Search

Heating and cooling requirement differences across climates not only have carbon emissions and energy efficiency implications but also impact indoor air quality (IAQ) and health. Energy and IAQ building simulation models help understand tradeoffs or co-benefits, but these have not been applied to evaluate climate zone or multi-family home differences. We modeled a four-story multi-family home in six U.S. climate zones and quantified energy, IAQ, and health outcomes with EnergyPlus, CONTAM, and a pediatric asthma systems science model. Pollutant sources included cooking and ambient. Outputs were daily PM

Most epidemiologic studies fail to capture the impact of spatiotemporal fluctuations in traffic on exposure to traffic-related air pollutants in the near-road population. Using a case-crossover design and the Research LINE source (R-LINE) dispersion model with spatiotemporally resolved highway traffic data, we quantified associations between primary pollutants generated by highway traffic—particulate matter with an aerodynamic diameter less than or equal to 2.5 μm (PM2.5), oxides of nitrogen (NOx), and black carbon (BC)—and daily nonaccidental, respiratory, cardiovascular, and cerebrovascular mortality among persons who had resided within 1 km (0.6 mile) of major highways in the Puget Sound area of Washington State between 2009 and 2013. We estimated these associations using conditional logistic regression, adjusting for time-varying covariates. Although highly resolved modeled concentrations of PM2.5, NOx, and BC from highway traffic in the hours before death were used, we found no evidence of an association between mortality and the preceding 24-hour average PM2.5 exposure (odds ratio = 0.99, 95% confidence interval: 0.96, 1.02) or exposure during shorter averaging periods. This work did not support the hypothesis that mortality risk was meaningfully higher with greater exposures to PM2.5, NOx, and BC from highways in near-road populations, though we did incorporate a novel approach to estimate exposure to traffic-generated air pollution based on detailed traffic congestion data.

The intake fraction (iF) expresses population exposure resulting from pollutant emissions. City-wide iFs estimated using simple one-compartment models, which have been used in a number of previous studies, have significant uncertainties and do not capture the intra-urban variation in exposure that is important for estimating health effects associated with traffic-related air pollutants. We present a novel and efficient approach for developing spatially-resolved iF estimates using dispersion modeling for near-road exposures that accounts for the spatial and temporal variation in meteorology, emissions and the population living and working near major roads. Using the new approach, iF estimates are developed for emissions of traffic-related fine particulate matter (PM2.5) in Lisbon, Portugal, and compared to estimates from a one-compartment model. Both methods use local meteorological and population data and represent exposures for a total of 2.8 million people. The new method produces an overall iF value of 16.4 ppm for the Lisbon metropolitan area, over twice that of the one-compartment model (8.1 ppm). Most of the exposure (12.0 ppm) occurs for the subset of the population (1.0 million people) living or working within 500 m of highways and major arterials. The iF for the remainder of the population (1.8 million people) is only 4.3 ppm. The spatially-resolved iF estimate accounts for high concentration areas, which can be densely populated, and accounts for much or most of the exposure from traffic-related emissions. The new method is computationally efficient and can improve estimates of exposure and health impacts occurring in urban areas, leading to more effective urban and transportation planning decisions to mitigate impacts.

In air quality management, reducing emissions from pollutant sources often forms the primary response to attaining air quality standards and guidelines. Despite the broad success of air quality management in the US, challenges remain. As examples: allocating emissions reductions among multiple sources is complex and can require many rounds of negotiation; health impacts associated with emissions, the ultimate driver for the standards, are not explicitly assessed; and long dispersion model run-times, which result from the increasing size and complexity of model inputs, limit the number of scenarios that can be evaluated, thus increasing the likelihood of missing an optimal strategy. A new modeling framework, called the “Framework for Rapid Emissions Scenario and Health impact ESTimation” (FRESH-EST), is presented to respond to these challenges. FRESH-EST estimates concentrations and health impacts of alternative emissions scenarios at the urban scale, providing efficient computations from emissions to health impacts at the Census block or other desired spatial scale. In addition, FRESH-EST can optimize emission reductions to meet specified environmental and health constraints, and a convenient user interface and graphical displays are provided to facilitate scenario evaluation. The new framework is demonstrated in an SO2 non-attainment area in southeast Michigan with two optimization strategies: the first minimizes emission reductions needed to achieve a target concentration; the second minimizes concentrations while holding constant the cumulative emissions across local sources (e.g., an emissions floor). The optimized strategies match outcomes in the proposed SO2 State Implementation Plan without the proposed stack parameter modifications or shutdowns. In addition, the lower health impacts estimated for these strategies suggest that FRESH-EST could be used to identify potentially more desirable pollution control alternatives in air quality management planning. Keywords: Air quality management, Optimization, Health impact assessment, FRESH-EST

Localized variations in traffic volume and speed can influence air pollutant emissions and corresponding concentrations in nearby communities, but most studies have utilized only aggregated traffic activity data. In this study, we compared the estimated influence of highway traffic activity on concentrations of primary oxides of nitrogen (NOx) and fine particulate matter (PM2.5) in communities near highways using a dispersion model informed by highly spatiotemporally-resolved variations of traffic volume and flow compared to the use of Annual Average Daily Traffic (AADT) data at a few locations. We used two sources of traffic activity data on 500 half-mile roadway segments on the five major highways in the Washington State Puget Sound during 2013. The first consisted of vehicle counts available every half-mile and 5 min; the second was traffic information (e.g., AADT) aggregated across the year and roadway network. Using the Motor Vehicle Emissions Simulator (MOVES) and the Research Line source dispersion model (RLINE), we modeled hourly concentrations of primary NOx and PM2.5 generated by highway traffic at nearly 4000 residences within 1 km of major highways. These concentrations were aggregated to daily and annual average concentrations, which were compared by input data source. At most locations, concentrations of primary NOx and PM2.5 modeled using the resolved traffic data had similar spatial and temporal distributions to concentrations predicted using the AADT data. However, several areas showed large differences. For example, 25% of residences within 150 m of a highway had concentrations that differed by more than 19% (8 ppb) for NOx and 32% (0.7 μg/m3) for PM2.5, and the AADT data consistently predicted lower concentrations than the resolved traffic data. Our findings indicate that temporal and spatial variation in traffic patterns can result in complex spatiotemporal variations of air pollutant concentrations that can be captured with the use of dispersion modeling with the appropriate inputs. The use of spatiotemporally resolved traffic activity data can improve exposure estimates and help reduce exposure measurement error in epidemiological studies, especially in communities near highly congested highways.

Background: Traffic varies dramatically by time and place, within and between highways. These variations likely influence the distribution of traffic-generated pollutants in nearby communities but ...

Background: A large proportion of the US population lives and works near highways where levels of traffic-generated air pollutants are the highest. While these pollutants have been linked to advers...

The environmental burden of disease is the mortality and morbidity attributable to exposures of air pollution and other stressors. The inequality metrics used in cumulative impact and environmental justice studies can be incorporated into environmental burden studies to better understand the health disparities of ambient air pollutant exposures. This study examines the diseases and health disparities attributable to air pollutants for the Detroit urban area. We apportion this burden to various groups of emission sources and pollutants, and show how the burden is distributed among demographic and socioeconomic subgroups. The analysis uses spatially-resolved estimates of exposures, baseline health rates, age-stratified populations, and demographic characteristics that serve as proxies for increased vulnerability, e.g., race/ethnicity and income. Based on current levels, exposures to fine particulate matter (PM2.5), ozone (O3), sulfur dioxide (SO2), and nitrogen dioxide (NO2) are responsible for more than 10,000 disability-adjusted life years (DALYs) per year, causing an annual monetized health impact of $6.5 billion. This burden is mainly driven by PM2.5 and O3 exposures, which cause 660 premature deaths each year among the 945,000 individuals in the study area. NO2 exposures, largely from traffic, are important for respiratory outcomes among older adults and children with asthma, e.g., 46% of air-pollution related asthma hospitalizations are due to NO2 exposures. Based on quantitative inequality metrics, the greatest inequality of health burdens results from industrial and traffic emissions. These metrics also show disproportionate burdens among Hispanic/Latino populations due to industrial emissions, and among low income populations due to traffic emissions. Attributable health burdens are a function of exposures, susceptibility and vulnerability (e.g., baseline incidence rates), and population density. Because of these dependencies, inequality metrics should be calculated using the attributable health burden when feasible to avoid potentially underestimating inequality. Quantitative health impact and inequality analyses can inform health and environmental justice evaluations, providing important information to decision makers for prioritizing strategies to address exposures at the local level.

Comparative evaluations are needed to assess the suitability of near-road air pollution models for traffic-related ultrafine particle number concentration (PNC). Our goal was to evaluate the ability of dispersion (CALINE4, AERMOD, R-LINE, and QUIC) and regression models to predict PNC in a residential neighborhood (Somerville) and an urban center (Chinatown) near highways in and near Boston, Massachusetts. PNC was measured in each area, and models were compared to each other and measurements for hot (>18 °C) and cold (