23 results on '"Karbowniczek, Magdalena"'
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2. Upregulation of acid ceramidase contributes to tumor progression in Tuberous Sclerosis Complex
3. Tumorigenesis in tuberous sclerosis complex is autophagy and p62/sequestosome 1 (SQSTM1)-dependent
4. Abstract 141: Role of TP53 codon 72 polymorphism in macrophage polarization
5. Estrogen Promotes the Survival and Pulmonary Metastasis of Tuberin-Null Cells
6. The evolutionarily conserved TSC/Rheb pathway activates Notch in tuberous sclerosis complex and Drosophila external sensory organ development
7. The Role of Complement in Angiogenesis
8. Recurrent Lymphangiomyomatosis after Transplantation: Genetic Analyses Reveal a Metastatic Mechanism
9. The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis
10. Mutational Analysis of the von Hippel Lindau Gene in Clear Cell Renal Carcinomas from Tuberous Sclerosis Complex Patients
11. Complementing Cancer Metastasis
12. Notch transactivates Rheb to maintain the multipotency of TSC-null cells
13. Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
14. The TSC1/2 Complex Controls Drosophila Pigmentation through TORC1-Dependent Regulation of Catecholamine Biosynthesis
15. mTOR Is Activated in the Majority of Malignant Melanomas
16. Constitutive mTOR activation in TSC mutants sensitizes cells to energy starvation and genomic damage via p53
17. Rheb Inhibits C-Raf Activity and B-Raf/C-Raf Heterodimerization
18. Regulation of B-Raf Kinase Activity by Tuberin and Rheb Is Mammalian Target of Rapamycin (mTOR)-independent
19. Renal Angiomyolipomas from Patients with Sporadic Lymphangiomyomatosis Contain Both Neoplastic and Non-Neoplastic Vascular Structures
20. Tumorigenesis in tuberous sclerosis complex is autophagy and p62/sequestosome 1 (SQSTM 1)-dependent.
21. Constitutive mTOR activation in TSC mutants sensitizes cells to energy starvation and genomic damage via p53.
22. Diet and exercise as potential anticonvulsant therapies in mouse seizure models
23. Extracellular vesicles regulate metastable phenotypes of lymphangioleiomyomatosis cells via shuttling ATP synthesis to pseudopodia and activation of integrin adhesion complexes.
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