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25 results on '"Lu, Xiaolin"'

1. Inhibition of HDAC2 sensitises antitumour therapy by promoting NLRP3/GSDMD‐mediated pyroptosis in colorectal cancer

Author

Guan, Xin, primary, Liu, Ruiqi, additional, Wang, Bojun, additional, Xiong, Ruxin, additional, Cui, Luying, additional, Liao, Yuanyu, additional, Ruan, Yuli, additional, Fang, Lin, additional, Lu, Xiaolin, additional, Yu, Xuefan, additional, Su, Dan, additional, Ma, Yue, additional, Dang, Tianjiao, additional, Chen, Zhuo, additional, Yao, Yuanfei, additional, Liu, Chao, additional, and Zhang, Yanqiao, additional

Published
2024
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6. Interlaminar fracture toughness, adhesion and mechanical properties of MWCNT–glass fiber fabric composites: Effect of MWCNT aspect ratios

Author

Pengpeng Duan, Shaohua Zeng, Lu Yang, Lu Xiaolin, Mingxia Shen, and Yijiao Xue

Subjects
Materials science, Polymers and Plastics, Glass fiber, 02 engineering and technology, General Chemistry, Adhesion, 010402 general chemistry, 021001 nanoscience & nanotechnology, 01 natural sciences, 0104 chemical sciences, Fracture toughness, Materials Chemistry, Ceramics and Composites, Composite material, 0210 nano-technology
Published
2018

10. Digital planning workflow for partial maxillectomy using an osteotomy template and immediate rehabilitation of maxillary Brown II defects with prosthesis

Author

Wang, Yang, primary, Yang, Xudong, additional, Gan, Ronglin, additional, Liu, Huifen, additional, Wu, Guofeng, additional, Yu, Qing, additional, Wang, Zhiyong, additional, Lu, Xiaolin, additional, Jing, Jianlong, additional, Ma, Wenjie, additional, Quan, Yi, additional, Sun, Ziang, additional, Fan, Lei, additional, and Wang, Yuxin, additional

Published
2019
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11. The effect of folic acid deficiency on FGF pathwayviaBrachyury regulation in neural tube defects

Author

Chang, Shaoyan, primary, Lu, Xiaolin, additional, Wang, Shan, additional, Wang, Zhigang, additional, Huo, Junsheng, additional, Huang, Jian, additional, Shangguan, Shaofang, additional, Li, Shen, additional, Zou, Jizhen, additional, Bao, Yihua, additional, Guo, Jin, additional, Wang, Fang, additional, Niu, Bo, additional, Zhang, Ting, additional, Qiu, Zhiyong, additional, Wu, Jianxin, additional, and Wang, Li, additional

Published
2018
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19. MicroRNA-191, by promoting the EMT and increasing CSC-like properties, is involved in neoplastic and metastatic properties of transformed human bronchial epithelial cells

Author

Xu, Wenchao, primary, Ji, Jie, additional, Xu, Yuan, additional, Liu, Yawei, additional, Shi, Le, additional, Liu, Yi, additional, Lu, Xiaolin, additional, Zhao, Yue, additional, Luo, Fei, additional, Wang, Bairu, additional, Jiang, Rongrong, additional, Zhang, Jianping, additional, and Liu, Qizhan, additional

Published
2014
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25. Association between PTCH1 polymorphisms and risk of neural tube defects in a Chinese population.

Author

Wang Z, Wang L, Shangguan S, Lu X, Chang S, Wang J, Zou J, Wu L, Zhang T, and Luo Y

Subjects
DNA Methylation, Female, Genetic Association Studies, Genotype, Hedgehog Proteins genetics, Humans, Logistic Models, Patched Receptors, Patched-1 Receptor, Polymorphism, Single Nucleotide genetics, Risk Factors, Signal Transduction genetics, Asian People genetics, Neural Tube Defects epidemiology, Neural Tube Defects genetics, Receptors, Cell Surface genetics, Signal Transduction physiology
Abstract

Background: SHH signaling pathway plays an important role in the formation of the neural plate and is involved in the regulation of the dorsoventral (DV) axis of the neural tube. Some neural tube defects (NTDs) may be caused through overactivation of the SHH signaling pathway. The PTCH1 gene, encoding a negative regulator of SHH signaling, affects neural tube closure in animal models. However, in humans, the relationship between single nucleotide polymorphisms (SNPs) of the PTCH1 gene and neural tube defects remains unclear., Methods: MassARRAY®GENOTYPER™ was used to genotype 18 SNPs of the PTCH1 gene in 187 NTDs and 212 control samples, to determine whether PTCH1 polymorphisms are related to NTDs. MassARRAY®EpiTYPER™ was performed to assess whether methylation modifications may be associated with SNP genotypes in this Chinese population., Result: Increased risk for spina bifida was observed with the G allele of c.3944C>T and the T allele of c.1729™2350G>A in female patients when compared to the normal control group. High methylation levels were detected in those controls bearing the G allele of c.3944C>T., Conclusion: In summary, polymorphisms of the PTCH1 gene may be genetic predisposing factors for spina bifida in the population studied. In addition, methylation modifications associated with the c.3944C>T polymorphism, may provide protection., (Copyright © 2013 Wiley Periodicals, Inc.)

Published
2013
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