andras, additional, Pons-Estel, Guillermo J., additional, Qu, Bo, additional, Rahman, Anisur, additional, Raman, Ziaur S.M., additional, Ramsey-Goldman, Rosalind, additional, Reeves, Westley H., additional, Richez, Christophe, additional, Rosetti, Florencia, additional, Rovin, Brad H., additional, Rubin, Robert L., additional, Saeli, Stephanie, additional, Saggu, G., additional, Sammaritano, Lisa R., additional, Satoh, Minoru, additional, Sawalha, Amr H., additional, Saxena, Amit, additional, Sciascia, Savino, additional, Shaharir, Syahrul Sazliyana, additional, Sharabi, Amir, additional, Shen, Nan, additional, Shmerling, Robert H., additional, Simard, Julia F., additional, Sisirak, Vanja, additional, Slight-Webb, Samantha, additional, Stillman, Isaac Ely, additional, Sung, Sun-Sang J., additional, Thakkar, Payal, additional, Theofilopoulos, Argyrios N., additional, Thomas, Jr, Donald E., additional, Tissera, Hiromi, additional, Touma, Zahi, additional, Tsao, Betty P., additional, Ugarte-Gil, Manuel F., additional, Urowitz, Murray B., additional, Vieira, Silvio Manfredo, additional, Wainwright, Benjamin, additional, Wallace, Daniel J., additional, Wang, Hongyang, additional, Wu, Haijing, additional, Yahia, Soad Haj, additional, Yu, C. Yung, additional, Zhao, Zhenhuan, additional, and Zhuang, Haoyang, additional

Published
2021
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4. A Large-Scale Genetic Analysis Reveals a Strong Contribution of the HLA Class II Region to Giant Cell Arteritis Susceptibility

Author

David Carmona, F, Mackie, SL, Martin, J-E, Taylor, JC, Vaglio, A, Eyre, S, Bossini-Castillo, L, Castaneda, S, Cid, MC, Hernandez-Rodriguez, J, Prieto-Gonzalez, S, Solans, R, Ramentol-Sintas, M, Francisca Gonzalez-Escribano, M, Ortiz-Fernandez, L, Morado, IC, Narvaez, J, Miranda-Filloy, JA, Beretta, L, Lunardi, C, Cimmino, MA, Gianfreda, D, Santilli, D, Ramirez, GA, Soriano, A, Muratore, F, Pazzola, G, Addimanda, O, Wijmenga, C, Witte, T, Schirmer, JH, Moosig, F, Schoenau, V, Franke, A, Palm, O, Molberg, O, Diamantopoulos, AP, Carette, S, Cuthbertson, D, Forbess, LJ, Hoffman, GS, Khalidi, NA, Koening, CL, Langford, CA, McAlear, CA, Moreland, L, Monach, PA, Pagnoux, C, Seo, P, Spiera, R, Sreih, AG, Warrington, KJ, Ytterberg, SR, Gregersen, PK, Pease, CT, Gough, A, Green, M, Hordon, L, Jarrett, S, Watts, R, Levy, S, Patel, Y, Kamath, S, Dasgupta, B, Worthington, J, Koeleman, BPC, de Bakker, PIW, Barrett, JH, Salvarani, C, Merkel, PA, Gonzalez-Gay, MA, Morgan, AW, Martin, J, Carmona, F. David, Mackie, Sarah L., Martín, Jose-Ezequiel, Taylor, John C., Vaglio, Augusto, Eyre, Stephen, Bossini-Castillo, Lara, Castañeda, Santo, Cid, Maria C., Hernández-Rodríguez, José, Prieto-González, Sergio, Solans, Roser, Ramentol-Sintas, Marc, González-Escribano, M. Francisca, Ortiz-Fernández, Lourde, Morado, Inmaculada C., Narváez, Javier, Miranda-Filloy, José A., Beretta, Lorenzo, Lunardi, Claudio, Cimmino, Marco A., Gianfreda, Davide, Santilli, Daniele, Ramirez, Giuseppe A., Soriano, Alessandra, Muratore, Francesco, Pazzola, Giulia, Addimanda, Olga, Wijmenga, Cisca, Witte, Torsten, Schirmer, Jan H., Moosig, Frank, Schönau, Verena, Franke, Andre, Palm, Oyvind, Molberg, Oyvind, Diamantopoulos, Andreas P., Carette, Simon, Cuthbertson, David, Forbess, Lindsy J., Hoffman, Gary S., Khalidi, Nader A., Koening, Curry L., Langford, Carol A., Mcalear, Carol A., Moreland, Larry, Monach, Paul A., Pagnoux, Christian, Seo, Philip, Spiera, Robert, Sreih, Antoine G., Warrington, Kenneth J., Ytterberg, Steven R., Gregersen, Peter K., Pease, Colin T., Gough, Andrew, Green, Michael, Hordon, Lesley, Jarrett, Stephen, Watts, Richard, Levy, Sarah, Patel, Yusuf, Kamath, Sanjeet, Dasgupta, Bhaskar, Worthington, Jane, Koeleman, Bobby P.C., De Bakker, Paul I.W., Barrett, Jennifer H., Salvarani, Carlo, Merkel, Peter A., González-Gay, Miguel A., Morgan, Ann W., and Martín, Javier

Subjects
Multifactorial Inheritance, Genotype, European Continental Ancestry Group, Genes, MHC Class II, Giant Cell Arteritis, Genetic Association Studie, Article, White People, MHC Class II, Cohort Studies, Genetic, Genes, Genetic Association Studies, Humans, Multivariate Analysis, Odds Ratio, Genetics, Genetics(clinical), Cohort Studie, Multivariate Analysi, Giant Cell Arteriti, Genetics (clinical), Human
Abstract

We conducted a large-scale genetic analysis on giant cell arteritis (GCA), a polygenic immune-mediated vasculitis. A case-control cohort, comprising 1,651 case subjects with GCA and 15,306 unrelated control subjects from six different countries of European ancestry, was genotyped by the Immunochip array. We also imputed HLA data with a previously validated imputation method to perform a more comprehensive analysis of this genomic region. The strongest association signals were observed in the HLA region, with rs477515 representing the highest peak (p = 4.05 × 10(-40), OR = 1.73). A multivariate model including class II amino acids of HLA-DRβ1 and HLA-DQα1 and one class I amino acid of HLA-B explained most of the HLA association with GCA, consistent with previously reported associations of classical HLA alleles like HLA-DRB1(∗)04. An omnibus test on polymorphic amino acid positions highlighted DRβ1 13 (p = 4.08 × 10(-43)) and HLA-DQα1 47 (p = 4.02 × 10(-46)), 56, and 76 (both p = 1.84 × 10(-45)) as relevant positions for disease susceptibility. Outside the HLA region, the most significant loci included PTPN22 (rs2476601, p = 1.73 × 10(-6), OR = 1.38), LRRC32 (rs10160518, p = 4.39 × 10(-6), OR = 1.20), and REL (rs115674477, p = 1.10 × 10(-5), OR = 1.63). Our study provides evidence of a strong contribution of HLA class I and II molecules to susceptibility to GCA. In the non-HLA region, we confirmed a key role for the functional PTPN22 rs2476601 variant and proposed other putative risk loci for GCA involved in Th1, Th17, and Treg cell function.

Published
2015

5. The Role of Imaging in Diagnosis and Monitoring of Large Vessel Vasculitis.

Author

Horomanski A and Forbess LJ

Subjects
Humans, Positron Emission Tomography Computed Tomography, Fluorodeoxyglucose F18, Positron-Emission Tomography, Takayasu Arteritis diagnostic imaging, Giant Cell Arteritis diagnostic imaging
Abstract

Technological advances and increased recognition of the prevalence and implications of large vessel vasculitis have led to robust research into various imaging techniques. Although there is still debate about which modality to choose in specific clinical scenarios, Ultrasound, PET/CT, MRI/A, and CT/A offer complementary information regarding diagnosis, disease activity, and vascular complication monitoring. Recognition of the strengths and limitations of each technique is important for appropriate application in clinical practice., (Copyright © 2023 Elsevier Inc. All rights reserved.)

Published
2023
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6. Polyarteritis nodosa.

Author

Forbess L and Bannykh S

Subjects
Adrenal Cortex Hormones therapeutic use, Glucocorticoids therapeutic use, HIV Infections complications, Hepatitis B complications, Hepatitis C complications, Humans, Immunosuppressive Agents therapeutic use, Polyarteritis Nodosa drug therapy, Polyarteritis Nodosa etiology, Angiography, Biopsy, Polyarteritis Nodosa diagnosis
Abstract

Polyarteritis nodosa (PAN) is a systemic disease, but variants are cutaneous PAN and single-organ disease. Histologic confirmation of vasculitis in medium-sized arteries is desirable, and biopsies should be obtained from the symptomatic and least invasive sites. Angiography can show multiple microaneurysms in the viscera. Treatment includes high-dose corticosteroids, which are combined with immunosuppressive agents when internal organs are involved and with life-threatening disease. Once remission is achieved, maintenance agents are initiated. PAN is becoming a rare disease. International collaborative efforts are under way to establish better diagnostic and classification for all vasculitides, including PAN.

Published
2015
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