50 results on '"Matsuo, Hidemasa"'
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2. Author Correction: A RUNX-targeted gene switch-off approach modulates the BIRC5/PIF1-p21 pathway and reduces glioblastoma growth in mice
3. A RUNX-targeted gene switch-off approach modulates the BIRC5/PIF1-p21 pathway and reduces glioblastoma growth in mice
4. Machine learning-based prediction of relapse in rheumatoid arthritis patients using data on ultrasound examination and blood test
5. Fusion partner–specific mutation profiles and KRAS mutations as adverse prognostic factors in MLL-rearranged AML
6. Transcriptome analysis offers a comprehensive illustration of the genetic background of pediatric acute myeloid leukemia
7. P527: DISTINCTIVE CLONAL EVOLUTION PATTERN AND PROGNOSTIC SIGNIFICANCE OF THE CLONALITY OF KRAS MUTATIONS IN KMT2A-REARRANGED ACUTE MYELOID LEUKEMIA
8. Pivotal role of DPYSL2A in KLF4-mediated monocytic differentiation of acute myeloid leukemia cells
9. RUNX1–Survivin Axis Is a Novel Therapeutic Target for Malignant Rhabdoid Tumors
10. Genetic regulation of the RUNX transcription factor family has antitumor effects
11. RUNX1 transactivates BCR‐ABL1 expression in Philadelphia chromosome positive acute lymphoblastic leukemia
12. Clonal Evolution Pattern and Prognostic Significance of Clonal Architecture in KMT2A-Rearranged Acute Myeloid Leukemia
13. Efficacy of a combination therapy targeting CDK4/6 and autophagy in a mouse xenograft model of t(8;21) acute myeloid leukemia
14. RUNX inhibitor suppresses graft‐versus‐host disease through targeting RUNX‐NFATC2 axis
15. Clinical significance of RAS pathway alterations in pediatric acute myeloid leukemia
16. RUNX-NFAT Axis As a Novel Therapeutic Target for AML and T Cell Immunity
17. KRAS mutations Frequently Coexist with High-Risk MLL Fusions and Are Independent Adverse Prognostic Factors in MLL-Rearranged Acute Myeloid Leukemia
18. Autophagy Inhibition Enhances CDK4/6 Inhibitor-Induced Apoptosis in t(8;21) Acute Myeloid Leukemia Cells
19. RUNX1 transactivates BCR‐ABL1 expression in Philadelphia chromosome positive acute lymphoblastic leukemia.
20. TXNIP induces growth arrest and enhances ABT263‐induced apoptosis in mixed‐lineage leukemia‐rearranged acute myeloid leukemia cells
21. The Detection of Minor Clones with Somatic KIT D816V Mutations Using Droplet Digital PCR in Pediatric De Novo AML: AML-05 Trial from the Japanese Pediatric Leukemia/Lymphoma Study Group
22. Clinical Features of Pediatric Acute Myeloid Leukemia with TP53 and CDKN2A/2B copy Number Alterations
23. Coexistence and Prognostic Significance of EVI1 Expression and Driver Mutations in KMT2A-Rearranged Acute Myeloid Leukemia
24. Recurrent Gene Mutations in Pediatric Patients with AML By Targeted Sequencing ―the Jccg Study, JPLSG AML-05―
25. レーザーマイクロダイセクションを用いた血液塗抹標本からの白血病細胞の純化)
26. EVI1 triggers metabolic reprogramming associated with leukemogenesis and increases sensitivity to L-asparaginase
27. Comprehensive Analysis of 343 Genes Using Targeted Sequencing Panel By Next-Generation Sequencer in 77 Pediatric AML Patients with Normal and Complex Karyotypes: Jccg Study, JPLSG AML-05
28. Recurrent Genomic Aberrations of D-Type Cyclins Are Therapeutic Targets of CDK4/6 Inhibitors in t(8;21) and MLL-Rearranged Acute Myeloid Leukemia
29. <平成29年度博士授与者 3>レーザーマイクロダイセクションを用いた血液塗抹標本からの白血病細胞の純化
30. Recurrent CCND3 mutations in MLL-rearranged acute myeloid leukemia
31. Progressive Restrictive Ventilatory Impairment in Idiopathic Diffuse Pulmonary Ossification
32. Autonomous feedback loop of RUNX1-p53-CBFB in acute myeloid leukemia cells
33. <活動報告>アメリカ臨床検査技師の国際資格International Medical Technologist, MT(ASCP^[i])取得に挑戦して
34. Purification of leukemic blast cells from blood smears using laser microdissection
35. Fusion partner–specific mutation profiles and KRASmutations as adverse prognostic factors in MLL-rearranged AML
36. Label-Free Cell Detection of Acute Leukemia Using High-Content Morphological Profiling in Flow
37. Suppression of Super-Enhancer-Driven TAL1Expression By KLF4 in T-Cell Acute Lymphoblastic Leukemia
38. Prognostic Significance of CXCR4 Overexpression in Pediatric Acute Myeloid Leukemia with Low-Risk: A Report from the Japanese Pediatric Leukemia/Lymphoma Study Group
39. Cell-Autonomous Feedback Loop of RUNX1-p53-CBFB in Acute Myeloid Leukemia Cells
40. Enhancement of neutrophil autophagy by an IVIG preparation against multidrug-resistant bacteria as well as drug-sensitive strains
41. Double CEBPA Mutations Are Not Associated With Favorable Clinical Outcome In Pediatric AML: A Report From The Japanese Pediatric Leukemia/Lymphoma Study Group (JPLSG)
42. KRASmutations Frequently Coexist with High-Risk MLLFusions and Are Independent Adverse Prognostic Factors in MLL-Rearranged Acute Myeloid Leukemia
43. Clinical Features of Pediatric Acute Myeloid Leukemia with TP53and CDKN2A/2Bcopy Number Alterations
44. Coexistence and Prognostic Significance of EVI1Expression and Driver Mutations in KMT2A-Rearranged Acute Myeloid Leukemia
45. Prognostic Significance of CXCR4Overexpression in Pediatric Acute Myeloid Leukemia with Low-Risk: A Report from the Japanese Pediatric Leukemia/Lymphoma Study Group
46. Double CEBPAMutations Are Not Associated With Favorable Clinical Outcome In Pediatric AML: A Report From The Japanese Pediatric Leukemia/Lymphoma Study Group (JPLSG)
47. Clinical significance of RAS pathway alterations in pediatric acute myeloid leukemia.
48. EVI1 triggers metabolic reprogramming associated with leukemogenesis and increases sensitivity to L-asparaginase.
49. The subtype-specific features of EVI1 and PRDM16 in acute myeloid leukemia.
50. EVI1 overexpression is a poor prognostic factor in pediatric patients with mixed lineage leukemia-AF9 rearranged acute myeloid leukemia.
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