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Your search keyword '"YAMAGATA, Kunihiro"' showing total 49 results
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49 results on '"YAMAGATA, Kunihiro"'

2. Recommended configuration for personal health records by standardized data item sets for diabetes mellitus and associated chronic diseases: a report from a collaborative initiative by six Japanese associations

Author

Nakashima, Naoki, Noda, Mitsuhiko, Ueki, Kohjiro, Koga, Tatsuhiko, Hayashi, Michio, Yamazaki, Katsuya, Nakagami, Tomoko, Ohara, Makoto, Gochi, Akira, Matsumura, Yasushi, Kimura, Michio, Ohe, Kazuhiko, Kang, Dongchon, Toya, Yoshiyuki, Yamagata, Kunihiro, Yokote, Koutaro, Ikeda, Shunya, Mitsutake, Naohiro, Yamamoto, Ryuichi, and Tanizawa, Yukio

Published
2019
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5. Mortality risk among screened subjects of the specific health check and guidance program in Japan 2008–2012

Author

Iseki, Kunitoshi, Asahi, Koichi, Yamagata, Kunihiro, Fujimoto, Shouichi, Tsuruya, Kazuhiko, Narita, Ichiei, Konta, Tsuneo, Kasahara, Masato, Shibagaki, Yugo, Yoshida, Hisako, Moriyama, Toshiki, Kondo, Masahide, Iseki, Chiho, Watanabe, Tsuyoshi, and for the “Design of the comprehensive health care system for chronic kidney disease (CKD) based on the individual risk assessment by Specific Health Check”

Published
2017
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11. Underestimating chronic kidney disease by urine dipstick without serum creatinine as a screening tool in the general Japanese population

Author

Uchida, Daisuke, Kawarazaki, Hiroo, Shibagaki, Yugo, Yasuda, Takashi, Tominaga, Naoto, Watanabe, Tsuyoshi, Asahi, Koichi, Iseki, Kunitoshi, Iseki, Chiho, Tsuruya, Kazuhiko, Yamagata, Kunihiro, Moriyama, Toshiki, Narita, Ichiei, Fujimoto, Shoichi, Konta, Tsuneo, Kondo, Masahide, Kasahara, Masato, and Kimura, Kenjiro

Published
2015
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12. Comparison of predictive value for first cardiovascular event between Japanese GFR equation and coefficient-modified CKD-EPI equation

Author

Terawaki, Hiroyuki, Nakayama, Masaaki, Asahi, Koichi, Kakamu, Takeyasu, Hayakawa, Takehito, Iseki, Kunitoshi, Kimura, Kenjiro, Moriyama, Toshiki, Yamagata, Kunihiro, Narita, Ichiei, Fujimoto, Shouichi, Tsuruya, Kazuhiko, Konta, Tsuneo, Kondo, Masahide, Kurahashi, Issei, Ohashi, Yasuo, Fukushima, Tetsuhito, and Watanabe, Tsuyoshi

Published
2015
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48. A case report of progressive multifocal leukoencephalopathy during steroid treatment for ANCA-associated renal vasculitis.

Author

Tawara T, Kai H, Kageyama M, Akiyama T, Matsunaga T, Sakuma A, Ishii R, Tsunoda R, Kawamura T, Fujita A, Kaneko S, Morito N, Saito C, Usui J, and Yamagata K

Subjects
Administration, Intravenous, Aged, 80 and over, Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis complications, Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis immunology, Antibodies, Antineutrophil Cytoplasmic immunology, Diagnosis, Differential, Fatal Outcome, Female, Glomerulonephritis immunology, Humans, Immunocompromised Host, JC Virus genetics, JC Virus immunology, Leukoencephalopathy, Progressive Multifocal cerebrospinal fluid, Leukoencephalopathy, Progressive Multifocal etiology, Leukoencephalopathy, Progressive Multifocal virology, Magnetic Resonance Imaging methods, Peroxidase metabolism, Severity of Illness Index, Steroids administration & dosage, Steroids therapeutic use, Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis drug therapy, Glomerulonephritis diagnosis, Leukoencephalopathy, Progressive Multifocal diagnosis, Steroids adverse effects
Abstract

Case Report: an 80-year-old woman presented with rapidly progressive glomerulonephritis and was admitted to our hospital. Myeloperoxidase-specific antineutrophil cytoplasmic antibody (MPO-ANCA) was positive. We diagnosed ANCA-associated renal vasculitis (ANCA-RV). Treatment was initiated with intravenous methylprednisolone pulse therapy, followed by prednisolone (PSL) at 30 mg/day. We gradually reduced the PSL dose to 7.5 mg/day over 6 months. At that time, the patient developed disturbances of consciousness which progressed subacutely. MRI revealed regions of patchy white matter with an increased signal on T2-weighted, fluid attenuated inversion recovery (FLAIR) sequences and diffusion-weighted sequences. JC virus DNA was detected in the cerebrospinal fluid (CSF) by polymerase chain reaction (PCR), leading to a diagnosis of progressive multifocal leukoencephalopathy (PML). PML is a rare infectious demyelinating disease of the central nervous system caused by JC virus infection, occurring in highly immunosuppressed individuals such as HIV-infected patients and patients using some biological agents, and having a very poor prognosis. In the present case, PML may have been associated with steroid use, although there are very few case reports of PML in patients taking only steroids. We report progressive multifocal leukoencephalopathy during steroid treatment of ANCA-RV. When patients show progressive disturbance of consciousness during treatment for ANCA-RV, we need to take PML into consideration for differential diagnosis.

Published
2020
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49. Kidney transplant patient with immunoglobulin A nephropathy subsequently diagnosed as concurrent autosomal dominant polycystic kidney disease during 17-year follow-up.

Author

Usui J, Kai H, Kaneko S, Takahashi-Kobayashi M, Hagiwara M, Takahashi K, Oda T, and Yamagata K

Subjects
Adolescent, Adult, Follow-Up Studies, Glomerulonephritis, IGA complications, Humans, Kidney diagnostic imaging, Kidney pathology, Kidney Diseases, Cystic pathology, Kidney Failure, Chronic etiology, Male, Polycystic Kidney, Autosomal Dominant complications, Polycystic Kidney, Autosomal Dominant genetics, Polycystic Kidney, Autosomal Dominant pathology, Proteinuria diagnosis, Proteinuria etiology, Tomography, X-Ray Computed methods, Glomerulonephritis, IGA diagnosis, Kidney Failure, Chronic surgery, Kidney Transplantation methods, Polycystic Kidney, Autosomal Dominant diagnosis
Abstract

A 14-year-old Japanese boy was diagnosed with immunoglobulin A nephropathy resulting in end-stage kidney disease (ESKD). He underwent ABO-compatible living kidney transplantation from his father at the age of 27. In the process of selecting a donor before the transplantation, it turned out that his mother had polycystic kidneys and that her family had a history of hypertension and cerebrovascular diseases. The patient himself also had bilateral multiple kidney cysts, with a normal-sized kidney, confusing us to make the diagnosis of acquired cystic kidney disease (ACKD) or ADPKD difficult at that point. Seventeen years later, his native kidneys showed bilateral swelling with multiple cysts. This, along with the histories of his mother and her relatives and with the existence of multiple liver cysts, led us to confirm the diagnosis of autosomal dominant polycystic kidney disease, not of ACKD. Contrary to previous studies that have suggested the size of cysts both in ADPKD and ACKD reduced with time, the present case showed an increase of 3.0% per year in total kidney volume (TKV) by computed tomography. It suggested the possibility that TKV, after decreasing in the relatively early stage after transplantation, may later increase in the long term after ESKD due to another kidney injury.

Published
2020
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