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Your search keyword '"Wu, Weifeng"' showing total 28 results
Start Over Author "Wu, Weifeng" Publisher springer science and business media llc
28 results on '"Wu, Weifeng"'

5. Disturbance of plateau zokor-made mound stimulates plant community regeneration in the Qinghai-Tibetan Plateau, China

Author

WU Weifeng, Zhou SaiXia, Tang Zhongbin, Zhang Jiaxin, Peng Yansong, David Gallacher, Xiang Zeyu, Wang Jingxuan, and Arvind Bhatt

Subjects
geography, Plateau, geography.geographical_feature_category, Disturbance (geology), biology, Ecology, Plant community, Management, Monitoring, Policy and Law, biology.organism_classification, Qinghai tibetan plateau, Environmental science, Zokor, China, Regeneration (ecology), Earth-Surface Processes, Water Science and Technology
Published
2021

9. Risk prediction in pulmonary hypertension due to chronic heart failure: incremental prognostic value of pulmonary hemodynamics

Author

Quan, Ruilin, primary, Huang, Shian, additional, Pang, Lingpin, additional, Shen, Jieyan, additional, Wu, Weifeng, additional, Tang, Fangming, additional, Zhu, Xiulong, additional, Su, Weiqing, additional, Sun, Jingzhi, additional, Yu, Zaixin, additional, Wang, Lemin, additional, Zhu, Xianyang, additional, Xiong, Changming, additional, and He, Jianguo, additional

Published
2022
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15. NOD1/RIP2 signalling enhances the microglia-driven inflammatory response and undergoes crosstalk with inflammatory cytokines to exacerbate brain damage following intracerebral haemorrhage in mice

Author

Wang, Miao, primary, Ye, Xinchun, additional, Hu, Jinxia, additional, Zhao, Qiuchen, additional, Lv, Bingchen, additional, Ma, Weijing, additional, Wang, Weiwei, additional, Yin, Hanhan, additional, Hao, Qi, additional, Zhou, Chao, additional, Zhang, Tao, additional, Wu, Weifeng, additional, Wang, Yan, additional, Zhou, Mingyue, additional, Zhang, Cong-hui, additional, and Cui, Guiyun, additional

Published
2020
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23. Distinct different expression of Th17 and Th9 cells in coxsackie virus B3-induced mice viral myocarditis

Author

Wu Weifeng, Yang Fan, Pang Yu, Huang Yanlan, Kong Qing, and Yan Yuluan

Subjects
Male, Viral Myocarditis, medicine.medical_treatment, Coxsackievirus Infections, Spleen, Biology, Virus, lcsh:Infectious and parasitic diseases, Flow cytometry, Rodent Diseases, Pathogenesis, Mice, T-Lymphocyte Subsets, Virology, medicine, Animals, lcsh:RC109-216, Interleukin 9, Enterovirus, Mice, Inbred BALB C, medicine.diagnostic_test, Research, Myocardium, Interleukin-17, Interleukin-9, Flow Cytometry, Disease Models, Animal, Myocarditis, Infectious Diseases, medicine.anatomical_structure, Cytokine, Immunology, Interleukin 17
Abstract

Background Recently, a new subset of CD4+T helper(Th) cell that predominantly secret cytokine interleukin-9(IL-9) is identified, termed Th9 cell. It has been reported to participate in tissue inflammation and autoimmune responses, and induce disease which differed from Th17 cells. Th17 cells have been shown to play a critical role in viral myocarditis (VMC), but whether Th9 cells are involved in the pathogenesis of VMC remains unclear. Results BALB/c mice were intraperitoneally (i.p) injected with coxsackie virus B3(CVB3) for establishing VMC models. Control mice were treated with phosphate-buffered saline i.p. On day 0,7,14,21,28,35,42 after injection, myocardial histopathological changes were evaluated by hematoxylin-eosin staining. Splenic Th17 and Th9 cells subsets were analyzed by flow cytometry. And cardiac IL-17, IL-9 mRNA were measured by semi-quantitative reverse transcription-PCR and nested PCR, respectively. Results showed the levels of Th17 cells and IL-17 mRNA obviously increased in VMC mice on 7 day after infection, peaked on day 28, and highly persisted to at least day 42 (p < 0.05). While the frequencies of Th9 cells and IL-9 mRNA showed no significant difference between VMC and control group throughout the course of the experiment(p > 0.05). Conclusions It was differentiated Th17 but not Th9 cells significantly elevated in the development of CVB3-induced VMC. The microenvironment of VMC seemed to contribute to the differentiation and proliferation of Th17 rather than Th9 cells. Our preliminary data implied Th9 cells could not protect against VMC nor promote the disease.

Published
2011

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