42 results on '"Turcq, Béatrice"'
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2. In vivo vulnerabilities to GPX4 and HDAC inhibitors in drug-persistent versus drug-resistant BRAFV600E lung adenocarcinoma
3. Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells
4. CRISPR Base Editing to Create Potential Charcot–Marie–Tooth Disease Models with High Editing Efficiency: Human Induced Pluripotent Stem Cell Harboring SH3TC2 Variants.
5. Novel analytical methods to interpret large sequencing data from small sample sizes
6. Loss of mutL homolog-1 (MLH1) expression promotes acquisition of oncogenic and inhibitor-resistant point mutations in tyrosine kinases
7. ABT-737 increases tyrosine kinase inhibitor–induced apoptosis in chronic myeloid leukemia cells through XIAP downregulation and sensitizes CD34+ CD38− population to imatinib
8. ANTIMETABOLIC COOPERATIVITY WITH THE CLINICALLY-APPROVED L-ASPARAGINASE AND TYROSINE KINASE INHIBITORS TO ERADICATE CML STEM CELLS
9. Inactivation of the Podospora anserina Vegetative Incompatibility Locus het-c, Whose Product Resembles a Glycolipid Transfer Protein, Drastically Impairs Ascospore Production
10. A Protein Required for RNA Processing and Splicing in Neurospora Mitochondria is Related to Gene Products Involved in Cell Cycle Protein Phosphatase Functions
11. A genome-scale CRISPR knock-out screen in chronic myeloid leukemia identifies novel drug resistance mechanisms along with intrinsic apoptosis and MAPK signaling
12. Inhibition of DDR1 enhances in vivo chemosensitivity in KRAS-mutant lung adenocarcinoma
13. In vivovulnerabilities to GPX4 and HDAC inhibitors in drug-persistent versus drug-resistant BRAFV600Elung adenocarcinoma
14. Additional file 7: of Novel analytical methods to interpret large sequencing data from small sample sizes
15. The Expression of Myeloproliferative Neoplasm-Associated Calreticulin Variants Depends on the Functionality of ER-Associated Degradation
16. CRISPR-Cas9 genome editing induces megabase-scale chromosomal truncations
17. Dasatinib-Loaded Erythrocytes Trigger Apoptosis in Untreated Chronic Myelogenous Leukemic Cells
18. Vegetative incompatibility in filamentous fungi: a roundabout way of understanding the phenomenon
19. Killer immunoglobulin‐like receptor genotypes and chronic myeloid leukemia outcomes after imatinib cessation for treatment‐free remission.
20. Loss of mutL homolog-1 (MLH1) expression promotes acquisition of oncogenic and inhibitor-resistant point mutations in tyrosine kinases.
21. Identification of Imatinib-Sensitizing Genes in Chronic Myeloid Leukemia with a Genome-Scale Crispr Knock-out Screen
22. A Single Nucleotide Polymorphism in cBIM Is Associated with a Slower Achievement of Major Molecular Response in Chronic Myeloid Leukaemia Treated with Imatinib
23. Multidrug resistance gene (MDR1) polymorphisms are associated with major molecular responses to standard-dose imatinib in chronic myeloid leukemia
24. α-Defensin 1-3 And α-Defensin 4 as Predictive Markers of Imatinib Resistance and Relapse in CML Patients
25. Evidence that Resistance to Nilotinib May Be Due to BCR-ABL, Pgp, or Src Kinase Overexpression
26. Heat Shock Protein 70 over Expression Is Associated to Imatinib Resistance in Chronic Myelogenous Leukemia.
27. The Proteome Analysis of an Imatinib-Resistant Cell Line Identifies Changes in Molecular Chaperones Such as Heat Shock Proteins (Hsp): A Possible New Mechanism of Imatinib Resistance.
28. Under-Expression of LINE-1 Retrotransposons in Chronic Myeloid Leukemia Patients: Effect of Tyrosine Kinase Inhibitors
29. HET-E and HET-D Belong to a New Subfamily of WD40 Proteins Involved in Vegetative Incompatibility Specificity in the Fungus Podospora anserina
30. MOD-D, a Gα Subunit of the Fungus Podospora anserina, Is Involved in Both Regulation of Development and Vegetative Incompatibility
31. A Mutation in an HSP90 Gene Affects the Sexual Cycle and Suppresses Vegetative Incompatibility in the Fungus Podospora anserina
32. An additional copy of the adenylate cyclase-encoding gene relieves developmental defects produced by a mutation in a vegetative incompatibility-controlling gene in Podospora anserina
33. A gene responsible for vegetative incompatibility in the fungus Podospora anserina encodes a protein with a GTP-binding motif and Gβ homologous domain
34. repa, a repetitive and dispersed DNA sequence of the filamentous fungusPodospora anserina
35. Defensine Alpha1 as a New Marker of Imatinib Resistance in Chronic Myelogenous Leukemia
36. repa, a repetitive and dispersed DNA sequence of the filamentous fungus Podospora anserina.
37. Generation of Resistance Cell Lines to AMN107, a New Inhibitor of BCR-ABL and Its Effects on Cell Lines Sensitive and Resistant to Imatinib.
38. The ura5 gene of the filamentous fungus Podospora anserina: nucleotide sequence and expression in transformed strains
39. Etude des rétrotransposons LINE-1 dans la leucémie myéloïde chronique
40. Identification and characterisation of genetic polymorphisms associated to imatinib sensitivity in chronic myeloid leukemia
41. A genome-scale CRISPR knock-out screen in chronic myeloid leukemia identifies novel drug resistance mechanisms along with intrinsic apoptosis and MAPK signaling.
42. Dasatinib-Loaded Erythrocytes Trigger Apoptosis in Untreated Chronic Myelogenous Leukemic Cells: A Cellular Reservoir Participating in Dasatinib Efficiency.
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