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1. Distinct and additive effects of calorie restriction and rapamycin in aging skeletal muscle

2. Molecular and phenotypic analysis of rodent models reveals conserved and species-specific modulators of human sarcopenia

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3. Alterations to mTORC1 signaling in the skeletal muscle differentially affect whole-body metabolism

4. mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis

5. eIF3-f function in skeletal muscles: To stand at the crossroads of atrophy and hypertrophy

6. The initiation factor eIF3-f is a major target for Atrogin1/MAFbx function in skeletal muscle atrophy

7. Mechanisms regulating neuromuscular junction development and function and causes of muscle wasting

8. Activation of mTORC1 in skeletal muscle regulates whole-body metabolism through FGF21

9. Degradation of MyoD Mediated by the SCF (MAFbx) Ubiquitin Ligase

10. Cyclin E–Cdk2 Phosphorylation Promotes Late G1-Phase Degradation of MyoD in Muscle Cells

11. Sustained Activation of mTORC1 in Skeletal Muscle Inhibits Constitutive and Starvation-Induced Autophagy and Causes a Severe, Late-Onset Myopathy

12. Myostatin inactivation increases myotube size through regulation of translational initiation machinery

13. The Carboxy-Terminal Modulator Protein (CTMP) regulates mitochondrial dynamics

14. PKB and the mitochondria: AKTing on apoptosis

15. MyoD undergoes a distinct G2/M-specific regulation in muscle cells

16. Protein kinase B/Akt at a glance

17. Critical role for lysine 133 in the nuclear ubiquitin-mediated degradation of MyoD

18. Stabilization of MyoD by direct binding to p57(Kip2)

19. The Translation Regulatory Subunit eIF3f Controls the Kinase-Dependent mTOR Signaling Required for Muscle Differentiation and Hypertrophy in Mouse

20. Mutant MyoD lacking Cdc2 phosphorylation sites delays M-phase entry