32 results on '"Hu, Chunyan"'
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2. Abstract MP14: Endothelial Cullin3 Mutation Causes Decreased Nitric Oxide (NO) Bioavailability And Vascular Dysfunction Through Protein Phosphatase 2A
3. Sex-Dependent Effects of Nephron Ift88 Disruption on BP, Renal Function, and Cystogenesis
4. Loss of Soluble (Pro)renin Receptor Attenuates Angiotensin-II Induced Hypertension and Renal Injury
5. Abstract P086: Endothelial Cullin3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, And Hypertension
6. Renomedullary Interstitial Cell Endothelin A Receptors Regulate BP and Renal Function
7. Nephron-Specific Disruption of Polycystin-1 Induces Cyclooxygenase-2–Mediated Blood Pressure Reduction Independent of Cystogenesis
8. Abstract 078: Nephron-Specific Deletion of Intraflagellar Transport 88 Alters Renal Electrolyte Excretion in the Absence of Cystic Kidney Disease in a Sex-Specific Manner
9. Abstract 065: Endothelial CULLIN3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
10. Interference With Endothelial PPAR (Peroxisome Proliferator–Activated Receptor)-γ Causes Accelerated Cerebral Vascular Dysfunction in Response to Endogenous Renin-Angiotensin System Activation
11. Glutathione-S-transferases M1/T1 gene polymorphisms and male infertility risk in Chinese populations
12. Abstract 110: Vascular Smooth Muscle RhoBTB1 Protects From Hypertension and Arterial Stiffness by Cullin-3 Dependent Ubiquitination of Phosphodiesterase 5
13. Abstract 036: Interference With PPARγ in the Endothelium Produces Endothelial Dysfunction in the Cerebral Circulation in Response to Activation of the Endogenous Renin-Angiotensin System
14. Abstract 121: Endothelial Cullin3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
15. Abstract 140: Endogenous Renin-angiotensin System Activation Causes Accelerated Cerebral Vascular Dysfunction in Mice Expressing Dominant-negative Mutations in PPARγ in Endothelium
16. Abstract 062: Vascular Dysfunction and Hypertension are Prevented by a Novel PPARγ Target Gene, RhoBTB1
17. Abstract P189: Endothelial Cullin3 Mutation Causes Vascular Dysfunction, Arterial Stiffening, and Hypertension
18. Genetic Interference With Endothelial PPAR-γ (Peroxisome Proliferator–Activated Receptor-γ) Augments Effects of Angiotensin II While Impairing Responses to Angiotensin 1–7
19. Abstract 124: RhoBTB1, a Novel PPARγ Target Gene, Rescues Hypertension and Vascular Dysfunction Caused by PPARγ Dysfunction
20. Abstract P283: Effects of High Salt Diet on Vascular Function and Renal Injury in a Novel Mouse Model of Neurogenic Hypertension
21. Investigation of the expression of apoptosis-inducing factor-mediated apoptosis in Hirschsprung’s disease
22. Abstract P347: Retinol-binding Protein 7 (RBP7) is Required for PPARG-mediated Endothelial Protection via Adiponectin
23. Abstract 053: RhoBTB1 is a Novel Gene Protecting Against Hypertension
24. Abstract P158: Cullin3 Regulated Endothelial Function by Modulating eNOS Activity
25. Abstract P205: Endothelium-specific Interference with PPARG Causes Cerebral Vascular Dysfunction in Response to Endogenous Renin-angiotensin System Activation
26. Abstract 048: Expression of a Hypertension-causing Mutation in Cullin 3 (CUL3Δ9) Specifically in Smooth Muscle Causes Vascular Dysfunction and Hypertension
27. Abstract P123: Retinol-Binding Protein 7 (RBP7) Regulates Endothelial Function via an Adiponectin-Dependent Mechanism in Angiotensin II-Infused Mice
28. Abstract P128: RhoBTB1, a Novel PPARγ Target Gene Regulates Vascular Function
29. Abstract 332: Retinol-binding Protein 7 (RBP7) Regulates Endothelial Function, Independent of Metabolic Responses in High-fat Diet Fed Mice
30. Abstract 21: Retinol-binding Protein 7 (RBP7), a Peroxisome Proliferator-Activated Receptor Gamma (PPARG) Target Gene, is a Novel Regulator of Endothelial Function
31. Role of specific T-type calcium channel blocker R(−) efonidipine in the regulation of renal medullary circulation
32. Effects of Renal Perfusion Pressure on Renal Medullary Hydrogen Peroxide and Nitric Oxide Production
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