51 results on '"Wang, David Q-H."'
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2. Targeting mitochondria to oppose the progression of nonalcoholic fatty liver disease
3. Silencing steroid receptor coactivator-1 in the nucleus of the solitary tract reduces estrogenic effects on feeding and apolipoprotein A-IV expression
4. Gut vagal afferents are necessary for the eating-suppressive effect of intraperitoneally administered ginsenoside Rb1 in rats
5. Estrogen induces two distinct cholesterol crystallization pathways by activating ERα and GPR30 in female mice
6. Ginsenoside Rb1 reduces fatty liver by activating AMP-activated protein kinase in obese rats
7. Role of mitochondria in nonalcoholic fatty liver disease-from origin to propagation
8. Apolipoprotein E reduces food intake via PI3K/Akt signaling pathway in the hypothalamus
9. Diet-induced lipid accumulation in phospholipid transfer protein-deficient mice: its atherogenicity and potential mechanism
10. Biliary lipids and cholesterol gallstone disease
11. Up-regulation of apolipoprotein E by leptin in the hypothalamus of mice and rats
12. 65 - Gallstone Disease
13. Effect of Ezetimibe on the Prevention and Dissolution of Cholesterol Gallstones
14. Parallel intestinal and liver injury during early cholestasis in the rat: Modulation by bile salts and antioxidants
15. Evidence That Gallbladder Epithelial Mucin Enhances Cholesterol Cholelithogenesis in MUC1 Transgenic Mice
16. Overexpression of estrogen receptor α increases hepatic cholesterogenesis, leading to biliary hypersecretion in mice
17. Gallbladder histopathology during murine gallstone formation: relation to motility and concentrating function
18. New Insights Into the Genetic Regulation of Intestinal Cholesterol Absorption
19. Cholesterol absorption is mainly regulated by the jejunal and ileal ATP-binding cassette sterol efflux transporters Abcg5 and Abcg8 in mice
20. Targeted disruption of the murine mucin gene 1 decreases susceptibility to cholesterol gallstone formation
21. Estrogen receptor α, but not β, plays a major role in 17β-estradiol-induced murine cholesterol gallstones
22. Measurement of intestinal cholesterol absorption by plasma and fecal dual-isotope ratio, mass balance, and lymph fistula methods in the mouse: an analysis of direct versus indirect methodologies
23. Effect of β-muricholic acid on the prevention and dissolution of cholesterol gallstones in C57L/J mice
24. Aging per se is an independent risk factor for cholesterol gallstone formation in gallstone susceptible mice
25. Dietary sphingomyelin suppresses intestinal cholesterol absorption by decreasing thermodynamic activity of cholesterol monomers
26. Lith genes control mucin accumulation, cholesterol crystallization, and gallstone formation in A/J and AKR/J inbred mice
27. Genetic factors at the enterocyte level account for variations inintestinal cholesterol absorption efficiency among inbred strains of mice
28. Contributors
29. Chapter 65 - Gallstone Disease
30. Phenotypic characterization of Lith genes that determine susceptibility to cholesterol cholelithiasis in inbred mice: pathophysiology of biliary lipid secretion
31. Phenotypic characterization of Lith genes that determine susceptibility to cholesterol cholelithiasis in inbred mice: integrated activities of hepatic lipid regulatory enzymes
32. No pathophysiologic relationship of soluble biliary proteins to cholesterol crystallization in human bile
33. Cryoelectron Microscopy of a Nucleating Model Bile in Vitreous Ice: Formation of Primordial Vesicles
34. Bile Formation and Pathophysiology of Gallstones
35. Hepatocytes and Bile Formation
36. 3 - Absorption and Excretion of Intestinal Cholesterol and Other Sterols
37. Cholecystectomy: a way forward and back to metabolic syndrome?
38. Contributors
39. Contributors
40. Targeted disruption of mucin gene 1 (Mucl) decreases susceptibility to cholesterol (Ch) gallstone formation in mice
41. Dietary sphingomyelin (SM) significantly inhibits intestinal cholesterol (Ch) absorption by lowering thermodynamic activity (TA) of Ch in bile salt (BS) mixed micellar solution
42. Genetic determinants of mucin accumulation in the murine gallbladder prior to gallstone formation: Identification of susceptibility loci and candidate genes
43. Effect of β-muricholic acid (β-MCA), a natural trihydroxy bile acid, on the prevention and dissolution of cholesterol (ch) gallstones in gallstone-susceptible C57L mice
44. Targeted disruption of the HDL receptor (Seavenger receptor B1, SR-B1) in mice decreases billary cholesterol (Ch) secretion in the basal state and modestly influences Ch gallstone susceptibility
45. Good cholesterol, bad cholesterol: Role of oxysterols in biliary tract diseases
46. Chapter 65 - Gallstone Disease
47. The major gallstone gene (Lith 1) is responsible for biliary cholesterol (CH) hypersecretion and CH gallstone formation in congenic mice (AKR.L-Lith1s)
48. Essential role of Mdr2 P-glycoprotein in regulation of hepatic activities of major cholesterol regulatory enzymes
49. Spontaneous cholesterol gallstone formation on normal diet characterizes mice with disrupted Mdr2 P-glycoprotein gene and provides new insights into cholesterol crystalization and bile formation
50. Cholesterol (CH) crystal (C) nucleation paths in human gallbladder bile are identical to model bile: Increased secondary bile salts (BS) and CH saturation index (CSI) are partly responsible for accelerating CH nucleation
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