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22 results on '"Conlon, Peter J"'

2. Renal transplant outcomes in patients with autosomal dominant tubulointerstitial kidney disease

Author

Cormican, Sarah, primary, Kennedy, Claire, additional, Connaughton, Dervla M., additional, O'Kelly, Patrick, additional, Murray, Susan, additional, Živná, Martina, additional, Kmoch, Stanislav, additional, Fennelly, Neil K., additional, Benson, Katherine A., additional, Conlon, Eoin T., additional, Cavalleri, Gianpiero L., additional, Foley, Claire, additional, Doyle, Brendan, additional, Dorman, Anthony, additional, Little, Mark A., additional, Lavin, Peter, additional, Kidd, Kendrah, additional, Bleyer, Anthony J., additional, and Conlon, Peter J., additional

Published
2020
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3. Post‐transplant malignancy in solid organ transplant recipients in Ireland, The Irish Transplant Cancer Group

Author

O'Neill, James Paul, primary, Sexton, Donal J., additional, O'Leary, Eamonn, additional, O'Kelly, Patrick, additional, Murray, Susan, additional, Deady, Sandra, additional, Daly, Fergus, additional, Williams, Yvonne, additional, Dean, Ben, additional, Fitzgerald, Conall, additional, Murad, Aizuri, additional, Mansoor, Nazish, additional, O'Neill, Jim O., additional, Egan, Jim, additional, Houlihan, Diarmaid D., additional, McCormick, P. Aiden, additional, Morris, Patrick G., additional, Ni Raghallaigh, Siona, additional, Little, Dilly, additional, Moloney, Fergal J., additional, and Conlon, Peter J., additional

Published
2019
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5. Do patient-reported measures of symptoms and health status predict mortality in hemodialysis? An assessment of POS-S Renal and EQ-5D

Author

Sexton, Donal J., primary, Lowney, Aoife C., additional, O'Seaghdha, Conall M., additional, Murphy, Marie, additional, O'Brien, Tony, additional, Casserly, Liam F., additional, McQuillan, Regina, additional, Plant, William D., additional, Eustace, Joseph A., additional, Kinsella, Sinead M., additional, and Conlon, Peter J., additional

Published
2016
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6. Urinary biomarkers of chronic allograft nephropathy

Author

Cassidy, Hilary, primary, Slyne, Jennifer, additional, O'Kelly, Patrick, additional, Traynor, Carol, additional, Conlon, Peter J., additional, Johnston, Olwyn, additional, Slattery, Craig, additional, Ryan, Michael P., additional, and McMorrow, Tara, additional

Published
2015
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10. A genome-wide association study of recipient genotype and medium-term kidney allograft function

Author

O'Brien, Robert P., primary, Phelan, Paul J., additional, Conroy, Judith, additional, O'Kelly, Patrick, additional, Green, Andrew, additional, Keogan, Mary, additional, O'Neill, Derek, additional, Jennings, Susan, additional, Traynor, Carol, additional, Casey, Jillian, additional, McCormack, Mark, additional, Conroy, Ronan, additional, Chubb, Anthony, additional, Ennis, Sean, additional, Shields, Denis C., additional, Cavalleri, Gianpiero L., additional, and Conlon, Peter J., additional

Published
2013
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14. Identification of β2-microglobulin as a urinary biomarker for chronic allograft nephropathy using proteomic methods

Author

Johnston, Olwyn, primary, Cassidy, Hilary, additional, O'Connell, Séin, additional, O'Riordan, Aisling, additional, Gallagher, William, additional, Maguire, Patricia B., additional, Wynne, Kieran, additional, Cagney, Gerard, additional, Ryan, Michael P., additional, Conlon, Peter J., additional, and McMorrow, Tara, additional

Published
2011
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15. Left versus right deceased donor renal allograft outcome

Author

Phelan, Paul J., primary, Shields, William, additional, O’Kelly, Patrick, additional, Pendergrass, Melissa, additional, Holian, John, additional, Walshe, Joseph J., additional, Magee, Colm, additional, Little, Dilly, additional, Hickey, David, additional, and Conlon, Peter J., additional

Published
2009
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21. The influence of socioeconomic status on patient survival on chronic dialysis

Author

Patrick O'Kelly, Declan G. deFreitas, Conall M. O'Seaghdha, Frank L. Ward, Peter J. Conlon, Howard Johnson, Trutz Haase, Jonathan Pratschke, Coilín ÓhAiseadha, Fionnuala Donohue, Ward Frank, L., O'Kelly, Patrick, Donohue, Fionnuala, O'Haiseadha, Coilín, Haase, Trutz, Pratschke, Jonathan, Defreitas Declan, G., Johnson, Howard, O'Seaghdha Conall, M., and Conlon Peter, J.

Subjects
Pediatrics, medicine.medical_specialty, Socioeconomic status, End-stage kidney disease, Patient survival, chronic dialysis, medicine.medical_treatment, medicine, Socioeconomic statu, Survival analysis, Dialysis, business.industry, Hazard ratio, social sciences, Hematology, medicine.disease, Confidence interval, Transplantation, Quartile, Nephrology, population characteristics, Hemodialysis, business, Demography, Kidney disease
Abstract

Socioeconomic status (SES) has been linked to worse end-stage kidney disease survival. The effect of SES on survival on chronic dialysis, including the impact of transplantation, was examined. A retrospective, observational study investigated the association of SES with dialysis patient survival, with censoring at time of transplantation. Adult patients commencing dialysis from 1990 to 2009 in an Irish tertiary center received a spatial SES score using the 2011 Pobal Haase-Pratschke Deprivation Index and were compared by quartile. Cox proportional hazard models and Kaplan–Meier survival analysis examined any association of SES with survival. The 1794 patients included had a median follow-up of 3.8 years. Patients in the lowest SES area quartile were significantly younger than the highest, mean age 56.7 vs. 59 years, P = 0.006, respectively. There was no association between SES area score and survival in an unadjusted model (hazard ratio [HR] 1.00, 95% confidence interval [CI] 0.99–1.01). Survival in the highest SES area quartile was superior to the lowest SES in a multivariable adjusted model including age, gender, and dialysis modality (HR 0.83, 95% CI 0.70–0.99, P = 0.04). These results were only mildly attenuated by censoring at time of transplantation (highest SES area quartile deprived vs. lowest SES area quartile, HR 0.85, 95% CI 0.70–1.03, P = 0.09). Superior patient survival was identified in the highest SES areas compared with the lowest following age-adjusted analyses, despite the older population in the most affluent areas. Further research should focus on identifying modifiable targets for intervention that account for this socioeconomic-related survival advantage.

Published
2015

22. Adult presentation of a rare mitochondrial tRNA Val gene mutation-an expanding clinical phenotype.

Author

Joyce E, Ali M, Richard G, Kelly S, Martin F, Conlon PJ, Whelehan A, Ng YS, and Lefter S

Abstract

Background and Purpose: Late-onset mitochondrial disorders are diagnostically challenging with significant heterogeneity in disease presentation. A case is reported of a 67-year-old gentleman who presented with a 3-month history of seizures, recurrent encephalopathy, ataxia and weight loss, preceded by recent initiation of haemodialysis for end-stage chronic kidney disease., Methods: Extensive work-up including serological, cerebrospinal fluid, magnetic resonance imaging and electroencephalography was performed. Whole exome sequencing and muscle biopsy confirmed the diagnosis., Results: Magnetic resonance imaging brain demonstrated a single non-enhancing T2 fluid attenuated inversion recovery hyperintense cortical/subcortical signal change in the right temporal lobe and cerebellar atrophy. Given the subacute presentation of uncertain aetiology, he was empirically treated for autoimmune/paraneoplastic encephalitis. Despite radiological resolution of the cortical abnormality 2 weeks later, there was no clinical improvement. Further collateral history unveiled a mildly ataxic gait and longstanding hearing loss suggestive of a genetic cause. Whole exome sequencing revealed a likely pathogenic, heteroplasmic mitochondrial DNA variant in the MT-TV gene, m.1659T>C, present at higher levels of heteroplasmy in muscle (91%) compared to other mitotic tissues. A high fat/protein diet and multivitamins including co-enzyme Q10 were commenced. Treatment of the nutritional deficiency and avoidance of intermittent fasting due to unreliable oral intake secondary to encephalopathy probably contributed to the clinical improvement seen over the ensuing few months, with resolution of his encephalopathy and return to his baseline gait and weight., Conclusion: An adult case is reported with an acute neurological presentation mimicking encephalitis, caused by a heteroplasmic m.1659T>C MT-TV variant, previously reported once in a child who displayed a different clinical phenotype., (© 2024 The Author(s). European Journal of Neurology published by John Wiley & Sons Ltd on behalf of European Academy of Neurology.)

Published
2024
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