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1. KSHV infection drives poorly cytotoxic CD56-negative natural killer cell differentiation in vivo upon KSHV/EBV dual infection

2. KSHV Infection Drives Poorly Cytotoxic CD56 Negative Natural Killer Cell Differentiation In vivo

3. Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression

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