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1. Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases.

2. Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors.

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3. A cell-based screen for resistance of Bcr-Abl-positive leukemia identifies the mutation pattern for PD166326, an alternative Abl kinase inhibitor.

4. Inhibition of bcr-abl gene expression by small interfering RNA sensitizes for imatinib mesylate (STI571).

5. Adhesion to fibronectin selectively protects Bcr-Abl+ cells from DNA damage-induced apoptosis.

6. Bcr-Abl kinase down-regulates cyclin-dependent kinase inhibitor p27 in human and murine cell lines.