43 results on '"Yewdell, Jonathan W."'
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2. The extracellular tail of IFITM3 promotes SARS-CoV-2 entry into cells and is diversifying in primates
3. MLN4924 Inhibits Defective Ribosomal Product Antigen Presentation Independently of Direct NEDDylation of Protein Antigens
4. Detection of SARS-CoV-2 specific antibodies in breastfeeding infant stool following mother’s COVID-19 vaccination
5. Myosin 18A is a novel checkpoint regulator in B cell differentiation and antibody-mediated immunity
6. Cutting Edge: Myosin 18A Is a Novel Checkpoint Regulator in B Cell Differentiation and Antibody-Mediated Immunity
7. Targeted protein degradation via NEDD8 conjugation does not enhance direct MHC class I antigen presentation.
8. Targeted protein degradation via NEDD8 conjugation does not enhance direct MHC class I antigen presentation
9. Influenza A Virus Infection Induces Viral and Cellular Defective Ribosomal Products Encoded by Alternative Reading Frames
10. Lymph node conduits transport virions for rapid T cell activation
11. Evaluation of epitope specificity in the immune response to PR8 H1N1 influenza virus infection in neonatal and adult African Green monkeys
12. Neuraminidase inhibition governs protection efficacy of broadly neutralizing antiinfluenza hemagglutinin stem antibodies
13. Correction: Influenza A Virus Negative Strand RNA Is Translated for CD8+ T Cell Immunosurveillance
14. Influenza A Virus Negative Strand RNA Is Translated for CD8+ T Cell Immunosurveillance
15. Varied Role of Ubiquitylation in Generating MHC Class I Peptide Ligands
16. Wild mouse gut microbiome protects laboratory mice against lethal influenza virus infection and colorectal cancer
17. Protein Translation Activity: A New Measure of Host Immune Cell Activation
18. Defining Viral Defective Ribosomal Products: Standard and Alternative Translation Initiation Events Generate a Common Peptide from Influenza A Virus M2 and M1 mRNAs
19. Mixed Proteasomes Function To Increase Viral Peptide Diversity and Broaden Antiviral CD8+ T Cell Responses
20. Distinct Pathways Generate Peptides from Defective Ribosomal Products for CD8+ T Cell Immunosurveillance
21. RNA Polymerase II Inhibitors Dissociate Antigenic Peptide Generation from Normal Viral Protein Synthesis: A Role for Nuclear Translation in Defective Ribosomal Product Synthesis?
22. Correction: Efficient Cross-Priming of Antiviral CD8+ T Cells by Antigen Donor Cells Is GRP94 Independent
23. Unexpected Role for the Immunoproteasome Subunit LMP2 in Antiviral Humoral and Innate Immune Responses
24. Defective Ribosomal Products Are the Major Source of Antigenic Peptides Endogenously Generated from Influenza A Virus Neuraminidase
25. Cutting Edge: Sympathetic Nervous System Increases Proinflammatory Cytokines and Exacerbates Influenza A Virus Pathogenesis
26. Efficient Cross-Priming of Antiviral CD8+ T Cells by Antigen Donor Cells Is GRP94 Independent
27. Quantitating T Cell Cross-Reactivity for Unrelated Peptide Antigens
28. Terminal Deoxynucleotidyl Transferase Establishes and Broadens Antiviral CD8+ T Cell Immunodominance Hierarchies
29. Cutting Edge: MHC Class I–Ly49 Interaction Regulates Neuronal Function
30. Signaling pathways involved in foreign antigen expression by a recombinant vaccinia virus (89.38)
31. CD8αα-Mediated Intraepithelial Lymphocyte Snatching of Thymic Leukemia MHC Class Ib Molecules In Vitro and In Vivo
32. Tight Linkage between Translation and MHC Class I Peptide Ligand Generation Implies Specialized Antigen Processing for Defective Ribosomal Products
33. HLA-A*0201, HLA-A*1101, and HLA-B*0702 Transgenic Mice Recognize Numerous Poxvirus Determinants from a Wide Variety of Viral Gene Products
34. Regulatory T Cells Suppress CD8+ T Cell Responses Induced by Direct Priming and Cross-Priming and Moderate Immunodominance Disparities
35. Reversal in the Immunodominance Hierarchy in Secondary CD8+ T Cell Responses to Influenza A Virus: Roles for Cross-Presentation and Lysis-Independent Immunodomination
36. Inhibitory Effects of Cytomegalovirus Proteins US2 and US11 Point to Contributions from Direct Priming and Cross-Priming in Induction of Vaccinia Virus-Specific CD8+ T Cells
37. Recycling CD1d1 Molecules Present Endogenous Antigens Processed in an Endocytic Compartment to NKT Cells
38. Multiple Paths for Activation of Naive CD8+ T Cells: CD4-Independent Help
39. Cutting Edge: Recombinant Adenoviruses Induce CD8 T Cell Responses to an Inserted Protein Whose Expression Is Limited to Nonimmune Cells
40. Multiple Antigen-Specific Processing Pathways for Activating Naive CD8+ T Cells In Vivo
41. Cutting Edge: Adenovirus E19 Has Two Mechanisms for Affecting Class I MHC Expression1
42. Assembly of MHC Class I Molecules with Biosynthesized Endoplasmic Reticulum-Targeted Peptides Is Inefficient in Insect Cells and Can Be Enhanced by Protease Inhibitors
43. Dissociation of Proteasomal Degradation of Biosynthesized Viral Proteins from Generation of MHC Class I-Associated Antigenic Peptides
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