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1. Development of AML with t(8;21)(q22;q22) and RUNX1-RUNX1T1 fusion following Philadelphia-negative clonal evolution during treatment of CML with Imatinib.

Author

Schafhausen P, Dierlamm J, Bokemeyer C, Bruemmendorf TH, Bacher U, Zander AR, Schnittger S, and Hochhaus A

Subjects
Benzamides, Chromosomes, Human, Pair 21 genetics, Chromosomes, Human, Pair 8 genetics, Female, Humans, Imatinib Mesylate, Karyotyping, Leukemia, Myeloid, Chronic, Atypical, BCR-ABL Negative pathology, Middle Aged, RUNX1 Translocation Partner 1 Protein, Antineoplastic Agents therapeutic use, Core Binding Factor Alpha 2 Subunit genetics, Leukemia, Myelogenous, Chronic, BCR-ABL Positive drug therapy, Leukemia, Myeloid, Acute genetics, Leukemia, Myeloid, Chronic, Atypical, BCR-ABL Negative genetics, Oncogene Proteins, Fusion genetics, Piperazines therapeutic use, Pyrimidines therapeutic use, Translocation, Genetic genetics
Published
2009
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