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We present central-to-peripheral nuclear modification factors, R CP , for the p T distributions of K S 0 , Λ, Λ ¯ , and negatively charged particles, measured at central rapidity in Pb–Pb collisions at top SPS energy. The data cover the 55% most central fraction of the inelastic cross section. The K S 0 and Λ R CP ( p T ) are similar in shape to those measured at s NN = 200 GeV at RHIC, though they are larger in absolute value. We have compared our K S 0 R CP data to a theoretical calculation. The prediction overestimates the data at p T ≈ 3 – 4 GeV / c , unless sizeable parton energy loss is included in the calculation.

A measurement of strange baryon and antibaryon production in Pb-Pb collisions has been carried out by the NA57 experiment at the CERN SPS, with 40 and 158 A GeV/c beam momentum. Results on $\Lambda$, $\Xi$ and $\Omega$ hyperon yields at mid-rapidity in the most central 53% of Pb-Pb collisions at 40 A GeV/c are presented and compared with those obtained at higher energy, in the same collision centrality range. The $\Lambda$ and $\Xi^-$ yields per unit rapidity stay roughly constant while those of $\Omega^-$, $\bar\Lambda$, $\bar\Xi^+$ and $\bar\Omega^+$ increase when going to the higher SPS energy. Hyperon yields at the SPS are compared with those from the STAR experiment in $\sqrt{s_{NN}}$ = 130 GeV Au-Au collisions at RHIC., Comment: 9 pages, 4 figures, 3 tables, accepted for publication in Physics Letters B

F. Antinori’, A. Bada&‘, R. Barberag, A. Belogiannia, A. Bhasine, I.J. Bloodworthe, G.E. Bruno*, S.A. Bulle, R. Caliandro*, M. Campbellh, W. Carenah, N. Carrerh, R.F. Clarkee, A. Dainese’, A.P. de Haa.9, P.C. de Rijkes, D. Di Bari*, S. Di Liberto’, R. Diviah, D. Elia*, D. Evanse, K. Fanebus@, F. Fayazzadeh”, J. Fedorisin j, G.A. Feofilov*, R.A. Fini*, P. Ganotia, B. Ghidini*, G. Grellap, H. Helstrupd, M. Henriquez”, A.K. Holmek, A. Jacholkowski*, G.T. Jonese, P. Jovanovice, A. Juskoi, R. KamermansS, J.B. Kinsone, K. Knudsonh, A.A. Kolozhvariq, V. Kondratiev ‘3, I. Kr&liki, A. KravEBkova*, P. Kuije?, V. Lenti*, R. Lietavae, G. Lovhoiden”, V. Manzari*, G. Martinskaj, M.A. MazzoniO, F. Meddi’, A. Michalonr, M. Morando’, E. Nappi*, F. Navach*, P.I. Normane, A. Palmeris, G.S. Pappalardog, B. PastirE&ki, J. Pi&, N. Pisutovaf, F. Posa*, E. Quercigh’, F. Rig@, D. RGhrich’, G. RomanoP, K. Safafikh, L. SBndo$‘, E. Schillin&, G. Segato’, M. SenCm, R. Sent?, W. Snoeysh, F. Soramel’, M. Spyropoulou-Stassinaki ‘, P. Staroba”, T.A. ToulinaQ, R. Turrisi’, T.S. Tveterk, J. Urbinj, F. Valievq, A. van den Brinks, P. van de Ven’, P. Vande Vyvreh, N. van Eijndhovens, J. van Hunenh, A. Vascottoh, T. Vik”, 0. Villalobos Bailliee, L. Vinogradovq, T. Virgil?‘, M.F. Votrubae, J. Vrlikovd and P. Zavadan

This paper combines the results from a preliminary survey of occurrence of Cryptosporidium species in faecal samples from a range of wild mammal species inhabiting mainland Britain with a tabulated literature review of world-wide reports of the parasite in those British mammals. In the literature, C. parvum was reported from 11 wild mammals found in Britain and elsewhere, mainly in rodents but also in insectivores, lagomorphs and ungulates. C. muris has been reported only in wild rodents. The sample survey detected C. parvum in seven additional British species, including carnivores. Overall, 12% of 184 faecal samples tested with a genus-specific monoclonal antibody contained oocysts of C. parvum. The results further emphasise the widespread distribution of Cryptosporidium amongst wild mammals in Britain, highlight the potential for transmission between host species and warn of the possibility of direct exposure for anybody using the countryside for professional or recreational purposes (e.g. farmers and ramblers) to previously unregarded sources of infection. It seems increasingly likely that most, if not all, mammalian species can be infected with C. parvum.

The new results on hyperon production in Pb-Pb collisions at 158 A GeV/c and 40 A GeV/c are presented. Publisher Summary This chapter discusses the yields of hyperon in Pb–Pb collisions from the NA57 experiment. The aim of NA57 was to study the dependence of the strangeness enhancement on the interaction volume and the collision energy per incoming nucleon. The centrality range of the NA57 experiment extended down to a limit of about 50 wounded nucleons. The experiment collected data using 158 and 40 A GeV/c beams at the CERN SPS. There was indication of an increase of the enhancements with the centrality of the collisions. The data were corrected for acceptance and efficiency losses by calculating weight for each particle with a GEANT-based Monte-Carlo simulation.

The effects of antibiotics on bovine luteinizing hormone (bLH)-stimulated output of androstenedione were examined in intact small white follicles (SWF, n = six hens), aliquots of 100,000 Fl and F2 theca cells (n = six hens), and aliquots of 10,000 Fl and F2 granulosa cells (n = eight hens). Cells and SWF were isolated from ovaries of Leghorn hens killed 20 h prior to ovulation and were incubated for 3 h in 1 mL of Medium 199 (Sigma Chemical Co., St. Louis, MO) at 37 C. Cells and follicles were incubated with 10 ng bLH and Penicillin-streptomycin (Pen-Strep; 20, 100, and 200 μ.g), gentamicin sulfate (gentamicin; 10, 50, and 100 μg), and fungizone (.5, 2.5, and 5.0 μg). Two control treatments consisted of tissues incubated alone (no bLH, no antibiotic) and tissues incubated with 10 ng bLH (no antibiotic). Androstenedione output by SWF and Fl and F2 theca cells and progesterone output by Fl and F2 granulosa cells were determined by radioimmunoassay of incubation media. The steroid contents of the incubation media were significantly increased in SWF and theca and granulosa cells with the addition of bLH. Neither Pen-Strep nor gentamicin significantly affected steroid output. Progesterone output by Fl and F2 granulosa cells was significantly reduced by the inclusion of fungizone. The inhibition of steroid output with the use of fungizone should be considered when using this antibiotic in studies of steroidogenesis in preparations of ovarian cells.