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Category: 27. Clinical Electrophysiology—Ventricular ArrhythmiasSession-Poster Board Number: 1057-381 Authors: Yoshihiro Yamashina, Tetsuo Yagi, Akio Namekawa, Akihiko Ishida, Hirokazu Sato, Takashi Nakagawa, Manjirou Sakuramoto, Eiji Sato, Tomoyuki Yambe, Sendai City Hospital, Sendai, Japan, Tohoku University School of Medicine, Sendai, Japan Background: The relationship between J waves during sinus rhythm and idiopathic ventricular tachycardia (VT) or premature ventricular complexes (PVCs) originating from the right ventricular outflow tract (RVOT) has not been reported. The aim of this study was to investigate the prevalence and characteristics of idiopathic RVOT-VT/PVCs associated with J waves. Methods: The study enrolled 121 consecutive idiopathic RVOT-VT/PVC patients (mean age 45 ± 16 years, 46 males) undergoing radiofrequency catheter ablation (RFCA). The prevalence of J waves was assessed. The clinical and electrophysiological data were compared between the patients with J waves (J-wave group) and those without (Non-J-wave group). Results: J waves were present in 53 (43.8%) idiopathic RVOT-VT/PVC patients. The distribution of the J waves was as follows: Inferior leads = 62%, inferior-lateral leads = 24.5% and lateral leads = 13.2%. The J-wave group had a higher incidence of sustained VT (22.6% vs. 3.5%, P

Programmed electrical stimulation (PES) was performed in 17 patients, mean age 31 +/- 8 years, with minor forms of right ventricular dysplasia diagnosed because of (1) premature ventricular beats (PVBs) of left bundle branch block (LBBB) morphology; (2) no clinical or non-invasive evidence of cardiac abnormalities; (3) angiographic evidence of right ventricular wall motion abnormalities and bioptic findings of fibro-adipose infiltration. Fifteen patients had frequent and complex PVBs while two had sustained ventricular tachycardia (VT). During PES, sustained VT was induced in 2/2 patients with spontaneous sustained VT; ventricular repetitive responses were induced in 2/15 cases (13%) with complex and frequent PVBs. In conclusion, in minor forms of right ventricular dysplasia, PES induces VT only in patients with clinical VT; on the contrary, in patients with PVBs it is only possible to induce repetitive ventricular responses in a small proportion of cases; it is therefore not possible to select patients at high risk of developing severe ventricular arrhythmias.