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8. Design and Analysis of Tri-Band Conformal Monopolar Antenna

Author

Ren, Chaofan, primary, Geng, Junping, additional, Yang, Silei, additional, Zhou, Han, additional, Lu, Jingzheng, additional, Zhao, Rui, additional, Tang, Xudong, additional, Zhang, Yangzhou, additional, Han, Jiawei, additional, Su, Da, additional, Zhang, Ao, additional, Li, Haotian, additional, and Jin, Ronghong, additional

Published
2022
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12. A Novel Phase Mode Antenna With Triport and Its Application in a 1-D Generalized Phased Array to 2-D Scanning

Author

Wang, Kun, Geng, Junping, Yang, Silei, Lu, Jingzheng, Zhao, Rui, Tang, Xudong, Li, Xuepeng, Su, Da, Zhang, Ao, Li, Haotian, Li, Enyu, and Jin, Ronghong

Abstract

A novel triport phase mode antenna is proposed for wide-angle and polarization scanning, along with a 1-D linear generalized phased array (LGPA) for 2-D space (xo z, yo z, and xoy planes) scanning and polarization scanning. To produce continuous beam scanning characteristics, the phase difference ( $\Delta \varphi $ ) between the feed ports can be adjusted from 0° to 180°, which can generate different operating states of the odd mode, even mode, and hybrid modes. It is worth noting that each feed port can work independently, and any two or all feed ports can be combined to produce new beams. When the linear array is expanded based on the phase mode antenna and combined with the improved generalized principle of pattern multiplication (GPPM), it can realize continuous wide-angle scanning, 2-D scanning, and polarization scanning. Simulation and experiment results demonstrate that the proposed phase mode antenna achieves continuous wide-angle and polarization scanning performances, and the proposed 1-D LGPA realizes wide-angle scanning, 2-D scanning, and polarization scanning. This illustrates that the introduction of triport provides greater freedom for 1-D linear array scanning and offers an effective solution for the miniaturization of phased arrays and the design of multidimensional spatial scanning.

Published
2023
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15. The Ultra-Compact ELF Magneto-Mechanical Transmission Antenna With the Speed Modulated EM Signal Based on Three-Phase Induction Motor

Author

Han, Jiawei, primary, Geng, Junping, additional, Wu, Haobo, additional, Wang, Kun, additional, Zhou, Han, additional, Ren, Chaofan, additional, Hayat, Qaisar, additional, Zhao, Xiaonan, additional, Yang, Silei, additional, He, Chong, additional, Liang, Xianling, additional, Jin, Ronghong, additional, Ma, Bo, additional, and Shen, Jingshi, additional

Published
2021
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16. Glucocorticoid regulation of astrocytic fate and function

Author

Yu, Shuang, Yang, Silei, Holsboer, F., Sousa, Nuno, Almeida, O. F. X., and Universidade do Minho

Subjects
Aging, Neurogenesis, lcsh:Medicine, Apoptosis, Hippocampus, Receptors, Glucocorticoid, Molecular Cell Biology, Neurobiology of Disease and Regeneration, Animals, Cell Lineage, RNA, Messenger, lcsh:Science, Glucocorticoids, Biology, Cells, Cultured, Cell Proliferation, Cellular Stress Responses, Neurons, Psychiatry, Science & Technology, Cell Death, Caspase 3, Mood Disorders, Gene Expression Profiling, lcsh:R, Gene Expression Regulation, Developmental, Rats, Enzyme Activation, Phenotype, Mental Health, Animals, Newborn, Astrocytes, Culture Media, Conditioned, Cytokines, Medicine, lcsh:Q, Research Article, Neuroscience
Abstract

Glial loss in the hippocampus has been suggested as a factor in the pathogenesis of stress-related brain disorders that are characterized by dysregulated glucocorticoid (GC) secretion. However, little is known about the regulation of astrocytic fate by GC. Here, we show that astrocytes derived from the rat hippocampus undergo growth inhibition and display moderate activation of caspase 3 after exposure to GC. Importantly, the latter event, observed both in situ and in primary astrocytic cultures is not followed by either early- or late-stage apoptosis, as monitored by stage I or stage II DNA fragmentation. Thus, unlike hippocampal granule neurons, astrocytes are resistant to GC-induced apoptosis; this resistance is due to lower production of reactive oxygen species (ROS) and a greater buffering capacity against the cytotoxic actions of ROS. We also show that GC influence hippocampal cell fate by inducing the expression of astrocyte-derived growth factors implicated in the control of neural precursor cell proliferation. Together, our results suggest that GC instigate a hitherto unknown dialog between astrocytes and neural progenitors, adding a new facet to understanding how GC influence the cytoarchitecture of the hippocampus., S. Yu was supported by a fellowship from the Max Planck Society. This study was supported by the Max Planck Institute of Psychiatry and by grants from the German Academic Exchange Service (DAAD) and the Portuguese Rectors' Conference (CRUP), and an Integrated Project grant from the European Commission (CRESCENDO, Contract No. LSHM-CT-2005-01852). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.

Published
2011

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