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1. Inhibiting glycogen biosynthesis by mTORC1 suppression as an adjunct therapy for Pompe disease

2. Inhibition of glycogen biosynthesis via mTORC1 suppression as an adjunct therapy for Pompe disease

4. Glycoengineered Acid α-Glucosidase With Improved Efficacy at Correcting the Metabolic Aberrations and Motor Function Deficits in a Mouse Model of Pompe Disease

6. Replacing acid α-glucosidase in Pompe disease: recombinant and transgenic enzymes are equipotent, but neither completely clears glycogen from type II muscle fibers

12. Two human 35 kd inhibitors of phospholipase A2 are related to substrates of pp60v-src and of the epidermal growth factor receptor/kinase

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