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The drivetrain has an important impact on the stability and reliability of wind turbines operating in complex conditions, which belongs to the electromechanical coupling system. This work studies the two domains of research on drivetrain vibration analysis and control method in detail. The electromechanically coupled torsional vibration model is first established by considering the air gap magnetic field energy of the PMSG. Preliminary analysis in terms of Hamiltonian energy indicates that there is sufficient energy to sustain the exciting oscillation behaviors. Moreover, to reduce the vibration amplitude caused by wind speed or torsional stiffness, a novel adaptive fixed‐time control method is proposed to solve the issue of wind turbine drivetrain chaotic oscillation with uncertainties and disturbances. Furthermore, the proposed method not only can prevent the jumping and bifurcation of torsional vibration from happening, but also there are the advantages of the control scheme with high accuracy, fast convergence rate and strong robustness. For the fair comparison, three different control methods have been used to demonstrate the superiority of the control scheme by the simulation results. The research results can provide a theoretical basis for the parameter design and control of wind turbine drivetrain. [ABSTRACT FROM AUTHOR]

The tumor immune microenvironment has manifested a crucial correlation with tumor occurrence, development, recurrence, and metastasis. To explore the mechanisms intrinsic to osteosarcoma (OS) initiation and progression, this study synthesizes multiple single‐cell RNA sequencing data sets, constructing a comprehensive landscape of the OS microenvironment. Integrating single‐cell RNA sequencing with bulk RNA sequencing data has enabled the identification of a significant correlation between heightened expression of the fatty acid metabolism‐associated gene (C1QBP) and patient survival in OS. C1QBP not only amplifies the proliferation, migration, invasion, and anti‐apoptotic properties of OS but also instigates cisplatin resistance. Subsequent investigations suggest that C1QBP potentially promotes macrophage polarization from monocytes/macrophages toward M2 and M3 phenotypes. Consequently, C1QBP may emerge as a novel target for modulating OS progression and resistance therapy. [ABSTRACT FROM AUTHOR]