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19. Comprehensive analysis of clinical and laboratory features of 440 published cases of Sjögren's syndrome and renal tubular acidosis.

Author

Sandhya, Pulukool

Subjects
*SJOGREN'S syndrome, *RENAL tubular transport disorders, *DIABETES insipidus, *FANCONI syndrome, *KIDNEY stones, *ACIDOSIS, *PATHOLOGICAL laboratories
Abstract

Aim: To describe the clinical and laboratory features of Sjögren's syndrome (SS) with renal tubular acidosis (RTA) from published literature. Methods: A systematic search of indexed publications in all languages till December 2021 identified cases of SS with RTA (SS‐RTA) and were included if either antibody (anti‐SSA/anti‐SSB) or salivary gland histopathology were positive. Results: There were 440 cases of SS‐RTA (63.9% from Asia, 95.5% women). The median (range) age was 37 (6‐78) years. Only 7.7% had a previous diagnosis of SS. Oral or ocular sicca symptoms were present in 63.7%. Positive ocular tests, oral tests, anti‐SSA, anti‐SSB and salivary gland histopathology were reported in 256/331 (77.3%), 123/128 (96%), 382/407 (93.9%), 298/379 (78.6%), and 246/268 (91.8%), respectively. Hypokalemic paralysis (HP) was the presenting feature in 63.6%; 25% had multiple episodes of HP and 8.4% had respiratory paralysis. Type 1, type 2, combined type 1 & 2, and type 4 RTA was seen in 388, 8, 38, and 3 patients, respectively. Proximal dysfunction and RTA complications were infrequently evaluated. Fanconi syndrome, nephrogenic diabetes insipidus, proteinuria, and low estimated glomerular filtration rate were found in 45, 21, 178, and 157, respectively. Nephrocalcinosis, renal stones, and osteomalacia were reported in 92, 79, and 72, respectively. Tubulointerstitial nephritis was found in 142 out of 152 renal biopsies. Conclusion: SS‐RTA is an early manifestation of SS characterized by younger age and subclinical sicca symptoms. Although evaluated less frequently, oral sicca signs and salivary gland biopsy have a high positive yield. HP is the most common presentation. RTA is mostly distal; proximal dysfunction and complications were infrequently assessed. [ABSTRACT FROM AUTHOR]

Published
2023
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20. Anti-C1q Antibody is Associated with Renal and Cutaneous Manifestations in Asian Indian Patients with Systemic Lupus Erythematosus

Author

Jayakanthan Kabeerdoss, Nikhil Gupta, Sandhya Pulukool, Hindhumathi Mohan, Gowri Mahasampath, and Debashish Danda

Subjects
anti nucleosome antibody, complement c4, lupus nephritis, Medicine
Abstract

Introduction: C1q play an important role in clearance of immune complexes and apoptotic cell debris. Impaired clearance leads to exposure of C1 native antigen and development of antiC1q antibody formation. Anti-C1q antibody is well studied in Systemic Lupus Erythematosus (SLE). Significance of anti-C1q Ab in Indian SLE patients and their clinical manifestations is not clear. Aim: The aim of this study was to investigate associations between anti-C1q antibody and clinical as well as serological markers of SLE. Materials and Methods: Retrospective study of SLE patients fulfilling either American College of Rheumatology (ACR) 1990 or Systemic Lupus International Collaborating Clinics (SLICC) 2012 classification criteria were recruited from inpatients and outpatients services of the Clinical immunology and Rheumatology Department, Christian Medical College at Vellore, India between March 2013 and January 2015. AntiC1q antibody was assayed by ELISA (Demeditec Diagnostics GmbH, Germany). Logistic regression analysis was performed to find the association of anti-C1q antibodies with serological and clinical parameters in SLE including Lupus Nephritis (LN). Results: Sixty nine patients (54.76%) out of 126 SLE patients had LN. Anti-C1q levels were higher in patients with LN as compared to those without (p

Published
2017
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22. Influence of the age at diagnosis in the disease expression of primary Sjögren syndrome. Analysis of 12,753 patients from the Sjögren Big Data Consortium

Author

Retamozo, Soledad, primary, Acar-Denizli, Nihan, additional, Horváth, Ildiko Fanny, additional, Ng, Wan-Fai, additional, Rasmussen, Astrid, additional, Dong, Xu, additional, Li, Xiaomei, additional, Baldini, Chiara, additional, Olsson, Peter, additional, Priori, Roberta, additional, Seror, Raphaèle, additional, Gottenberg, Jacques-Eric, additional, Kruize, Aike A., additional, Hernandez-Molina, Gabriela, additional, Vissink, Arjan, additional, Sandhya, Pulukool, additional, Armagan, Berkan, additional, Quartuccio, Luca, additional, Sebastian, Agata, additional, Praprotnik, Sonja, additional, Bartoloni, Elena, additional, Kwok, Seung-Ki, additional, Kvarnstrom, Marika, additional, Rischmueller, Maureen, additional, Soláns-Laqué, Roser, additional, Sene, Damien, additional, Pasoto, Sandra G., additional, Suzuki, Yasunori, additional, Isenberg, David A., additional, Valim, Valéria, additional, Nordmark, Gunnel, additional, Nakamura, Hideki, additional, Fernandes Moça Trevisani, Virginia, additional, Hofauer, Benedikt, additional, Sisó-Almirall, Antoni, additional, Giacomelli, Roberto, additional, Devauchelle-Pensec, Valerie, additional, Bombardieri, Michele, additional, Atzeni, Fabiola, additional, Hammenfors, Daniel, additional, Maure, Brenda, additional, Carsons, Steven E., additional, Gheita, Tamer, additional, Sánchez-Berná, Isabel, additional, López-Dupla, Miguel, additional, Morel, Jacques, additional, Inanç, Nevsun, additional, Fonseca-Aizpuru, Eva, additional, Morcillo, César, additional, Vollenweider, Cristina, additional, Melchor, Sheila, additional, Vázquez, Marcos, additional, Díaz-Cuiza, Ericka, additional, Consani-Fernández, Sandra, additional, de-Miguel-Campo, Borja, additional, Szántó, Antónia, additional, Bombardieri, Stefano, additional, Gattamelata, Angelica, additional, Hinrichs, Anneline, additional, Sánchez-Guerrero, Jorge, additional, Danda, Debashish, additional, Kilic, Levent, additional, De Vita, Salvatore, additional, Wiland, Piotr, additional, Gerli, Roberto, additional, Park, Sung-Hwan, additional, Wahren-Herlenius, Marie, additional, Bootsma, Hendrika, additional, Mariette, Xavier, additional, Ramos-Casals, Manuel, additional, and Brito-Zerón, Pilar, additional

Published
2021
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23. Salivary free light chains and salivary immunoglobulins as potential non‐invasive biomarkers in primary Sjögren's syndrome

Author

Sandhya, Pulukool, primary, Kabeerdoss, Jayakanthan, additional, Christudoss, Pamela, additional, Arulraj, Roy, additional, Mandal, Santosh Kumar, additional, Janardana, Ramya, additional, Chebbi, Pramod Prahlad, additional, Ganesan, Maya Pavalasseril, additional, Mahasampath, Gowri, additional, and Danda, Debashish, additional

Published
2021
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32. Systemic phenotype related to primary Sjogren's syndrome in 279 patients carrying isolated anti-La/SSB antibodies

Author

Acar-Denizli, Nihan, Horváth, Ildiko-Fanny, Mandl, Thomas, Priori, Roberta, Vissink, Arjan, Hernandez-Molina, Gabriela, Armagan, Berkan, Praprotnik, Sonja, Sebastian, Agata, Bartoloni, Elena, Rischmueller, Maureen, Pasoto, Sandra G, Nordmark, Gunnel, Nakamura, Hideki, Fernandes Moça Trevisani, Virginia, Retamozo, Soledad, Carsons, Steven E, Maure-Noia, Brenda, Sánchez-Berná, Isabel, López-Dupla, Miguel, Fonseca-Aizpuru, Eva, Melchor Díaz, Sheila, Vázquez, Marcos, Díaz Cuiza, P Ericka, de Miguel Campo, Borja, Ng, Wan-Fai, Rasmussen, Astrid, Dong, Xu, Li, Xiaomei, Baldini, Chiara, Seror, Raphaele, Gottenberg, Jacques-Eric, Kruize, Aike A, Sandhya, Pulukool, Gandolfo, Saviana, Kwok, Seung-Ki, Kvarnstrom, Marika, Solans, Roser, Sene, Damien, Suzuki, Yasunori, Isenberg, David A, Valim, Valeria, Hofauer, Benedikt, Giacomelli, Roberto, Devauchelle-Pensec, Valerie, Atzeni, Fabiola, Gheita, Tamer A, Morel, Jacques, Izzo, Raffaella, Bootsma, Hendrika, Personalized Healthcare Technology (PHT), and Translational Immunology Groningen (TRIGR)

Subjects
musculoskeletal diseases, anti-Ro/SSA antibodies, primary Sjogren's syndrome, systemic disease, DIAGNOSIS, Cohort Studies, stomatognathic system, big data, LYMPHOMA, Humans, isolated La/SSB auto-antibodies, CLASSIFICATION CRITERIA, Registries, DISEASE-ACTIVITY INDEX, IDENTIFICATION, eye diseases, Sjogren's Syndrome/diagnosis, stomatognathic diseases, Phenotype, MANIFESTATIONS, LOWER PREVALENCE, Female, AUTOANTIBODIES, LA, CONSENSUS, ESSDAI
Abstract

Objective. To evaluate the systemic phenotype associated with the presence of isolated anti-La/SSB antibodies in a large international registry of patients with primary Sjogren's syndrome (pSS) fulfilling the 2002 classification criteria. Methods. The Big Data Sjogren Project Consortium is an international, multicentre registry created in 2014. Baseline clinical information from leading centres on clinical research in SS of the 5 continents was collected. Combination patterns of anti-Ro/SSA-La/SSB antibodies at the time of diagnosis defined the following four immunological phenotypes: double positive (combined Ro/SSA and La/SSB,) isolated anti-Ro/SSA, isolated anti-La/SSB, and immunonegative. Results. The cohort included 12,084 patients (11,293 females, mean 52.4 years) with recorded ESSDAI scores available. Among them, 279 (2.3%) had isolated anti-La/SSB antibodies. The mean total ESSDAI score at diagnosis of patients with pSS carrying isolated anti-La/SSB was 6.0, and 80.4% of patients had systemic activity (global ESSDAI score >= 1) at diagnosis. The domains with the highest frequency of active patients were the biological (42.8%), glandular (36.8%) and articular (31.2%) domains. Patients with isolated anti-La/ SSB showed a higher frequency of active patients in all ESSDAI domains but two (articular and peripheral nerve) in comparison with immune-negative patients, and even a higher absolute frequency in six clinical ESSDAI domains in comparison with patients with isolated anti-Ro/SSA. In addition, patients with isolated anti-La/SSB showed a higher frequency of active patients in two ESSDAI domains (pulmonary and glandular) with respect to the most active immunological subset (double-positive antibodies). Meanwhile, systemic activity detected in patients with isolated anti-La/SSB was overwhelmingly low. Even in ESSDAI domains where patients with isolated anti-La/SSB had the highest frequencies of systemic activity (lymphadenopathy and muscular), the percentage of patients with moderate or high activity was lower in comparison with the combined Ro/SSA and La/SSB group. Conclusion. Patients carrying isolated La/SSB antibodies represent a very small subset of patients with a systemic SS phenotype characterised by a significant frequency of active patients in most clinical ESSDAI domains but with a relative low frequency of the highest severe organ-specific involvements. Primary SS still remains the best clinical diagnosis for this subset of patients.

Published
2020

35. Spectrum of clinical features and genetic variants in mevalonate kinase (MVK) gene of South Indian families suffering from Hyperimmunoglobulin D Syndrome

Author

Govindaraj, Geeta Madathil, primary, Jain, Abhinav, additional, Peethambaran, Geetha, additional, Bhoyar, Rahul C., additional, Vellarikkal, Shamsudheen Karuthedath, additional, Ganapati, Arvind, additional, Sandhya, Pulukool, additional, Edavazhippurath, Athulya, additional, Dhanasooraj, Dhananjayan, additional, Puthenpurayil, Jayakrishnan Machinary, additional, Chakkiyar, Krishnan, additional, Mishra, Anushree, additional, Batra, Arushi, additional, Punnen, Anu, additional, Kumar, Sathish, additional, Sivasubbu, Sridhar, additional, and Scaria, Vinod, additional

Published
2020
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36. Salivary free light chains and salivary immunoglobulins as potential non‐invasive biomarkers in primary Sjögren's syndrome.

Author

Sandhya, Pulukool, Kabeerdoss, Jayakanthan, Christudoss, Pamela, Arulraj, Roy, Mandal, Santosh Kumar, Janardana, Ramya, Chebbi, Pramod Prahlad, Ganesan, Maya Pavalasseril, Mahasampath, Gowri, and Danda, Debashish

Subjects
*IMMUNOGLOBULIN light chains, *SJOGREN'S syndrome, *IMMUNOGLOBULINS, *SIALADENITIS, *SYSTEMIC lupus erythematosus, *XEROSTOMIA, *CARDIAC amyloidosis
Abstract

Background: B cells contribute significantly to the pathogenesis of primary Sjögren's syndrome (pSS). Free light chains (FLCs) are generated during the production of immunoglobulins (Igs) and are surrogates of B cell activity. We hypothesized that salivary FLCs and salivary Igs could represent salivary gland inflammation and therefore, serve as biomarkers in pSS. Methods: Patients >18 years old fulfilling the American College of Rheumatology / European League Against Rheumatism (EULAR) 2016 criteria for pSS and age‐matched healthy and disease controls (sicca non‐pSS, rheumatoid arthritis, systemic lupus erythematosus) were recruited for this cross‐sectional study. FLCs in saliva and serum were measured by immunoturbidimetry. Serum and salivary Igs were measured by nephelometry and enzyme‐linked immunosorbent assay, respectively. Area under the receiver operator characteristic curve was determined. The factors influencing the serum and salivary FLCs in pSS were determined using backward linear regression. Results: A total of 78 patients with pSS, 76 healthy controls and 62 disease controls were recruited. Median EULAR SS disease activity index (interquartile range) was 1 (3.75). Serum FLCκ and FLCλ, salivary FLCλ, serum and salivary IgG, salivary IgM was significantly higher in the pSS group compared to the controls. Areas under the curve for salivary FLCλ, serum FLCκ, serum and salivary IgG were 0.75, 0.72, 0.78 and 0.77, respectively. Regression analysis showed that salivary FLCκ, salivary FLCλ and salivary IgG were associated with positive salivary gland histopathology. Use of immunosuppressants and glucocorticoids was associated with lower values of salivary parameters. Conclusion: Salivary FLCλ and salivary IgG were significantly different between pSS and control groups and could be potential non‐invasive biomarkers in pSS. These findings should be confirmed in larger longitudinal studies. [ABSTRACT FROM AUTHOR]

Published
2022
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39. Childhood-onset of primary Sjögren's syndrome: phenotypic characterization at diagnosis of 158 children.

Author

Ramos-Casals, Manuel, Acar-Denizli, Nihan, Vissink, Arjan, Brito-Zerón, Pilar, Li, Xiaomei, Carubbi, Francesco, Priori, Roberta, Toplak, Nataša, Baldini, Chiara, Faugier-Fuentes, Enrique, Kruize, Aike A, Mandl, Thomas, Tomiita, Minako, Gandolfo, Saviana, Hashimoto, Kunio, Hernandez-Molina, Gabriela, Hofauer, Benedikt, Mendieta-Zerón, Samara, Rasmussen, Astrid, and Sandhya, Pulukool

Subjects
SJOGREN'S syndrome diagnosis, ANALYSIS of variance, AUTOIMMUNE diseases, PEDIATRICS, DESCRIPTIVE statistics, CHI-squared test, DATA analysis software, SJOGREN'S syndrome, PHENOTYPES, CHILDREN
Abstract

Objectives To characterize the phenotypic presentation at diagnosis of childhood-onset primary SS. Methods The Big Data Sjögren Project Consortium is an international, multicentre registry using worldwide data-sharing cooperative merging of pre-existing clinical SS databases from the five continents. For this study, we selected those patients in whom the disease was diagnosed below the age of 19 years according to the fulfilment of the 2002/2016 classification criteria. Results Among the 12 083 patients included in the Sjögren Big Data Registry, 158 (1.3%) patients had a childhood-onset diagnosis (136 girls, mean age of 14.2 years): 126 (80%) reported dry mouth, 111 (70%) dry eyes, 52 (33%) parotid enlargement, 118/122 (97%) positive minor salivary gland biopsy and 60/64 (94%) abnormal salivary US study, 140/155 (90%) positive ANA, 138/156 (89%) anti-Ro/La antibodies and 86/142 (68%) positive RF. The systemic EULAR Sjögren's syndrome disease activity index (ESSDAI) domains containing the highest frequencies of active patients included the glandular (47%), articular (26%) and lymphadenopathy (25%) domains. Patients with childhood-onset primary SS showed the highest mean ESSDAI score and the highest frequencies of systemic disease in 5 (constitutional, lymphadenopathy, glandular, cutaneous and haematological) of the 12 ESSDAI domains, and the lowest frequencies in 4 (articular, pulmonary, peripheral nerve and CNS) in comparison with patients with adult-onset disease. Conclusions Childhood-onset primary SS involves around 1% of patients with primary SS, with a clinical phenotype dominated by sicca features, parotid enlargement and systemic disease. Age at diagnosis plays a key role in modulating the phenotypic expression of the disease. [ABSTRACT FROM AUTHOR]

Published
2021
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40. Phenotype of biopsy-proven patients with primary Sjögren syndrome lacking ro autoantibodies: high frequency of dryness symptoms with low systemic activity (big data Sjögren projct)

Author

Retamozo, Soledad, Acar-Denizli, Nihan, Ng, Wan Fai, Horvath, Ildiko Fanny, Rasmussen, Astrid, Seror, Raphaele, Xiaomei, LI, Baldini, Chiara, Gottenberg, Jacques-Eric, Sandhya, Pulukool, Quartuccio, Luca, Priori, Roberta, Hernandez-Molina, Gabriela, Armagan, Berkan, Kruize, Aike A, Kwok, Seung-Ki, Kvarnström, Marika, Praprotnik, Sonja, Sène, Damien, Gerli, Roberto, Solans-Laqué, Roser, Rischmueller, Maureen, Mandl, Thomas, Suzuki, Yasunori, Isenberg, David, Valim, Valeria, Sebastian, Agata, Nordmark, Gunnel, Bootsma, Hendrika, Nakamura, Hideki, Giacomelli, Roberto, Devauchelle-Pensec, Valérie, Hofauer, Benedikt, Bombardieri, Michele, Trevisani, Virginia Fernandes Moça, Hammenfors, Daniel, Pasoto, Sandra, Gheita, Tamer A, Atzeni, Fabiola, Morel, Jacques, Vollenveider, Cristina, Consani-Fernández, Sandra, Mariette, Xavier, Ramos-Casals, Manuel, Brito-Zerón, Pilar, Michel, Geneviève, INICSA, UNC, CONICED, Cordoba, Mimar Sinan Üniversitesi, University of Newcastle, Newcastle upon Tyne, UK, University of Debrecen Egyetem [Debrecen], Oklahoma Medical Research Foundation (OMRF), INSERM UMR972, SFR André Lwoff, Université Paris Sud, APHP Paul Brousse, Anhui Provincial Hosp, University of Pisa - Università di Pisa, Institut de Physique et Chimie des Matériaux de Strasbourg (IPCMS), Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS)-Matériaux et nanosciences d'Alsace (FMNGE), Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut de Chimie du CNRS (INC)-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut de Chimie du CNRS (INC)-Centre National de la Recherche Scientifique (CNRS)-Réseau nanophotonique et optique, Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS), Christian Medical College & Hospital, Rheumatology Clinic, Academic Hospital S.M. Della Misericordia, Udine, Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome], Instituto Nacional de Ciencias Médicas y Nutrición Salvador Zubirán - National Institute of Medical Science and Nutrition Salvador Zubiran [Mexico], Rheumatology, Hacettepe University Faculty of Medicine, Ankara, Hacettepe University = Hacettepe Üniversitesi, University Medical Center [Utrecht], Catholic University of Korea, Unit of Experimental Rheumatology, Stockhom (Department of Medicine), Department of Rheumatology (Dep Rheumato - Ljubljana - SLOVENIE), University Medical Centre Slovenia, Université Paris Diderot - Paris 7 (UPD7), University and Azienda Ospedaliera of Perugia, Vall d'Hebron University Hospital [Barcelona], The University of Western Australia (UWA), Malmö University Hospital, Lund University, Kanazawa University (KU), University College of London [London] (UCL), Department of Medicine, Universidade Federal do Espirito Santo, Vitoria, University of Wrocław [Poland] (UWr), Uppsala Universitet [Uppsala], University Medical Center Groningen [Groningen] (UMCG), Nagasaki University, University of L'Aquila [Italy] (UNIVAQ), CHRU Brest - Service de Rhumatologie (CHU - BREST - Rhumato), Centre Hospitalier Régional Universitaire de Brest (CHRU Brest), Lymphocytes B, Autoimmunité et Immunothérapies (LBAI), Université de Brest (UBO)-Institut National de la Santé et de la Recherche Médicale (INSERM)-LabEX IGO Immunothérapie Grand Ouest-Institut Brestois Santé Agro Matière (IBSAM), Université de Brest (UBO), Technische Universität Munchen - Université Technique de Munich [Munich, Allemagne] (TUM), Centre for Experimental Medicine and Rheumatology, London, Queen Mary University of London (QMUL), Federal University of Sao Paulo (Unifesp), Haukeland University Hospital, University of Bergen (UiB), Hospital das Clinicas, Cairo University, Messina and Milan Univ, Milan, University Hospital of Montpellier, German Hospital, Buenos Aires, Hospital Maciel, Université Paris-Sud - Paris 11 (UP11), Hospital Clinic (IDIBAPS), H. CIMA-Sanitas, Barcelona, Université de Strasbourg (UNISTRA)-Matériaux et nanosciences d'Alsace (FMNGE), Institut de Chimie du CNRS (INC)-Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Institut de Chimie du CNRS (INC)-Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Centre National de la Recherche Scientifique (CNRS)-Réseau nanophotonique et optique, Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA), Lymphocyte B et Auto-immunité (LBAI), Université de Brest (UBO)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut Brestois Santé Agro Matière (IBSAM), Université de Brest (UBO)-Institut Brestois Santé Agro Matière (IBSAM), and Université de Brest (UBO)-Institut National de la Santé et de la Recherche Médicale (INSERM)

Subjects
[SDV.IMM] Life Sciences [q-bio]/Immunology, [SDV.IMM]Life Sciences [q-bio]/Immunology, ComputingMilieux_MISCELLANEOUS
Abstract

International audience

Published
2019

42. THU0277 HOW THE AGE AT DIAGNOSIS MODIFIES THE PHENOTYPE OF PRIMARY SJÖGREN SYNDROME: ANALYSIS IN 11,420 PATIENTS (BIG DATA SJÖGREN PROJECT)

Author

Retamozo, Soledad, primary, Acar-Denizli, Nihan, additional, Ng, Wan Fai, additional, Horváth, Ildiko Fanny, additional, Rasmussen, Astrid, additional, Seror, Raphaèle, additional, Xiaomei, LI, additional, Baldini, Chiara, additional, Gottenberg, Jacques-Eric, additional, Sandhya, Pulukool, additional, Quartuccio, Luca, additional, Priori, Roberta, additional, Hernandez-Molina, Gabriela, additional, Armagan, Berkan, additional, Kruize, Aike A., additional, Kwok, Seung-Ki, additional, Kvarnstrom, Marika, additional, Praprotnik, Sonja, additional, Sene, Damien, additional, Bocci, Elena Bartoloni, additional, Solans-Laqué, Roser, additional, Rischmueller, Maureen, additional, Mandl, Thomas, additional, Suzuki, Yasunori, additional, Isenberg, David, additional, Valim, Valeria, additional, Sebastian, Agata, additional, Nordmark, Gunnel, additional, Bootsma, Hendrika, additional, Nakamura, Hideki, additional, Giacomelli, Roberto, additional, Devauchelle-Pensec, Valerie, additional, Hofauer, Benedikt, additional, Bombardieri, Michele, additional, Hammenfors, Daniel, additional, Pasoto, Sandra, additional, Gheita, Tamer A, additional, Atzeni, Fabiola, additional, Morel, Jacques, additional, Vollenveider, Cristina, additional, Consani-Fernández, Sandra, additional, Mariette, Xavier, additional, Ramos-Casals, Manuel, additional, and Brito-Zerón, Pilar, additional

Published
2019
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43. AB0550 PHENOTYPE OF BIOPSY-PROVEN PATIENTS WITH PRIMARY SJÖGREN SYNDROME LACKING RO AUTOANTIBODIES: HIGH FREQUENCY OF DRYNESS SYMPTOMS WITH LOW SYSTEMIC ACTIVITY (BIG DATA SJÖGREN PROJECT)

Author

Retamozo, Soledad, primary, Acar-Denizli, Nihan, additional, Ng, Wan Fai, additional, Horváth, Ildiko Fanny, additional, Rasmussen, Astrid, additional, Seror, Raphaèle, additional, Xiaomei, LI, additional, Baldini, Chiara, additional, Gottenberg, Jacques-Eric, additional, Sandhya, Pulukool, additional, Quartuccio, Luca, additional, Priori, Roberta, additional, Hernandez-Molina, Gabriela, additional, Armagan, Berkan, additional, Kruize, Aike A., additional, Kwok, Seung-Ki, additional, Kvarnstrom, Marika, additional, Praprotnik, Sonja, additional, Sene, Damien, additional, Gerli, Roberto, additional, Solans-Laqué, Roser, additional, Rischmueller, Maureen, additional, Mandl, Thomas, additional, Suzuki, Yasunori, additional, Isenberg, David, additional, Valim, Valeria, additional, Sebastian, Agata, additional, Nordmark, Gunnel, additional, Bootsma, Hendrika, additional, Nakamura, Hideki, additional, Giacomelli, Roberto, additional, Devauchelle-Pensec, Valerie, additional, Hofauer, Benedikt, additional, Bombardieri, Michele, additional, Moça Trevisani, Virginia Fernandes, additional, Hammenfors, Daniel, additional, Pasoto, Sandra, additional, Gheita, Tamer A., additional, Atzeni, Fabiola, additional, Morel, Jacques, additional, Vollenveider, Cristina, additional, Consani-Fernández, Sandra, additional, Mariette, Xavier, additional, Ramos-Casals, Manuel, additional, and Brito-Zerón, Pilar, additional

Published
2019
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44. OP0126 LYMPHOMA ARISING AT THE TIME OF DIAGNOSIS OF PRIMARY SJÖGREN SYNDROME: A HIGHLY-ACTIVE SYSTEMIC SUBSET OF THE DISEASE

Author

Retamozo, Soledad, primary, Acar-Denizli, Nihan, additional, Ng, Wan Fai, additional, Szántó, Antónia, additional, Rasmussen, Astrid, additional, Seror, Raphaèle, additional, Xiaomei, LI, additional, Baldini, Chiara, additional, Gottenberg, Jacques-Eric, additional, Sandhya, Pulukool, additional, Quartuccio, Luca, additional, Priori, Roberta, additional, Hernandez-Molina, Gabriela, additional, Armagan, Berkan, additional, Kruize, Aike A., additional, Kwok, Seung-Ki, additional, Kvarnstrom, Marika, additional, Praprotnik, Sonja, additional, Sene, Damien, additional, Solans-Laqué, Roser, additional, Rischmueller, Maureen, additional, Mandl, Thomas, additional, Suzuki, Yasunori, additional, Isenberg, David, additional, Valim, Valeria, additional, Sebastian, Agata, additional, Nordmark, Gunnel, additional, Bootsma, Hendrika, additional, Nakamura, Hideki, additional, Giacomelli, Roberto, additional, Devauchelle-Pensec, Valerie, additional, Hofauer, Benedikt, additional, Bombardieri, Michele, additional, Fernandes Moça Trevisani, Virginia, additional, Hammenfors, Daniel, additional, Pasoto, Sandra, additional, Gheita, Tamer A, additional, Atzeni, Fabiola, additional, Morel, Jacques, additional, Vollenveider, Cristina, additional, Consani-Fernández, Sandra, additional, Mariette, Xavier, additional, Ramos-Casals, Manuel, additional, Brito-Zerón, Pilar, additional, and Bocci, Elena Bartoloni, additional

Published
2019
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45. Preliminary Definitions for Sacroiliac Joint Pathologies in the OMERACT Juvenile Idiopathic Arthritis MRI Score (OMERACT JAMRIS‐SIJ)

Author

Otobo, Tarimobo Michael, primary, Tolend, Mirkamal, additional, Maksymowych, Walter P, additional, Conaghan, Philip G, additional, Heijd, Desiree, additional, Weiss, Pamela, additional, Sudol‐Szopinska, Iwona, additional, Herregods, Nele, additional, Jaremko, Jacob L, additional, Jans, Lennart, additional, Meyer, Arthur B, additional, Rumsey, Dax Rumsey, additional, Inarejos, Emilio C, additional, Kirkhus, Eva, additional, Thorpe, Kevin, additional, Rossum, Marion A J, additional, Perez, Manuela, additional, Tzaribachev, Nikolay, additional, Sandhya, Pulukool, additional, Tse, Shirley, additional, Simone, Appenzeller, additional, Swami, Vimarsha G, additional, Touma, Zahi, additional, Lambert, Robert G, additional, and Doria, Andrea S, additional

Published
2019
Full Text
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46. Preliminary Definitions for Sacroiliac Joint Pathologies in the OMERACT Juvenile Idiopathic Arthritis Magnetic Resonance Imaging Score (OMERACT JAMRIS-SIJ)

Author

Otobo, Tarimobo M., primary, Conaghan, Philip G., additional, Maksymowych, Walter P., additional, van der Heijde, Desiree, additional, Weiss, Pamela, additional, Sudol-Szopinska, Iwona, additional, Herregods, Nele, additional, Jaremko, Jacob L., additional, Meyers, Arthur B., additional, Rumsey, Dax, additional, Inarejos, Emilio C., additional, Kirkhus, Eva, additional, Stimec, Jennifer, additional, Panwar, Jyoti, additional, Thorpe, Kevin, additional, Jans, Lennart, additional, van Rossum, M.A., additional, Tolend, Mirkamal, additional, Perez, Manuela, additional, Tzaribachev, Nikolay, additional, Sandhya, Pulukool, additional, Tse, Shirley, additional, Simone, Appenzeller, additional, Swami, Vimarsha G., additional, Touma, Zahi, additional, Lambert, Robert, additional, and Doria, Andrea S., additional

Published
2019
Full Text
View/download PDF

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