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Your search keyword '"Immunopathology -- Research"' showing total 6 results
Start Over Descriptor "Immunopathology -- Research" Topic biological sciences
6 results on '"Immunopathology -- Research"'

1. Autonomous University Barcelona Researcher Updates Understanding of Cytokines (Immunopathological Mechanisms of Bird-Related Hypersensitivity Pneumonitis)

Subjects
Animals -- Diseases, Bacterial pneumonia -- Development and progression, Birds -- Diseases, Immunopathology -- Research, Zoological research, Pneumonia -- Development and progression, Biological sciences, Health
Abstract

2023 FEB 21 (NewsRx) -- By a News Reporter-Staff News Editor at Life Science Weekly -- New research on cytokines is the subject of a new report. According to news [...]

Published
2023

2. Immunopathogenesis of dengue hemorrhagic fever

Author

Lei, Huan-Yao, Huang, Kao-Jean, Lin, Yee-Shin, Yeh, Trai-Ming, Liu, Hsiao-Sheng, and Liu, Ching-Chuan

Subjects
Hemorrhagic fever -- Risk factors, Hemorrhagic fever -- Diagnosis, Hemorrhagic fever -- Care and treatment, Hemorrhagic fever -- Research, Immunopathology -- Research, Biological sciences
Abstract

Abstract: Dengue virus infection causes Dengue Fever (DF), Dengue Hemorrhagic Fever (DHF) and Dengue Shock Syndrome (DSS) whose pathogeneses were not clearly understood. A new hypothesis of immunopathogenesis is proposed [...]

Published
2008

3. Induction of the chemokines CCL3[alpha], CCL3[alpha] and CCL5 in atherosclerotic patients

Author

Mousa, Alyaa, Al-Zaki, Amani, Taha, Safa, and Bakhiet, Moiz

Subjects
Immunopathology -- Research, Chemokines -- Health aspects, Chemokines -- Genetic aspects, Ligands (Biochemistry) -- Health aspects, Atherosclerosis -- Research, Messenger RNA -- Health aspects, Gene expression -- Health aspects, Inflammation -- Research, Biological sciences
Abstract

Abstract: Chemokines recruit immune cells to inflammatory sites and promote the process of inflammation. The role of these mediators in the disease process in atherosclerosis is not fully studied. The [...]

Published
2007

4. Evolutionary causes and consequences of immunopathology

Author

Graham, Andrea L., Allen, Judith E., and Read, Andrew F.

Subjects
Immunopathology -- Research, Host-parasite relationships -- Research, Virulence (Microbiology) -- Research, Parasitological research, Biological sciences, Environmental issues
Abstract

A study of the evolutionary causes and effects of immunopathology is presented. The evolution of strategies for parasite virulence and host resistance might be shaped by immunity and future work might reveal whether immunopathology constrains the evolution of resistance in all host taxa.

Published
2005

5. Differential cytokine expression in avian cells in response to invasion by Salmonella typhimurium, Salmonella enteritidis and Salmonella gallinarum

Author

Kaiser, Pete, Rothwell, Lisa, Galyov, Edouard E., Barrow, Paul A., Burnside, Joan, and Wigley, Paul

Subjects
Microbiology -- Research, Cytokines -- Genetic aspects, Gene expression -- Physiological aspects, Cells -- Genetic aspects, Salmonella -- Genetic aspects, Immunopathology -- Research, Host-parasite relationships -- Genetic aspects, Host-parasite relationships -- Physiological aspects, Biological sciences
Abstract

Research has been conducted on Salmonella interactions with the host. The authors have determined cytokine levels including pro-inflammatory cytokines produced after the invasion of host range serotypes Salmonella typhimurium and S. enteritidis and host-specific serotype S. gallinarum into chicken cells in vitro, and have described the mechanisms of immunopathogenesis of salmonellosis in chicken.

Published
2000

6. Both CD4+ and CD8+ lymphocytes reduce the severity of tissue lesions in murine systemic candidiasis, and CD4+ cells also demonstrate strain-specific immunopathological effects

Author

Ashman, Robert B., Fulurija, Alma, and Papadimitrious, John M.

Subjects
T cells -- Research, Candidiasis -- Research, Immunopathology -- Research, Candida albicans -- Research, Cytokines -- Research, Biological sciences
Abstract

Research was conducted to examine the role of T lymphocytes in host responses to sublethal systemic infection with Candida albicans using mAb depletion of CD4+ and CD8+ cells from CBA/CaH and BALB/c mice which develop severe and mild tissue damage, respectively. Results reveal that both CD4+ and CD8+ lymphocytes contribute to the reduction of tissue damage after systemic infection with C. albicans. Findings also provide evidence that the development and expression of CD4+ lymphocyte effector function is influenced by the genetic background of the mouse.

Published
1999

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