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1. Phenotypic and functional assessment of two novel KCNQ2 gain-of-function variants Y141N and G239S and effects of amitriptyline treatment

3. KCNQ2 R144 variants cause neurodevelopmental disability with language impairment and autistic features without neonatal seizures through a gain-of-function mechanism

4. Cerebrospinal Fluid Ion Analysis in Neonatal Seizures

5. Kv7.4 channels regulate potassium permeability in neuronal mitochondria

6. Generation of an iPSC line (UNINAi001-A) from a girl with neonatal-onset epilepsy and non-syndromic intellectual disability carrying the homozygous KCNQ3 p.PHE534ILEfs*15 variant and of an iPSC line (UNINAi002-A) from a non-carrier, unaffected brother

7. Gabapentin treatment in a patient with KCNQ2 developmental epileptic encephalopathy

9. Effects of natural and synthetic isothiocyanate-based H 2 S-releasers against chemotherapy-induced neuropathic pain: Role of Kv7 potassium channels

10. β-Adrenergic response is counteracted by extremely-low-frequency pulsed electromagnetic fields in beating cardiomyocytes

11. Characterization of two de novo KCNT1 mutations in children with malignant migrating partial seizures in infancy

12. Epilepsy-causing mutations in Kv7.2 C-terminus affect binding and functional modulation by calmodulin

13. PIP2 and Surface Expression Underlie Apo-Calmodulin Dependent Kv7.2/KCNQ2 Current Potentiation

15. Critical role of large-conductance calcium- and voltage-activated potassium channels in leptin-induced neuroprotection of N-methyl-d-aspartate-exposed cortical neurons

16. The Ever Changing Moods of Calmodulin: How Structural Plasticity Entails Transductional Adaptability

23. Corrigendum to “Neutralization of a unique, negatively-charged residue in the voltage sensor of KV7.2 subunits in a sporadic case of benign familial neonatal seizures” [Neurobiol. Dis. 34 (2009) 501–510]

24. Neutralization of a unique, negatively-charged residue in the voltage sensor of KV7.2 subunits in a sporadic case of benign familial neonatal seizures

25. Gating currents from neuronal Kv7 channels

31. Decreased Subunit Stability as a Novel Mechanism for Potassium Current Impairment by a KCNQ2 C Terminus Mutation Causing Benign Familial Neonatal Convulsions

33. A Novel Hyperekplexia-causing Mutation in the Pre-transmembrane Segment 1 of the Human Glycine Receptor α1 Subunit Reduces Membrane Expression and Impairs Gating by Agonists

35. Inhibition of depolarization-induced [3H]noradrenaline release from SH-SY5Y human neuroblastoma cells by some second-generation H1 receptor antagonists through blockade of store-operated Ca2+ channels (SOCs)11Abbreviations: hERG, human Ether-a-go-go Related Gene; SOC, Ca2+ currents activated by [Ca2+]i store depletion; NE, norepinephrine; [K+]e, e xtracellular K+ concentration; [Ca2+]i, intracellular Ca2+ concentration; HBS, HEPES-buffered saline; SERCA, sarcoplasmic-endoplasmic reticulum calcium ATPase; and TTX, tetrodotoxin.

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