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154 results on '"Chen, Shao-Rui"'

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3. Calcineurin regulates synaptic Ca2+‐permeable AMPA receptors in hypothalamic presympathetic neurons via α2δ‐1‐mediated GluA1/GluA2 assembly.

13. α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic NMDA receptor hyperactivity in the nucleus accumbens.

15. Calcineurin Regulates Synaptic Plasticity and Nociceptive Transmission at the Spinal Cord Level.

20. α2δ‐1 protein promotes synaptic expression of Ca2+ permeable–AMPA receptors by inhibiting GluA1/GluA2 heteromeric assembly in the hypothalamus in hypertension.

39. Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension.

40. Presynaptic NMDA receptors control nociceptive transmission at the spinal cord level in neuropathic pain.

41. Endogenous transient receptor potential ankyrin 1 and vanilloid 1 activity potentiates glutamatergic input to spinal lamina I neurons in inflammatory pain.

42. μ‐Opioid receptors in primary sensory neurons are essential for opioid analgesic effect on acute and inflammatory pain and opioid‐induced hyperalgesia.

43. Mitogen‐activated protein kinase signaling mediates opioid‐induced presynaptic NMDA receptor activation and analgesic tolerance.

44. Increased α2δ‐1–NMDA receptor coupling potentiates glutamatergic input to spinal dorsal horn neurons in chemotherapy‐induced neuropathic pain.

45. Regulating nociceptive transmission by VGluT2‐expressing spinal dorsal horn neurons.

47. Differential responses of regional sympathetic activity and blood flow to visceral afferent stimulation

48. Cardiac interstitial bradykinin release during ischemia is enhanced by ischemic preconditioning

49. α2δ‐1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension.

50. Endogenous nitric oxide inhibits spinal NMDA receptor activity and pain hypersensitivity induced by nerve injury.

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