154 results on '"BUETTNER, Garry R."'
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2. Pharmacological ascorbate induces sustained mitochondrial dysfunction
3. Ascorbate Mediates the Non-Enzymatic Reduction of Nitrite to Nitric Oxide
4. Exposure to Static Magnetic and Electric Fields Treats Type 2 Diabetes
5. Extracellular biomolecular free radical formation during injury
6. Pharmacological ascorbate improves the response to platinum-based chemotherapy in advanced stage non-small cell lung cancer
7. Red blood cells contain enzymatically active GPx4 whose abundance anticorrelates with hemolysis during blood bank storage
8. N-alkyl triphenylvinylpyridinium conjugated dihydroartemisinin perturbs mitochondrial functions resulting in enhanced cancer versus normal cell toxicity
9. Magnetic resonance imaging (MRI) of pharmacological ascorbate-induced iron redox state as a biomarker in subjects undergoing radio-chemotherapy
10. Exposure to Static Magnetic and Electric Fields Treats Type 2 Diabetes
11. Exposure to Static Magnetic and Electric Fields Treats Type 2 Diabetes
12. The latency of peroxisomal catalase in terms of effectiveness factor for pancreatic and glioblastoma cancer cell lines in the presence of high concentrations of H2O2: Implications for the use of pharmacological ascorbate in cancer therapy
13. Response to Ling et al. regarding “An integrated physico-chemical approach for explaining the differential impact of FLASH versus conventional dose rate irradiation on cancer and normal tissue responses”
14. Simultaneous detection of the enzyme activities of GPx1 and GPx4 guide optimization of selenium in cell biological experiments
15. Disulfiram causes selective hypoxic cancer cell toxicity and radio-chemo-sensitization via redox cycling of copper
16. Corrigendum to: Augmentation of intracellular iron using iron sucrose enhances the toxicity of pharmacological ascorbate in colon cancer cells [Redox Biol. (2018) 82–87]
17. An integrated physico-chemical approach for explaining the differential impact of FLASH versus conventional dose rate irradiation on cancer and normal tissue responses
18. Response to letter regarding “An integrated physico-chemical approach for explaining the differential impact of FLASH versus conventional dose rate irradiation on cancer and normal tissue responses”
19. The relationship between vitamin C status, the gut-liver axis, and metabolic syndrome
20. Pharmacological Ascorbate as a Means of Sensitizing Cancer Cells to Radio-Chemotherapy While Protecting Normal Tissue
21. Extracellular superoxide dismutase (SOD3) regulates oxidative stress at the vitreoretinal interface
22. Calculated cell-specific intracellular hydrogen peroxide concentration: Relevance in cancer cell susceptibility during ascorbate therapy
23. Augmentation of intracellular iron using iron sucrose enhances the toxicity of pharmacological ascorbate in colon cancer cells
24. Redox active metals and H2O2 mediate the increased efficacy of pharmacological ascorbate in combination with gemcitabine or radiation in pre-clinical sarcoma models
25. Estimating and Partitioning the Flux of ATP in Cells in Culture using mol cell -1 s -1 and the Stoichiometries of Metabolism
26. DNA Damage and Energy Crisis are Central in the Mechanism for the Cytotoxicity of Pharmacological Ascorbate in Cancer Treatment
27. Penta-azamacrocyclic SOD Mimetics Enhance Pharmacological Ascorbate Oxidation and Anticancer effects in an H 2 O 2 -dependent Manner
28. Quantitative Differences in Physical Size, H 2 O 2 -Removal, and Cellular Bioenergetics in the Human Pancreatic Cancer Cell Lines MIA PaCa-2 and Panc-1: Impact on Susceptibilities to Ascorbate/H 2 O 2
29. Mitochondrial dynamics regulates metabolism during aging
30. O2⋅− and H2O2-Mediated Disruption of Fe Metabolism Causes the Differential Susceptibility of NSCLC and GBM Cancer Cells to Pharmacological Ascorbate
31. O 2 ⋅− and H 2 O 2 -Mediated Disruption of Fe Metabolism Causes the Differential Susceptibility of NSCLC and GBM Cancer Cells to Pharmacological Ascorbate
32. Tumor cells have decreased ability to metabolize H2O2: Implications for pharmacological ascorbate in cancer therapy
33. Dual Oxidase 1 Promotes Crosstalk between Extracellular H2O2 and Mitochondrial Respiration During Pharmacological Ascorbate Induced Toxicity of Pancreatic Cancer Cells
34. Catalase as a Potential Biomarker of Pharmacological Ascorbate Cancer Therapy
35. A Lipogenic-Switch Shifts Metabolism from Glycolysis to Mitochondrial Respiration During Aging
36. Leveraging Data from Bioenergetic Experiments to Determine the Absolute Flux of ATP in Cells from Respiration and Glycolysis
37. Quantitative Changes in Dihydroethidium (DHE) Oxidation Products from Isolated Mitochondria While Respiring on Select Substrates and the Effects Mitochondrial Inhibitors Commonly Used in Bioenergetic Profiling
38. Direct spectrophotometric measurement of supra-physiological levels of ascorbate in plasma
39. Going Beyond the Specification of Dose of a Xenobiotic on a Mol Per Cell Basis in Cell Culture Media
40. Mitofusin-1 Regulates Metabolic Reprogramming from Glycolysis to Mitochondrial Respiration During Aging 1
41. Using Quantitative Redox Biology to Achieve a More Universal and Global Bioenergetic Phenotype Analysis
42. The Role of Pharmacological Ascorbate on Tumor Cell Invasion and Metastasis
43. The Selective Toxicity of Pharmacological Ascorbate Is Mediated by Alterations in Iron Metabolism
44. Differences in the Capacities of Tumor and Normal Cells to Remove Extracellular Hydrogen Peroxide: Exploring the Mechanism of Pharmacological Ascorbate in Cancer Therapy
45. Role of labile iron in the toxicity of pharmacological ascorbate
46. Manganoporphyrins and ascorbate enhance gemcitabine cytotoxicity in pancreatic cancer
47. Moving free radical and redox biology ahead in the next decade(s)
48. The Addition of Manganoporphyrins and Ascorbate to Standard of Care Chemotherapy Enhances Tumor-Specific Cytotoxicity in Pancreatic Cancer
49. Age-Associated Metabolic Reprogramming in Stroma Promotes Pancreatic Cancer Progression
50. Superoxide Mediates Alterations in Complex I Activity Seen in Adult Versus Neonatal Fibroblasts
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