18 results on '"Crouch Peter J"'
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2. Iron overload and impaired iron handling contribute to the dystrophic pathology in models of Duchenne muscular dystrophy
3. SLN124, a GalNac‐siRNA targeting transmembrane serine protease 6, in combination with deferiprone therapy reduces ineffective erythropoiesis and hepatic iron‐overload in a mouse model of β‐thalassaemia
4. Iron accumulation in skeletal muscles of old mice is associated with impaired regeneration after ischaemia–reperfusion damage
5. Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic
6. Cu II (atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease
7. The Alzheimer’s therapeutic PBT2 promotes amyloid‐β degradation and GSK3 phosphorylation via a metal chaperone activity
8. A domain level interaction network of amyloid precursor protein and Aβ of Alzheimer's disease
9. O1-04-06: Zinc and oxidative stress induces depletion and aggregation of endogenous TDP-43
10. P3-242: The role of autophagy in potentially therapeutic copper complexes
11. Restored degradation of the Alzheimer’s amyloid-β peptide by targeting amyloid formation
12. Membrane-targeted strategies for modulating APP and Aβ-mediated toxicity
13. P2‐292: Second generation MPAC PBT2 inhibits tau phosphorylation and promotes Aβ degradation
14. P2-314: Neuroprotective metallo-complexes activate EGFR and inhibit amyloid-beta peptide accumulation
15. P1‐483: Abeta degradation by matrix metalloproteinase 2 is prevented by factors that induce Abeta amyloid formation
16. Investigating copper-regulated protein expression in Menkes fibroblasts using antibody microarrays
17. The modulation of metal bio-availability as a therapeutic strategy for the treatment of Alzheimer's disease
18. Copper‐dependent inhibition of cytochrome c oxidase by Aβ1−42 requires reduced methionine at residue 35 of the Aβ peptide
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