80 results on '"Ansari, Aftab A."'
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2. Viremia controls Env-specific antibody-secreting cell responses in simian immunodeficiency virus infected macaques pre and post-antiretroviral therapy.
3. Autoreactive monoclonal antibodies from patients with primary biliary cholangitis recognize environmental xenobiotics.
4. Early initiation of antiretroviral treatment postSIV infection does not resolve lymphoid tissue activation.
5. The fingerprint of antimitochondrial antibodies and the etiology of primary biliary cholangitis.
6. Up-regulation of Tim-3 on T cells during acute simian immunodeficiency virus infection and on antigen specific responders.
7. Ongoing activation of autoantigen-specific B cells in primary biliary cirrhosis.
8. Shotgun proteomics: Identification of unique protein profiles of apoptotic bodies from biliary epithelial cells.
9. IL-12/Th1 and IL-23/Th17 biliary microenvironment in primary biliary cirrhosis: Implications for therapy.
10. Clonality, activated antigen-specific CD8+ T cells, and development of autoimmune cholangitis in dnTGFβRII mice.
11. Antimitochondrial antibody heterogeneity and the xenobiotic etiology of primary biliary cirrhosis.
12. Deletion of interleukin (IL)-12p35 induces liver fibrosis in dominant-negative TGFβ receptor type II mice.
13. Therapeutic effect of cytotoxic T lymphocyte antigen 4/immunoglobulin on a murine model of primary biliary cirrhosis.
14. The immunobiology of colitis and cholangitis in interleukin-23p19 and interleukin-17a deleted dominant negative form of transforming growth factor beta receptor type ii mice.
15. Comparative analysis of portal cell infiltrates in antimitochondrial autoantibody-positive versus antimitochondrial autoantibody-negative primary biliary cirrhosis.
16. Immunoglobulin M levels inversely correlate with CD40 ligand promoter methylation in patients with primary biliary cirrhosis.
17. Fine phenotypic and functional characterization of effector cluster of differentiation 8 positive T cells in human patients with primary biliary cirrhosis.
18. Epithelial cell specificity and apotope recognition by serum autoantibodies in primary biliary cirrhosis.
19. Interaction between Toll-like receptors and natural killer cells in the destruction of bile ducts in primary biliary cirrhosis.
20. B cell depletion therapy exacerbates murine primary biliary cirrhosis.
21. Biliary apotopes and anti-mitochondrial antibodies activate innate immune responses in primary biliary cirrhosis.
22. Deletion of interleukin-6 in mice with the dominant negative form of transforming growth factor β receptor II improves colitis but exacerbates autoimmune cholangitis.
23. Deletion of interleukin-12p40 suppresses autoimmune cholangitis in dominant negative transforming growth factor β receptor type II mice.
24. Apotopes and the biliary specificity of primary biliary cirrhosis.
25. Differential mechanisms in the pathogenesis of autoimmune cholangitis versus inflammatory bowel disease in interleukin-2Rα−/− mice.
26. Loss of tolerance in C57BL/6 mice to the autoantigen E2 subunit of pyruvate dehydrogenase by a xenobiotic with ensuing biliary ductular disease.
27. Adoptive transfer of CD8+ T cells from transforming growth factor beta receptor type II (dominant negative form) induces autoimmune cholangitis in mice.
28. Natural killer T cells exacerbate liver injury in a transforming growth factor β receptor II dominant-negative mouse model of primary biliary cirrhosis.
29. A sensitive bead assay for antimitochondrial antibodies: Chipping away at AMA-negative primary biliary cirrhosis.
30. AMA production in primary biliary cirrhosis is promoted by the TLR9 ligand CpG and suppressed by potassium channel blockers.
31. IL-2 receptor α−/− mice and the development of primary biliary cirrhosis.
32. Liver-targeted and peripheral blood alterations of regulatory T cells in primary biliary cirrhosis.
33. Central memory CD4 T cells are the predominant cell subset resistant to anergy in SIV disease resistant sooty mangabeys.
34. Altered monocyte responses to defined TLR ligands in patients with primary biliary cirrhosis.
35. Genetic polymorphisms influencing xenobiotic metabolism and transport in patients with primary biliary cirrhosis.
36. Caspase induction by IgA antimitochondrial antibody: IgA-mediated biliary injury in primary biliary cirrhosis.
37. Epidemiology and pathogenesis of primary biliary cirrhosis.
38. Magnetic Resonance Imaging of Activated Proliferating Rhesus Macaque T Cells Labeled With Superparamagnetic Monocrystalline Iron Oxide Nanoparticles.
39. Patients with primary biliary cirrhosis react against a ubiquitous xenobiotic-metabolizing bacterium.
40. Myeloperoxidase-positive inflammatory cells participate in bile duct damage in primary biliary cirrhosis through nitric oxide-mediated reactions.
41. Characterization of recombinant monoclonal IgA anti-PDC-E2 autoantibodies derived from patients with PBC.
42. Differential expression of intestinal trefoil factor in biliary epithelial cells of primary biliary cirrhosis.
43. Comprehensive mapping of HLA-A*0201-restricted CD8 T-cell epitopes on PDC-E2 in primary biliary cirrhosis.
44. Analysis of TCR antagonism and molecular mimicry of an HLA-A*0201-restricted CTL epitope in primary biliary cirrhosis.
45. Contribution to antimitochondrial antibody production: Cleavage of pyruvate dehydrogenase complex-E2 by apoptosis-related proteases.
46. Immunoglobulin gene usage and immunohistochemical characteristics of human monoclonal antibodies to the mitochondrial autoantigens of primary biliary cirrhosis induced in the XenoMouse.
47. Heterogeneous response of antimitochondrial autoantibodies and bile duct apical staining monoclonal antibodies to pyruvate dehydrogenase complex E2: The molecule versus the mimic.
48. Mucosal immunity and primary biliary cirrhosis: Presence of antimitochondrial antibodies in urine.
49. Fetal microchimerisms in the mother: Immunologic implications.
50. Evidence for a locally driven mucosal response and the presence of mitochondrial antigens in saliva in primary biliary cirrhosis.
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