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We study global dynamics of an SIR model with vaccination, where we assume that individuals respond differently to dynamics of the epidemic. Their heterogeneous response is modeled by the Preisach hysteresis operator. We present a condition for the global stability of the infection-free equilibrium state. If this condition does not hold true, the model has a connected set of endemic equilibrium states characterized by different proportion of infected and immune individuals. In this case, we show that every trajectory converges either to an endemic equilibrium or to a periodic orbit. Under additional natural assumptions, the periodic attractor is excluded, and we guarantee the convergence of each trajectory to an endemic equilibrium state. The global stability analysis uses a family of Lyapunov functions corresponding to the family of branches of the hysteresis operator. [ABSTRACT FROM AUTHOR]

We analyze a simple macroeconomic model where rational inflation expectations are replaced by a boundedly rational, and genuinely sticky, response to changes in the actual inflation rate. The stickiness is introduced in a novel way using a mathematical operator that is amenable to rigorous analysis. We prove that, when exogenous noise is absent from the system, the unique equilibrium of the rational expectations model is replaced by an entire line segment of possible equilibria with the one chosen depending, in a deterministic way, upon the previous states of the system. The agents are sufficiently far-removed from the rational expectations paradigm that problems of indeterminacy do not arise. The response to exogenous noise is far more subtle than in a unique equilibrium model. After sufficiently small shocks the system will indeed revert to the same equilibrium but larger ones will move the system to a different one (at the same model parameters). The path to this new equilibrium may be very long with a highly unpredictable endpoint. At certain model parameters exogenously-triggered runaway inflation can occur. Finally, we analyze a variant model in which the same form of sticky response is introduced into the interest rate rule instead. [ABSTRACT FROM AUTHOR]

We propose a new epidemiological model, based on the classical SIR model, taking additionally into account a switching prevention strategy. The model has two distinct thresholds that determine the beginning and the end of an intervention and two different transmission rates. We study the global dynamics of the proposed two-dimensional model. [ABSTRACT FROM AUTHOR]

Memory allows organisms to forecast the future on the basis of experience, and thus, in some form, is important for the development of flexible adaptive behavior by animal communities. To model memory, we use the concept of hysteresis, which mathematically is described by the Preisach operator. As a case study, we consider anti-predator adaptation in the classic Lotka-Volterra predator-prey model. Despite its simplicity, the model allows us to naturally incorporate essential features of an adaptive system and memory. Our analysis and simulations show that a system with memory can have a continuum of equilibrium states with non-trivial stability properties. The main factor that determines the actual equilibrium state to which a trajectory converges is the maximal number achieved by the population of predator along this trajectory. [ABSTRACT FROM AUTHOR]

Seasonality is a complex force in nature that affects multiple processes in wild animal populations. In particular, seasonal variations in demographic processes may considerably affect the persistence of a pathogen in these populations. Furthermore, it has been long observed in computer simulations that under seasonal perturbations, a host-pathogen system can exhibit complex dynamics, including the transition to chaos, as the magnitude of the seasonal perturbation increases. In this paper, we develop a seasonally perturbed Susceptible-Infected-Recovered model of avian influenza in a seabird colony. Numerical simulations of the model give rise to chaotic recurrent epidemics for parameters that reflect the ecology of avian influenza in a seabird population, thereby providing a case study for chaos in a host- pathogen system. We give a computer-assisted exposition of the existence of chaos in the model using methods that are based on the concept of topological hyperbolicity. Our approach elucidates the geometry of the chaos in the phase space of the model, thereby offering a mechanism for the persistence of the infection. Finally, the methods described in this paper may be immediately extended to other infections and hosts, including humans. [ABSTRACT FROM AUTHOR]